Use your antibodies-online credentials, if available.
Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. De plus, nous expédions HAVCR1 Kits (70) et HAVCR1 Protéines (35) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 147 products:
Human Monoclonal HAVCR1 Primary Antibody pour FACS, IHC - ABIN4899579
Ivie, Fennessey, Sheng, Rubin, McClain: Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin. dans PLoS ONE 2011
Show all 4 references for ABIN4899579
Human Polyclonal HAVCR1 Primary Antibody pour WB - ABIN318766
Meyers, Sabatos, Chakravarti, Kuchroo: The TIM gene family regulates autoimmune and allergic diseases. dans Trends in molecular medicine 2005
Show all 2 references for ABIN318766
Mouse (Murine) Polyclonal HAVCR1 Primary Antibody pour ICC, IHC - ABIN1077715
Schindler, Bondeva, Schindler, Claus, Franke, Wolf: Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models. dans Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2016
Human Polyclonal HAVCR1 Primary Antibody pour WB - ABIN657951
Fontaine-Bisson, Renström, Rolandsson, , Payne, Hallmans, Barroso, Franks: Evaluating the discriminative power of multi-trait genetic risk scores for type 2 diabetes in a northern Swedish population. dans Diabetologia 2010
Human Polyclonal HAVCR1 Primary Antibody pour EIA, IHC (p) - ABIN500911
Feigelstock, Thompson, Mattoo, Zhang, Kaplan: The human homolog of HAVcr-1 codes for a hepatitis A virus cellular receptor. dans Journal of virology 1998
Human Polyclonal HAVCR1 Primary Antibody pour WB - ABIN2782775
de Souza, Oak, Jordanhazy, DeKruyff, Fruman, Kane: T cell Ig and mucin domain-1-mediated T cell activation requires recruitment and activation of phosphoinositide 3-kinase. dans Journal of immunology (Baltimore, Md. : 1950) 2008
Data suggest that T-cell immunoglobulin domain and mucin (Montrer SLC13A2 Anticorps) domain containing protein 1 (TIM1 (Montrer TIMELESS Anticorps)) to be under positive natural selection in primates.
Urinary L-FABP (Montrer FABP1 Anticorps), NGAL (Montrer LCN2 Anticorps), Kim-1 and albumin (Montrer ALB Anticorps) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (Montrer HAND1 Anticorps)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (Montrer LDLR Anticorps) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
data suggest that TIM-1 signaling plays a direct role in Breg maintenance and induction both under physiological conditions (in response to ACs (Montrer Acsl1 Anticorps)) and in response to therapy through TIM-1 ligation
Deletion of the mucin (Montrer SLC13A2 Anticorps) domain impaired KIM-1-mediated phagocytic function, resulting in increased proinflammatory cytokine production, decreased antiinflammatory growth factor secretion by proximal epithelial cells, and an increase in tissue macrophages.
Tim-1 is critical for maintaining self-tolerance by regulating IL-10 (Montrer IL10 Anticorps) production in Bregs
Tim-1 expression was lower in a herpes simplex virus-induced Behcet's disease (BD) mouse model compared to that in asymptomatic BD normal (BDN) mice.
Tim-1-Fc protects cardiac grafts from chronic rejection by suppressing CD4 (Montrer CD4 Anticorps) Th17 development and functionality.
KIM-1 shedding is accelerated by worsening renal cellular injury, and excess soluble KIM-1 competitively inhibits efferocytosis.
Endogenous Tim-1 promotes severe systemic autoimmunity and renal disease MRL-Fas(lpr (Montrer FAS Anticorps)) mice.
We conclude that KIM-1 might serve as a sensitive biomarker to screen children for kidney damage induced by environmental toxic agents, such as chromium and arsenic
These results suggest that a 6-amino acid deletion polymorphism in the mucin (Montrer SLC13A2 Anticorps) domain of TIM-1 (Montrer ARHGEF5 Anticorps) protects from HIV-1 infection with a recessive effect.
The present study shows that serum and urine levels of NGAL (Montrer LCN2 Anticorps) and KIM-1 are higher in patients with acute kidney injury than in those without acute kidney injury, and that serum NGAL (Montrer LCN2 Anticorps) and the presence of chronic kidney disease are significant predictors of acute kidney injury in scrub typhus.
KIM-1 increases albumin (Montrer ALB Anticorps) endocytosis in renal tubule epithelial cells.
preliminary data indicates that KIM-1 expression may be associated with stage in Wilms Tumor
Urinary KIM-1 and NAG (Montrer NAGLU Anticorps) levels were positively correlated.
TLR2 (Montrer TLR2 Anticorps), Tim1 (Montrer ARHGEF5 Anticorps), Tim3 (Montrer HAVCR2 Anticorps), TNF-alpha (Montrer TNF Anticorps), IgE and EOS (Montrer IKZF4 Anticorps) play a role in Mycoplasma pneumoniae Pneumonia-related wheezing in children.
The results of our study showed that there is no significant association between TIM-1 (Montrer ARHGEF5 Anticorps) 5383-5397ins/del and Multiple sclerosis (MS) (P = 0.38); however, the frequency of CT genotype of TIM-3 (Montrer HAVCR2 Anticorps) -1541C>T in patient group was significantly higher than the control group, and there was a significant association between CT genotype and MS
Study emphasizes a strong association of the tubular biomarker KIM-1 with chronic kidney disease progression in chronic heart failure and suggests usefulness as cardiorenal marker.
Accumulation of adipose tissue in the RS is related with the visceral adipose amount, KIM-1 and FGF-21 (Montrer FGF21 Anticorps) concentration increase in the blood serum.
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, hepatitis A virus cellular receptor 1 homolog
, kidney injury molecule 1
, t cell immunoglobulin and mucin domain-containing protein 1
, t cell membrane protein 1
, T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, rho guanine nucleotide exchange factor 5