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KRT8 is a member of the type II keratin family clustered on the long arm of chromosome 12. De plus, nous expédions Keratin 8 Protéines (50) et Keratin 8 Kits (18) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 795 products:
Human Monoclonal KRT8 Primary Antibody pour FACS, IHC (fro) - ABIN112173
Ivanyi, Groeneveld, Van Doornewaard, Mooi, Hageman: Keratin subtypes in carcinomas of the uterine cervix: implications for histogenesis and differential diagnosis. dans Cancer research 1990
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Human Monoclonal KRT8 Primary Antibody pour IHC (fro), IF - ABIN1106942
Odin, Obrink: Quantitative determination of the organ distribution of the cell adhesion molecule cell-CAM 105 by radioimmunoassay. dans Experimental cell research 1987
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Hamster Monoclonal KRT8 Primary Antibody pour IHC (fro), IF - ABIN112171
Bártek, Vojt?sek, Stasková, Bártková, Kerekés, Rejthar, Kovarík: A series of 14 new monoclonal antibodies to keratins: characterization and value in diagnostic histopathology. dans The Journal of pathology 1991
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Human Polyclonal KRT8 Primary Antibody pour IF, IHC (p) - ABIN197582
Nakamichi, Hatakeyama, Nakayama: Formation of Mallory body-like inclusions and cell death induced by deregulated expression of keratin 18. dans Molecular biology of the cell 2002
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Human Monoclonal KRT8 Primary Antibody pour FACS, IHC (fro) - ABIN112350
Guelstein, Tchypysheva, Ermilova, Litvinova, Troyanovsky, Bannikov: Monoclonal antibody mapping of keratins 8 and 17 and of vimentin in normal human mammary gland, benign tumors, dysplasias and breast cancer. dans International journal of cancer. Journal international du cancer 1988
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Human Polyclonal KRT8 Primary Antibody pour IF (cc), IF (p) - ABIN737751
Zhang, Li, Fu, Li: Calcitonin is expressed in the submaxillary glands of rats. dans Bosnian journal of basic medical sciences / Udruženje basi?nih mediciniskih znanosti = Association of Basic Medical Sciences 2014
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Human Monoclonal KRT8 Primary Antibody pour IHC (fro), IHC (p) - ABIN114674
Waseem, Karsten, Leigh, Purkis, Waseem, Lane: Conformational changes in the rod domain of human keratin 8 following heterotypic association with keratin 18 and its implication for filament stability. dans Biochemistry 2004
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Cow (Bovine) Polyclonal KRT8 Primary Antibody pour IHC, WB - ABIN2777113
Moon, Yau, Wright, Zahradka: Injury-induced expression of cytokeratins 8 and 18 by vascular smooth muscle cells requires concurrent activation of cytoskeletal and growth factor receptors. dans Canadian journal of physiology and pharmacology 2008
Cow (Bovine) Polyclonal KRT8 Primary Antibody pour IHC, WB - ABIN2777112
Oh, Yang, Hahn, Kim, Byun, Jeon, Kim, Song, Noh, Kim, Yoo, Kim, Kim: Transcriptome analysis of human gastric cancer. dans Mammalian genome : official journal of the International Mammalian Genome Society 2005
Loss of activating transcription factor 3 (ATF3 (Montrer ATF3 Anticorps)) in knockout mice promotes the emergence of keratins CK5 (Montrer KRT5 Anticorps)+CK8+ epithelial cells.
Data show that the filament elongation of both desmin (Montrer DES Anticorps) and keratin K8/K18 (Montrer KRT18 Anticorps) proceeds very similar to that of vimentin (Montrer VIM Anticorps).
New insights into interactions between the nucleotide-binding domain of CFTR (Montrer CFTR Anticorps) and keratin 8.
We present results of a multicentre study measuring UBC (Montrer RPS27A Anticorps)((R)) Rapid Test(test that detects fragments of cytokeratins 8 and 18 in urine) in bladder cancer patients and healthy controls with focus on carcinoma in situ (CIS (Montrer CISH Anticorps)) and high-grade bladder cancer.
Data show that after siRNA transfection, TGF-beta1 (Montrer TGFB1 Anticorps) protein level was decreased, and CK18 (Montrer KRT18 Anticorps) proteins was decreased, while CK8 proteins was increased, and TERT (Montrer TERT Anticorps) protein expression was slightly increased at 96 h.
The interplay between Solo protein and keratin 8/keratin 18 filaments plays a crucial role in tensile force-induced RhoA activation and consequent actin cytoskeletal reinforcement in endothelial cells.
K8/18 filaments provide resistance to apoptosis in granulosa cell tumor cells by impairing FAS (Montrer FAS Anticorps) expression.
