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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN411305
Yu, Wang, Yang: Lactobacillus amylophilus D14 protects tight junction from enteropathogenic bacteria damage in Caco-2 cells. dans Journal of dairy science 2012
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN625029
Kraus, Winter, Jepsen, Jäger, Meyer, Deschner: Interactions of adiponectin and lipopolysaccharide from Porphyromonas gingivalis on human oral epithelial cells. dans PLoS ONE 2012
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN2690568
Gimbrone, Obin, Brock, Luis, Hass, Hébert, Yip, Leung, Lowe, Kohr: Endothelial interleukin-8: a novel inhibitor of leukocyte-endothelial interactions. dans Science (New York, N.Y.) 1990
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN414887
Sun, Wang, Yu, Hu: Serial cytokine levels during wound healing in rabbit maxillary sinus mucosa. dans Acta oto-laryngologica 2010
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Cow (Bovine) Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN414016
Jiemtaweeboon, Shirasuna, Nitta, Kobayashi, Schuberth, Shimizu, Miyamoto: Evidence that polymorphonuclear neutrophils infiltrate into the developing corpus luteum and promote angiogenesis with interleukin-8 in the cow. dans Reproductive biology and endocrinology : RB&E 2011
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Rabbit Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN415998
Miroshnikov, Reznikov, Kokhanov: [Comparative evaluation of the action of serum proteins on tissue regeneration]. dans Biulleten' eksperimental'no? biologii i meditsiny 1987
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN365164
Zhang, Wu, Qu, Bai, Merrilees, Black: Pro-inflammatory phenotype of COPD fibroblasts not compatible with repair in COPD lung. dans Journal of cellular and molecular medicine 2012
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN612714
Harada, Mukaida, Matsushima: Interleukin 8 as a novel target for intervention therapy in acute inflammatory diseases. dans Molecular medicine today 1997
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Human Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN625030
Grbic, Degagné, Langlois, Dupuis, Gendron: Intestinal inflammation increases the expression of the P2Y6 receptor on epithelial cells and the release of CXC chemokine ligand 8 by UDP. dans Journal of immunology (Baltimore, Md. : 1950) 2008
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Rabbit Interleukin 8 Kit ELISA pour Sandwich ELISA - ABIN367974
Miyata, Bai, Vincent, Sin, Van Eeden: Statins reduce ambient particulate matter-induced lung inflammation by promoting the clearance of particulate matter, < 10 ?m from lung tissues. dans Chest 2013
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gene expression and epistasis analysis indicated that CXCL8(IL-8) was a functional target of vitamin D3-mediated HSPC (Montrer PSMA7 Kits ELISA) regulation. Together, these findings highlight the relevance of developmental 1,25(OH)D3 availability for definitive hematopoiesis and suggest potential therapeutic utility in HSPC (Montrer PSMA7 Kits ELISA) expansion
Targeted expression of the human oncogene (Montrer RAB1A Kits ELISA) KRASG12V in zebrafish gills induces inflammation and cxcl8-l1 expression.
Data show that both Cxcl8-l1 and Cxcl8-l2 are required for larva survival upon Salmonella Typhimurium infection.
results explain how early H2O2 signal regulates neutrophil recruitment at all phases, directly via Lyn (Montrer LYN Kits ELISA) oxidation or indirectly by modulating cxcl8 gene expression
The data identify HOXC9 (Montrer HOXC9 Kits ELISA) as an endothelial cell active transcriptional repressor promoting the resting, antiangiogenic endothelial cell phenotype in an interleukin 8-dependent manner
Here, we report the characterisation of genes encoding the zebrafish Cxcl8, Cxcr1 (Montrer CXCR1 Kits ELISA) and Cxcr2 (Montrer CXCR2 Kits ELISA)
this study shows that ponciretin may attenuate ethanol-induced gastritis via the regulation of IL-8 secretion
Adh (Montrer AVP Kits ELISA) binds to OR5M11 (Montrer OR5M11 Kits ELISA), which enhances Actinobacillus pleuropneumoniae pathogenicity by activating p38 (Montrer CRK Kits ELISA) which induces apoptosis of PAMs and IL-8 release
Findings suggest that IL8-dependent osteoclast activation may constitute an early event in the initiation of the joint specific inflammation in anti-citrullinated protein-positive rheumatoid arthritis.
