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The protein encoded by this intronless gene is a member of the JUN family, and a functional component of the AP1 transcription factor complex. De plus, nous expédions Jun D Proto-Oncogene Anticorps (162) et Jun D Proto-Oncogene Protéines (8) et beaucoup plus de produits pour cette protéine.
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identify AP-1(JunD/c-Fos) as a novel hematopoietic transcription factor and the requirement of AP-1(JunD/c-Fos) in BMP-4 (Montrer BMP4 Kits ELISA)-induced hematopoiesis during Xenopus hematopoiesis.
In T cells, after serum deprivation HBZ (Montrer HBZ Kits ELISA) induces the expression of Delta JunD isoform. Unlike JunD, Delta JunD induces proliferation and transformation of cells. HBZ (Montrer HBZ Kits ELISA) bypasses translational control of JunD uORF and favors the expression of Delta JunD. The truncated isoform Delta JunD has a central role in the oncogenic process leading to adult T-cell leukemia .
MiR (Montrer MLXIP Kits ELISA)-663a suppresses proliferation and invasion by targeting AP-1 (Montrer FOSB Kits ELISA) component JunD in non-small cell lung cancer cells.
studies showed that down-regulation of JunD in response to TGF-beta (Montrer TGFB1 Kits ELISA) treatment is mediated via the proteasomal degradation pathway.
CARMA1 (Montrer CARD11 Kits ELISA)- and MyD88 (Montrer MYD88 Kits ELISA)-dependent activation of Jun (Montrer JUN Kits ELISA)/ATF-type AP-1 (Montrer FOSB Kits ELISA) complexes is a hallmark of ABC (Montrer ABCB6 Kits ELISA) diffuse large B-cell lymphomas.
recombinant SERPINE2 (Montrer SERPINE2 Kits ELISA) induced a clear inhibition of MMP-13 (Montrer MMP13 Kits ELISA) expression in IL-1alpha-stimulated chondrocytes. This inhibitory effect is likely regulated through a pathway involving ERK 1 (Montrer MAPK3 Kits ELISA)/2, NF-kappaB (Montrer NFKB1 Kits ELISA) and AP-1 (Montrer FOSB Kits ELISA)
miR (Montrer MLXIP Kits ELISA)-494 is a novel regulator of HNPC apoptosis induced by TNF-alpha (Montrer TNF Kits ELISA)
JunD activates miR (Montrer MLXIP Kits ELISA)-29b by enhancing its transcription and processing, which contribute to the inhibitory effect of JunD
Binding of specific AP-1 (Montrer FOSB Kits ELISA) factors, we found JunD associated with the LILRB1 (Montrer LILRB1 Kits ELISA) distal promoter.
Cells in contact with basement membrane undergo transient oscillations between two molecular states defined by their TGFBR3 (Montrer TGFBR3 Kits ELISA)- JUND expression.
BAG3 (Montrer BAG3 Kits ELISA) stabilized JunD mRNA.
The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 (Montrer JUN Kits ELISA) DNA-binding activity and expression of several transcriptions factors in breast cancer cells.
Differentiation-mediated activation of JunD results in enhanced TUBB4a expression in P19 embryonal carcinoma cells.
JunD provides protection in aging-induced endothelial dysfunction and may represent a novel target to prevent reactive oxygen species-driven vascular aging.
AP-1 (Montrer JUN Kits ELISA) composed with JunD and Fra2 (Montrer FOSL2 Kits ELISA) protein plays a primary role in enhancing the transcription level of the CD11c (Montrer ITGAX Kits ELISA) gene in dendritic cells
Nfe2 (Montrer NFE2 Kits ELISA) modulates JunD binding to the Gcm1 (Montrer GCM1 Kits ELISA) promoter via acetylation
5-HT2B (Montrer HTR2B Kits ELISA) blockade enhances liver regeneration by inhibiting induction of TGF-beta1 (Montrer TGFB1 Kits ELISA) expression by hepatic stellate cells that is dependent on 5-HT (Montrer DDC Kits ELISA), ERK (Montrer EPHB2 Kits ELISA) and JunD.
CDK4 (Montrer CDK4 Kits ELISA)-mediated regulation of cell functions via c-Jun (Montrer JUN Kits ELISA) phosphorylation and AP-1 (Montrer JUN Kits ELISA) activation
Downregulation of JunD and cyclin D1 (Montrer CCND1 Kits ELISA) expression in myofibroblasts may be important regarding the mechanism of action of MTA (Montrer DNMT1 Kits ELISA) in Mdr2 (Montrer ABCB4 Kits ELISA)-/- mice
c-Jun (Montrer JUN Kits ELISA), JunB (Montrer JUNB Kits ELISA), and JunD are not functionally equivalent and have both overlapping and independent functions in controlling cellular proliferation, cell-cycle arrest/senescence and the response to stress.
Study supports potential roles for jun (Montrer JUN Kits ELISA) D, jun (Montrer JUN Kits ELISA) B, and fra-1 (Montrer FOSL1 Kits ELISA) in the dietary energy restriction regulation of AP-1 (Montrer JUN Kits ELISA) function in the Sencar mouse skin carcinogenesis model.
protects cells from p53 (Montrer TP53 Kits ELISA)-dependent senescence and apoptosis
The protein encoded by this intronless gene is a member of the JUN family, and a functional component of the AP1 transcription factor complex. It has been proposed to protect cells from p53-dependent senescence and apoptosis. Alternate translation initiation site usage, including non-AUG codons, results in the production of different isoforms.
transcription factor jun-D
, JunD-FL isoform
, activator protein 1
, Jun proto-oncogene related gene d1