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negatively regulates Wnt signaling by binding to the PDZ domain [RGD, Feb 2006].. De plus, nous expédions CXXC Finger Protein 4 Anticorps (55) et CXXC Finger Protein 4 Protéines (6) et beaucoup plus de produits pour cette protéine.
IDAX depletion prevents TET2 downr (Montrer CASP3 Kits ELISA)egulation in differe (Montrer TET2 Kits ELISA)ntiating mouse embryonic stem cells
Age of patients and levels of CXXC4 mRNA, hemoglobin, and marrow blast associated with survival of Myelodysplastic syndrome patients.
EZH2 (Montrer EZH2 Kits ELISA) can activate MAPK (Montrer MAPK1 Kits ELISA) signaling by inhibiting CXXC4 expression.
CXXC4 is a novel potential tumor suppressor directly regulated by EZH2 (Montrer EZH2 Kits ELISA), and its expression is a significant prognosis factor for patients with early stages of gastric cancer.
IDAX expression results in caspase (Montrer CASP3 Kits ELISA) activation and TET2 (Montrer TET2 Kits ELISA) protein downregulation, in a manner that depends on DNA binding through the IDAX CXXC domain, suggesting that IDAX recruits TET2 (Montrer TET2 Kits ELISA) to DNA before degradation
Decreased CXXC4 was associated with kidney cancer metastasis, poor survival, beta-catenin (Montrer CTNNB1 Kits ELISA) translocation, cell proliferation, reduced apoptosis in response to cancer drugs, and upregulation of genes involed in proliferation, invasion, and cell survival.
Idax functions are dependent on the canonical Wnt (Montrer WNT2 Kits ELISA) pathway and are crucial for the anterior neural development
negatively regulates Wnt signaling by binding to the PDZ domain
CXXC-type zinc finger protein 4
, inhibition of the Dvl and axin complex protein
, CXXC finger 4
, Dvl-binding protein IDAX (inhibition of the Dvl and Axin complex)
, inhibitor of the Dvl and Axin complex