Data suggest that CXCL1 (Montrer CXCL1 Kits ELISA)/IL-8, released from osteoclasts in an autoantibody-dependent manner, produces pain by activating sensory neurons.
IL-8 signaling is up-regulated in alcoholic hepatitis.
expressions of IL-1beta (Montrer IL1B Kits ELISA) and IL-8 in the brain increased after ApoE (Montrer APOE Kits ELISA) knockout in mice
CYLD (Montrer CYLD Kits ELISA) negatively regulates nontypeable Haemophilus influenzae-induced IL-8 expression via MKP-1 (Montrer DUSP1 Kits ELISA)-dependent inhibition of ERK (Montrer EPHB2 Kits ELISA).
Taken together, these results indicate that CD147 promotes lung cancer-induced osteoclastogenesis by modulating IL-8 secretion, and suggest that CD147 is a potential therapeutic target for cancer-associated bone resorption in lung cancer patients.
these data outline a novel role for the P2Y6 receptor (Montrer P2RY6 Kits ELISA) in the induction of CXCL8/IL-8 production and barrier dysfunction in response to C. difficile toxin exposure and may provide a new therapeutic target for the treatment of CDI.
these data suggest that IL-8 plays an important role in breast cancer osteolysis
induction of MCL-1 (Montrer MCL1 Kits ELISA) by IL-6 (Montrer IL6 Kits ELISA)/IL-8 may surmount any direct down-regulation by miR (Montrer MLXIP Kits ELISA)-29b via its 3'-UTR (Montrer UTS2R Kits ELISA).
IL-6 (Montrer IL6 Kits ELISA)/8 expression in airway endothelial cellss simultaneously exposed to weakly acidic media and LPS (Montrer IRF6 Kits ELISA) for 4 h was reduced.
Diagnostic value of sTREM-1, IL-8, PCT (Montrer UROD Kits ELISA), and CRP (Montrer CRP Kits ELISA) in febrile neutropenia after autologous stem cell transplantation
PROX1 (Montrer PROX1 Kits ELISA) activated nuclear factor kappaB signaling and selectively promoted expression of the proangiogenic interleukin-8 by epigenetically stimulating the IL-8 promoter.
High IL8 expression is associated with alcoholic hepatitis.
data showed that HDAC1 (Montrer HDAC1 Kits ELISA) can trigger the proliferation and migration of breast cancer cells via activation of Snail (Montrer SNAI1 Kits ELISA)/IL-8 signals
ATP-induced IL-8 release maybe involved in the pathogenesis of refractory gastroesophageal re fl ux disease.
Data suggest that primary cells from papillary renal cell carcinoma secrete the chemokines IL8, CXCL16 (Montrer CXCL16 Kits ELISA), and chemerin (Montrer RARRES2 Kits ELISA); these chemokines attract primary human monocytes and induce shift/transdifferentiation in monocytes toward M2 macrophage/foam cell phenotype. (IL8 = interleukin-8; CXCL16 (Montrer CXCL16 Kits ELISA) = C-X-C motif chemokine (Montrer CCL1 Kits ELISA) ligand 16)
findings strongly suggest that P. gingivalis infection plays an important role in the promotion of the invasive potential of oral squamous cell carcinoma cells via the upregulation of IL-8 and MMPs.
miR (Montrer MLXIP Kits ELISA)-424-5p expression in oral squamous cell carcinoma cells could be induced by IL-8 through increasing STAT5 (Montrer STAT5A Kits ELISA) transcriptional activity.
Molecular characterization of the bovine IER3 (Montrer IER3 Kits ELISA) gene: Down-regulation of IL-8 by blocking NF-kappaB (Montrer NFKB1 Kits ELISA) activity mediated by IER3 (Montrer IER3 Kits ELISA) overexpression in MDBK cells infected with bovine viral diarrhea virus-1.
STA3 (Montrer ARHGEF3 Kits ELISA) facilitates TLR4 (Montrer TLR4 Kits ELISA)-dependent IL-6 (Montrer IL6 Kits ELISA) and IL-8 production via IL-6 (Montrer IL6 Kits ELISA) receptor-positive feedback in endometrial cells.
