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anti-Human FSHR Anticorps:
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Human Polyclonal FSHR Primary Antibody pour IF (p), IHC (p) - ABIN729508
Luo, Yin, Zhang, Yuan, Zhao, Luan, Zhang: Role of SDF-1/CXCR4 and cytokines in the development of ovary injury in chemotherapy drug induced premature ovarian failure mice. dans Life sciences 2017
Human Polyclonal FSHR Primary Antibody pour ELISA, WB - ABIN560952
Ritter, Thuering, Saint Mezard, Luong-Nguyen, Seltenmeyer, Junker, Fournier, Susa, Morvan: Follicle-stimulating hormone does not impact male bone mass in vivo or human male osteoclasts in vitro. dans Calcified tissue international 2008
INVESTIGATION OF STAT5A (Montrer STAT5A Anticorps), FSHR AND LHR (Montrer LHCGR Anticorps) GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
The expression of FSHR mRNA in granulosa cells was highest in small antral follicles, then decreased significantly as follicles increased in size, and was lowest in cysts.
transfer. We conclude that variation at these loci of the FSHR gene has no significant effect on pregnancy rates in Luxi cattle.
Specific alleles of the bovine FSHR gene are associated with variations in embryo yield and in the number of unfertilised oocytes.
This study evaluated the relationships among aromatase (Montrer CYP19A1 Anticorps), IGF-1 (Montrer IGF1 Anticorps), IGF2R (Montrer IGF2R Anticorps), and follicle stimulating hormone (FSH) receptor levels expressed in ovarian follicles of cattle selected for twin pregnancies.
granulosa cell clustering is accompanied by marked increases in FSHr, IGF-1r (Montrer IGF1R Anticorps), and p450 arom (Montrer CYP19A1 Anticorps) expression, and precedes induction and subsequent peak E2 production
Dominant follicles experience a reduction in FSH (Montrer BRD2 Anticorps) dependence (diminished expression of FSHR), but acquire increased LH dependence (enhanced expression of LHCGR (Montrer LHCGR Anticorps)) as they grow during the low FSH (Montrer BRD2 Anticorps) milieu of follicular waves.
FSHR is specifically regulated through androgen receptor (Montrer AR Anticorps) in granulosa cells
Heterozygous heifers showed a higher pregnancy rate (67 and 66% for LHR (Montrer LHCGR Anticorps) and FSHR genes, respectively), but no significant effects were observed for the genes studied (
Data show that double mutation of follicle-stimulating hormone receptor (fshr) and luteinizing hormone receptor (lhcgr (Montrer LHCGR Anticorps)) resulted in infertile males.
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr and lhcgr (Montrer LHCGR Anticorps).
Characterization of the first functional zebrafish (Danio rerio) gonadotropic hormone I receptor (follicle stimulating hormone receptor).
The mutation p.R59X in FSHR is causative for primary ovarian insufficiency by means of arresting folliculogenesis.
two mutations, V(221)G and T(449)N, in the extracellular domain and transmembrane helix 3, of FSHR, respectively, are reported.
The reduced fertilisation and pregnancy rate was associated with a lower LH receptor (Montrer LHCGR Anticorps) density and a lack of essential down-regulation of the FSH (Montrer BRD2 Anticorps) and LH receptor (Montrer LHCGR Anticorps).
This work demonstrates that the expression of FSHR and LHCGR (Montrer LHCGR Anticorps) can be induced in hGL5 cells but that the FSHR-dependent cAMP/PKA pathway is constitutively silenced, possibly to protect cells from FSHR-cAMP-PKA-induced apoptosis.
The incidence of the Ser/Ser (Montrer SIGLEC1 Anticorps) genotype was higher in patients with higher recombinant human follicle-stimulating hormone consumption. Based on our results, we hypothesize an association between the follicle-stimulating hormone receptor polymorphisms and a "hyporesponse" to exogenous follicle-stimulating hormone.
The data suggest novel follicle-stimulating hormone receptor expression in endometriotic lesions, qualitatively and quantitatively different from that of normal endometrium.
