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anti-Human Claudin 1 Anticorps:
anti-Mouse (Murine) Claudin 1 Anticorps:
anti-Rat (Rattus) Claudin 1 Anticorps:
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Human Monoclonal Claudin 1 Primary Antibody pour IHC (p), IP - ABIN563931
Mee, Grove, Harris, Hu, Balfe, McKeating: Effect of cell polarization on hepatitis C virus entry. dans Journal of virology 2007
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Human Polyclonal Claudin 1 Primary Antibody pour WB - ABIN2801934
Halford, Spencer, Greenwood, Winton, Hunt, Adamson: Assignment of claudin-1 (CLDN1) to human chromosome 3q28-->q29 with somatic cell hybrids. dans Cytogenetics and cell genetics 2000
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Human Polyclonal Claudin 1 Primary Antibody pour ELISA, ICC - ABIN4298947
Dietze, Shihan, Stammler, Konrad, Scheiner-Bobis: Cardiotonic steroid ouabain stimulates expression of blood-testis barrier proteins claudin-1 and -11 and formation of tight junctions in Sertoli cells. dans Molecular and cellular endocrinology 2015
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Human Monoclonal Claudin 1 Primary Antibody pour CyTOF, FACS - ABIN4898991
Farquhar, Hu, Harris, Davis, Brimacombe, Fletcher, Baumert, Rappoport, Balfe, McKeating: Hepatitis C virus induces CD81 and claudin-1 endocytosis. dans Journal of virology 2012
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Cow (Bovine) Polyclonal Claudin 1 Primary Antibody pour IHC, WB - ABIN2778045
Coyne, Gambling, Boucher, Carson, Johnson: Role of claudin interactions in airway tight junctional permeability. dans American journal of physiology. Lung cellular and molecular physiology 2003
Human Polyclonal Claudin 1 Primary Antibody pour IF (p), IHC (p) - ABIN686407
Wang, Chen, Liang, Yan, Lin, Liu, Luo, Huang, Li, Liu, Tang: Notch inhibition promotes fetal liver stem/progenitor cells differentiation into hepatocytes via the inhibition of HNF-1?. dans Cell and tissue research 2014
Human Monoclonal Claudin 1 Primary Antibody pour FACS - ABIN4895295
Giugliano, Petroff, Warren, Jasti, Linscheid, Ward, Kramer, Dobrinskikh, Sheiko, Gale, Golden-Mason, Winn, Rosen: Hepatitis C Virus Sensing by Human Trophoblasts Induces Innate Immune Responses and Recruitment of Maternal NK Cells: Potential Implications for Limiting Vertical Transmission. dans Journal of immunology (Baltimore, Md. : 1950) 2015
Tumor necrosis factor-alpha (Montrer TNF Anticorps) increases claudin-1, 4, and 7 expression in renal tubular cells, altering permeability and transepithelial electrical resistance.
Cyclosporine A/sirolimus alter claudin-1 expression in renal proximal tubular cells via ERK1/2 (Montrer MAPK1/3 Anticorps) signaling pathway to alter barrier function.
Data show that miR-30a could bind to the 3'-untranslted region of Slug mRNA and increased expression of claudins, a family of tight junction transmembrane proteins.
These findings indicate a previously unrecognized mechanism that miR (Montrer MLXIP Anticorps)-142-5p, targeting CLDN1, plays an important role in Hashimoto's thyroiditis pathogenesis.
Here, the authors identified an interaction between claudin-1 (Montrer CLDN7 Anticorps) and Sec24C (Montrer SEC24C Anticorps), a cargo-sorting component of the coat protein (Montrer GOLPH3 Anticorps) complex II (COPII) vesicular transport system.
Aberrant expression of the tight junction molecules claudin-1 (Montrer CLDN7 Anticorps) and zonula occludens-1 (Montrer TJP1 Anticorps) mediates cell growth and invasion in oral squamous cell carcinoma cells.