As a pilot study, the present results revealed that identified SNPs in IL8 and TLR4 (Montrer TLR4 Kits ELISA) genes can be used as a genetic marker and predisposing factor for resistance/susceptibility to digital dermatitis in dairy cows. However, TLR4 (Montrer TLR4 Kits ELISA) gene may be a potential candidate for such disease.
Blood levels of cortisol, immunoglobulin G, and IL-8 in relationship to genetics and the incidence of bovine respiratory complex disease are reported.
Genetic variation at the IL8 locus explains a proportion of the inter-breed and inter-individual variation in immunity between neonatal calves which is likely to influence their resistance to infection.
In response to C. abortus infection, both ovine and bovine turbinate cells produce CXCL8 mRNA and protein late in the bacterial developmental cycle.
Studied association of IL8 -105G/A with mastitis somatic cell score in Chinese Holstein dairy cows. Results suggest genotype GG may be a useful genetic marker for mastitis resistance selection and breeding in Chinese Holstein dairy cows.
Data implicate a complex cascade of events during luteolysis, involving Il8 signaling, neutrophil recruitment, and immune cell action within the corpus luteum.
Cadmium exposure induced pulmonary inflammation and increased lung interleukin 8 secretion.
Exposure to follicular fluid transiently increased the transcript levels of IL8 and PTGS2 (Montrer PTGS2 Kits ELISA), and decreased the expression of SOD2 (Montrer SOD2 Kits ELISA), GPX3 (Montrer GPX3 Kits ELISA), DAB2 (Montrer DAB2 Kits ELISA), and NR3C1 (Montrer NR3C1 Kits ELISA). TNF (Montrer TNF Kits ELISA) and IL6 (Montrer IL6 Kits ELISA) levels were also decreased while those of NAMPT (Montrer NAMPT Kits ELISA) were unaffected.
The JNK (Montrer MAPK8 Kits ELISA)-activated AP-1 (Montrer JUN Kits ELISA) subunit c-Jun (Montrer JUN Kits ELISA) was critical for the up-regulation of IL-8 expression by PRRSV
These results suggest that porcine circovirus 2 induces IL-8 secretion via the TLR2 (Montrer TLR2 Kits ELISA)/MyD88 (Montrer MYD88 Kits ELISA)/NF-kappaB (Montrer NFKB1 Kits ELISA) signalling pathway.
Facilitated degradation of the RGS16 (Montrer RGS16 Kits ELISA) by porcine circovirus type 2 ORF3 (Montrer ASZ1 Kits ELISA) further enhances NFkappaB translocation into the nucleus through the ERK1/2 (Montrer MAPK1/3 Kits ELISA) signalling pathway and increased IL-6 (Montrer IL6 Kits ELISA) and IL-8 mRNA transcripts.
Suggest that the interplay between TGFbeta (Montrer TGFB1 Kits ELISA)-2 and LPS (Montrer IRF6 Kits ELISA) regulates the levels of IL-8 in the immature newborn intestine.
Data suggest that MSK1 (Montrer RPS6KA5 Kits ELISA) and MSK2 are the major CREB (Montrer CREB1 Kits ELISA) kinases in fibroblast-like synoviocytes from rheumatoid arthritis patients stimulated with lysophosphatidic acid and that phosphorylation of CREB1 (Montrer CREB1 Kits ELISA) at Ser (Montrer SIGLEC1 Kits ELISA)-133 plays a significant role in IL-8 production.
Study presents evidence demonstrating a single species of exotoxin ApxI, derived from A. pleuropneumoniae serotype 10, induces the expression and production of proinflammatory cytokines IL-1beta (Montrer IL1B Kits ELISA), IL-8 and TNF-alpha (Montrer TNF Kits ELISA) in porcine alveolar macrophages.
local expression of IL-1beta (Montrer IL1B Kits ELISA) and IL-8 in non-bacterial thrombotic endocarditis, Staphylococcus aureus infective endocarditis, animals with S. aureus sepsis without endocarditis and controls
In swine, IL-8, TNF-ALPHA (Montrer TNF Kits ELISA), INOS (Montrer NOS2 Kits ELISA) AND MIP-1BETA (Montrer CCL4 Kits ELISA) were increased during mechanical ventilation in a time-related fashion.