These metastatic tumors revealed no detectable expression of CK8/18, E-cadherin (Montrer CDH1 Anticorps), VCAM-1 (Montrer VCAM1 Anticorps), and ICAM-1 (Montrer ICAM1 Anticorps)
Data show that loss of epithelial membrane protein 2 (EMP2) is involved in sphingosylphosphorylcholine (SPC (Montrer UCN3 Anticorps))-induced phosphorylation of keratin 8 (K8) via ubiquitination of protein phosphatase 2 (PP2A (Montrer PPP2R4 Anticorps)) through alpha4 phosphoprotein (Montrer IGBP1 Anticorps) by caveolin-1 (cav-1 (Montrer CAV1 Anticorps)).
In human failing myocardium, where TNF-alpha (Montrer TNF Anticorps) expression is upregulated, K8/K18 (Montrer KRT18 Anticorps) were also ectopically expressed
This study reveals clear genetic-statistical evidence for a link of KRT8, FAF1 (Montrer FAF1 Anticorps) and PTH1R (Montrer PTH1R Anticorps) with some of leg weakness related traits in pigs.
Mechanical injury of vascular smooth muscle up-regulated keratin 8.
Results did not support that K8/K18 (Montrer KRT18 Anticorps) filaments influence the expression of Fas (Montrer FAS Anticorps) on the surface of luteal cells.
K8/K18 (Montrer KRT18 Anticorps)-dependent PKCdelta (Montrer PKCd Anticorps)- and ASMase (Montrer SMPD1 Anticorps)-mediated modulation of lipid raft size can explain the more prominent FasR-mediated signaling resulting from K8/K18 (Montrer KRT18 Anticorps) loss.
DRA mRNA levels were decreased by three- to fourfold and DRA protein was almost entirely lost in K8-/- cecum and proximal and distal colon compared with K8+/+.
Upregulation of IL-22 (Montrer IL22 Anticorps) in combination with a complete loss of its negative regulator IL-22BP (Montrer IL22RA2 Anticorps), and increased downstream STAT3 (Montrer STAT3 Anticorps)-signaling in K8(-/-) and K8(-/-)Apc (Montrer APC Anticorps)(Min/+) colonic epithelia confirmed that the IL-22 (Montrer IL22 Anticorps) pathway, important in inflammation, proliferation and tissue regeneration
Krt8 deletion (Krt8(-/-)) in a mouse model of cystic fibrosis (Montrer S100A8 Anticorps) (F508del-Cftr (Montrer CFTR Anticorps) mice) increased the levels of circulating markers of bone formation, corrected the expression of osteoblast phenotypic genes, promoted trabecular bone formation and improved bone mass and microarchitecture. Krt8 deletion corrected overactive NF-kappaB (Montrer NFKB1 Anticorps) signaling & decreased Wnt (Montrer WNT2 Anticorps)-beta-catenin (Montrer CTNNB1 Anticorps) signaling induced by F508del-Cftr (Montrer CFTR Anticorps) mutation in osteoblasts.
Autoantibodies to several antigens were identified in 81% of K8-null male mice 8 mo or older. Similar autoantibodies were detected in aging K18 (Montrer KRT18 Anticorps)-null male mice that had a related liver phenotype but normal colon compared with K8-null mice
The regulation of the SCFA-MCT1 (Montrer MCTS1 Anticorps)-HMGCS2 (Montrer HMGCS2 Anticorps) axis is disrupted in K8(-/-) colonocytes, suggesting a role for keratins in colonocyte energy metabolism and homeostasis.
In several liver stress models epiplakin and K8 genes displayed identical expression patterns and transgenic K8 overexpression resulted in elevated hepatic epiplakin levels.
The findings demonstrate the near complete loss of K8/K18 (Montrer KRT18 Anticorps) with concomitant high levels of vimentin (Montrer VIM Anticorps) in CT26 (Montrer DDX53 Anticorps) cells, a chemically-induced mouse colonic tumor.
Directed expression of a chimeric type II keratin (Montrer KRT80 Anticorps) partially rescues keratin 5 (Montrer KRT36 Anticorps)-null mice.
Data identify Danio rerio keratins 8/18 as the true orthologs of the human keratin pair 8/18.
This gene is a member of the type II keratin family clustered on the long arm of chromosome 12. Type I and type II keratins heteropolymerize to form intermediate-sized filaments in the cytoplasm of epithelial cells. The product of this gene typically dimerizes with keratin 18 to form an intermediate filament in simple single-layered epithelial cells. This protein plays a role in maintaining cellular structural integrity and also functions in signal transduction and cellular differentiation. Mutations in this gene cause cryptogenic cirrhosis. Alternatively spliced transcript variants have been found for this gene.
, keratin complex 2, basic, gene 5
, cytokeratin 8
, keratin, type II cytoskeletal 8
, type-II keratin Kb8
, cytokeratin endo A
, keratin complex 2, basic, gene 8
, keratin, type 2, gene 8