Increased expression of intestinal epithelial claudin-1 (Montrer CLDN7 Anticorps) with downregulation of claudin-3 (Montrer CLDN3 Anticorps) has been observed in intestinal inflammatory disorders.
Studies indicate claudin 1 (CLDN-1) as a target for improving epidermal drug absorption and preventing HCV infection and of claudin 4 (CLDN-4 (Montrer CLDN4 Anticorps)) as a target for anticancer therapeutics.
Data show that the charge of Lys65 in claudin 1 (Cldn1) and Glu158 in claudin 3 (Cldn3 (Montrer CLDN3 Anticorps)), and of Gln57 in claudin 5 (Cldn5 (Montrer CLDN5 Anticorps)) are necessary for tight junction (TJ) strand formation.
Studies indicate that claudin-1 (Montrer CLDN7 Anticorps) accumulate in early endosomes in both epithelial and endothelial cells.
These results revealed that CLDN1 contributed to cancer stem cell features of hepatocellular carcinoma, which was altered by TMPRSS4 (Montrer TMPRSS4 Anticorps) expression via ERK1/2 (Montrer MAPK1/3 Anticorps) signaling pathway, providing promising targets for novel specific therapies.
Data suggest that epithelial-mesenchymal transition (EMT (Montrer ITK Anticorps)) is regulated by ZMYND8 (Montrer ZMYND8 Anticorps) (receptor for activated protein kinase C (Montrer GNB2L1 Anticorps)) which selectively activates gene promoters of CLDN1 (claudin 1) and CDH1 (E-cadherin (Montrer CDH1 Anticorps)) in breast cancer cells; thus, the presence of ZMYND8 (Montrer ZMYND8 Anticorps) could be implicated in maintaining epithelial phenotype of cells; ZMYND8 (Montrer ZMYND8 Anticorps) regulates invasion/migration of breast cancer cells.
Taken together, these results demonstrate that BTZ (Montrer CASC3 Anticorps)-induced claudin 1 expression may be a valuable therapeutic approach for AD.
Results suggest that TLR4 (Montrer TLR4 Anticorps)-dependent claudin-1 internalization and secondary anion secretion contribute to irinotecan-induced diarrhea.
The data suggested that miR (Montrer MLXIP Anticorps)-29a may regulate tumor growth and migration by targeting CLDN1.
Data show that the spatiotemporal expression of claudin-1 is dysregulated in homeobox (Montrer PRRX1 Anticorps) (Msx) genes Msx1d/d/Msx2d/d uteri.
Cldn-1 is a positive regulator of osteoblast proliferation and differentiation.
We further demonstrate that KRT76 interacts with CLDN1 and propose that this interaction is necessary to correctly position CLDN1 in tight junctions.
MIR29 targets and reduces expression of CLDN1 and NKRF (Montrer NKRF Anticorps) to increase intestinal permeability in inflammatory bowel disease.
Occludin (Montrer OCLN Anticorps) and Claudin-1 expressions in the large intestine are under the circadian control, which is associated with temporal regulation of colonic permeability and also susceptibility to colitis.
regulates intestinal epithelial homeostasis via regulation of Notch (Montrer NOTCH1 Anticorps)-signalling
Downregulation of Sirt1 (Montrer SIRT1 Anticorps) and upregulation of the tight junction protein Claudin-1 by SIRT1 (Montrer SIRT1 Anticorps)-mediated epigenetic regulation in podocytes contributed to albuminuria.
Tight junctions represent one mode of cell-to-cell adhesion in epithelial or endothelial cell sheets, forming continuous seals around cells and serving as a physical barrier to prevent solutes and water from passing freely through the paracellular space. These junctions are comprised of sets of continuous networking strands in the outwardly facing cytoplasmic leaflet, with complementary grooves in the inwardly facing extracytoplasmic leaflet. The protein encoded by this gene, a member of the claudin family, is an integral membrane protein and a component of tight junction strands. Loss of function mutations result in neonatal ichthyosis-sclerosing cholangitis syndrome.
, claudin 19
, claudin 1
, senescence-associated epithelial membrane protein 1
, tight junction protein