Classical swine fever virus NS2 protein promotes interleukin-8 expression.
Developmental changes in IL-8 isoforms may serve to minimize its inflammatory effects in the fetus and also provide a mechanism to restore its full activity after birth
At the 2-wk time point, interleukin 8 gene expression was comparable in intervertebral discs injected with chondrocytes in controls, whereas its expression in IVDs injected with saline increased 50-fold
Data show that the serum levels of mast cell tryptase, monocyte chemoattractant protein-1 (MCP-1 (Montrer CCL2 Kits ELISA)) and interleukin-8 (IL-8) decreased significantly in Dachengqi decoction treatment group.
The JNK (Montrer MAPK8 Kits ELISA) pathway plays an important role in mechanical ventilation-stimulated TNF-alpha (Montrer TNF Kits ELISA) expression in alveolar macrophages, but the injury-stimulated IL-8 expression may be regulated by other signaling pathways.
IL-8 and IL-1ra (Montrer IL1RN Kits ELISA) showed correlation with the severity of colitis. These observations should significantly further understandings on the role of neutrophils and monocytes in the immunopathogenesis of ulcerative colitis
Results suggested that chemokine (Montrer CCL1 Kits ELISA) expression by cultured equine BECs following exposure to pulmonary hemorrhage conditions may contribute to the development of inflammatory airway disease in horses.
Mapped six genes (EIF4G3, HSP90 (Montrer HSP90AB1 Kits ELISA), RBBP6 (Montrer RBBP6 Kits ELISA), IL8, TERT (Montrer TERT Kits ELISA), and TERC) on the chromosomes of Equus caballus, Equus asinus, Equus grevyi, and Equus burchelli by fluorescence in situ hybridization.
This study examined effects of in vitro exposure to solutions of hay (Montrer GTF2H5 Kits ELISA) dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
These results suggest that MMP-1 (Montrer MMP1 Kits ELISA) and IL-8 are both involved in the exercise-induced stress response, and this represents a starting point from which to understand the adaptive responses to this phenomenon.
Stress decreased IL-8 gene expression in both CeA (Montrer CEACAM3 Kits ELISA) lesioned and non-lesioned animals. But, CeA (Montrer CEACAM3 Kits ELISA) lesions increased the tissue expression of IL-8 mRNA prior to and after stress exposure.
Duck IL-8 is good immunostimulant to enhance the immune efficiency of avian influenza vaccine in ducks.
The protein encoded by this gene is a member of the CXC chemokine family. This chemokine is one of the major mediators of the inflammatory response. This chemokine is secreted by several cell types. It functions as a chemoattractant, and is also a potent angiogenic factor. This gene is believed to play a role in the pathogenesis of bronchiolitis, a common respiratory tract disease caused by viral infection. This gene and other ten members of the CXC chemokine gene family form a chemokine gene cluster in a region mapped to chromosome 4q.
CXC chemokine K60
, interleukin 8
, C-X-C motif chemokine 15
, small inducible cytokine subfamily B, member 15
, small-inducible cytokine B15
, T-cell chemotactic factor
, alveolar macrophage chemotactic factor I
, beta endothelial cell-derived neutrophil activating peptide
, beta-thromboglobulin-like protein
, chemokine (C-X-C motif) ligand 8
, granulocyte chemotactic protein 1
, lung giant cell carcinoma-derived chemotactic protein
, lymphocyte derived neutrophil activating peptide
, monocyte-derived neutrophil chemotactic factor
, monocyte-derived neutrophil-activating peptide
, neutrophil-activating peptide 1
, small inducible cytokine subfamily B, member 8
, tumor necrosis factor-induced gene 1
, C-X-C motif chemokine 8
, neutrophil activating peptide 1
, alveolar macrophage-derived chemotactic factor-I
, neutrophil attractant/activation protein 1
, neutrophil attractant/activation protein-1
, permeability factor 1
, Interleukin-8-like protein
, Neutrophil attractant protein 1
, neutrophil attractant protein-1
, CEF-4/9E3 cytokine
, RSV-induced protein
, embryo fibroblast protein 1
, putatitve interleukin-8
, transformation-induced protein