GTPase NRas anticorps
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- Antigène Voir toutes GTPase NRas (NRAS) Anticorps
- GTPase NRas (NRAS)
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Reactivité
- Humain
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Hôte
- Souris
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Clonalité
- Monoclonal
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Conjugué
- Cet anticorp GTPase NRas est non-conjugé
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Application
- Western Blotting (WB), Enzyme Immunoassay (EIA)
- Specificité
- The antibody recognizes human RAS. Other species not tested.
- Purification
- Protein-G affinity chromatography
- Clone
- AT2G9
- Isotype
- IgG2a
- Top Product
- Discover our top product NRAS Anticorps primaire
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- Indications d'application
- Optimal working dilution should be determined by the investigator.
- Restrictions
- For Research Use only
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- Format
- Liquid
- Concentration
- 1.0 mg/mL
- Buffer
- PBS, pH 7.4, containing 0.09 % sodium azide
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
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- Antigène
- GTPase NRas (NRAS)
- Autre désignation
- NRAS (NRAS Produits)
- Synonymes
- anticorps NRAS, anticorps AV095280, anticorps N-ras, anticorps ALPS4, anticorps NRAS1, anticorps NS6, anticorps N-RAS, anticorps fj99a10, anticorps wu:fj99a10, anticorps zgc:100761, anticorps zras-b1, anticorps H-RAS, anticorps HRAS, anticorps GTPase NRas, anticorps neuroblastoma RAS viral (v-ras) oncogene homolog L homeolog, anticorps NRAS proto-oncogene, GTPase, anticorps neuroblastoma ras oncogene, anticorps neuroblastoma RAS viral oncogene homolog, anticorps Tsp_01117, anticorps nras.L, anticorps Nras, anticorps NRAS, anticorps nras
- Sujet
- The Ras protein is a subfamily of small GTPases that are involved in signal transduction. They are activated by ligand-binding receptor tyrosine kinases such as EGFR, PDGFR, CSF and FGF. These kinases transiently convert RAS-GDP (inactive form) to RAS-GTP, the active form of RAS. The main genes are HRAS, NRAS and KRAS.Synonyms: HRAS, HRAS1, KRAS, KRAS2, NRAS, RASK2
- ID gène
- 4893
- Pathways
- Signalisation p53, Signalisation MAPK, Signalisation RTK, Fc-epsilon Receptor Signaling Pathway, EGFR Signaling Pathway, Neurotrophin Signaling Pathway, Hepatitis C, Regulation of long-term Neuronal Synaptic Plasticity, VEGF Signaling
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