Genetic variation affecting FSH (Montrer BRD2 Anticorps) production (FSHB (Montrer FSHB Anticorps) c.-211G>T) was associated with age at pubertal onset, as assessed by testicular enlargement. The effect appeared further modified by coexistence of genetic variation affecting FSH (Montrer BRD2 Anticorps) sensitivity (FSHR c.-29G>A).
Novel mutations, c.419delA and c.1510C>T of the FSHR gene were associated with resistant ovarian syndrome.
Association of the FSHR G-29A, 919A > G, 2039A > G polymorphisms with male infertility in Han-Chinese
A polymorphism within the promoter of FSHR is determined to not be associated with ovarian reserve or response to controlled ovarian hyperstimulation.
Study demonstrates expression of follicle-stimulating hormone receptor (FSHR) and a direct action of follicle-stimulating hormone on testicular stem/germ cells possibly mediated via alternatively spliced growth factor type 1 receptor FSHR3 in mice.
FSHR and LHR (Montrer LHCGR Anticorps) proteins are significantly upregulated in CCs (Montrer CCS Anticorps) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (Montrer ESR1 Anticorps), LHR (Montrer LHCGR Anticorps), and FSHR
Brca1 (Montrer BRCA1 Anticorps)(GC-/-) models reveal that specific intra-follicular Brca1 (Montrer BRCA1 Anticorps) loss alone, or combined with cancer-promoting genetic (Trp53 (Montrer TP53 Anticorps) loss) and endocrine (high serum follicle-stimulating hormone) changes, was not sufficient to cause ovarian tumors.
Data (including date from knockout mice) suggest that Fshr is expressed early in pregnany in placenta and other extragonadal tissues of fetoplacental unit; expression is particularly strong at term.
Sertoli cell-specific expression of MTA2 (Montrer MTA2 Anticorps) is required for transcriptional regulation of FSHR gene during spermatogenesis.
By day 20 and in adult animals total AR or FSHR ablation significantly reduced Leydig cell numbers but Sertoli cell specific AR ablation had no effect.
The results demonstrate that gain-of-function mutations of the FSHR in mice bring about distinct and clear changes in ovarian function
haploinsufficiency of the follicle-stimulating hormone receptor accelerates oocyte loss inducing early reproductive senescence and biological aging in mice
show that FSH-R haploinsufficiency leads to a decrease in ovulation, altered ovarian steroidogenesis, and neuroendocrine impairments resulting in early reproductive senescence
The amount of dopamine d1 receptor (Montrer DRD1 Anticorps), dopamine D2 receptor (Montrer DRD2 Anticorps), and follicle stimulating hormone receptor (FSHr) mRNA were quantified in ovarian tissues in anestrous and mares expressing estrus during the breeding season are reported.
Activated TGF-beta (Montrer TGFB1 Anticorps) signaling rescued miR (Montrer MYLIP Anticorps)-143-reduced FSHR and intracellular signaling molecules, and miR (Montrer MYLIP Anticorps)-143-induced porcine granulosa cell apoptosis.
The results showed that polymorphisms in exon 10 of the FSHR gene had a significant effect on litter size traits of Wannan Black and Berkshire pigs. These results can be applied for marker-assisted selection in the 2 swine breeds
These results showed that an increased FSHR gene expression level was accompanied with an increase in histone H3K9 acetylation levels, suggesting that histone H3K9 acetylation could regulate the expression of the porcine FSHR gene.
248 F(2) animals from a Duroc and Meishan cross were genotyped for three FSHR SNPs at positions 74, 532 and 1166, and these were correlated with the phenotypes of litter size and corpus luteum number
findings suggest the FSH receptor may be involved in the early follicle formation in pigs, which begins during prenatal life.
FSHR is expressed in the hamster ovary starting from the fetal life to account for FSH (Montrer BRD2 Anticorps)-induced primordial follicle formation and cyclic AMP (Montrer TMPRSS5 Anticorps) production.
The protein encoded by this gene belongs to family 1 of G-protein coupled receptors. It is the receptor for follicle stimulating hormone and functions in gonad development. Mutations in this gene cause ovarian dysgenesis type 1, and also ovarian hyperstimulation syndrome. Alternative splicing results in multiple transcript variants.
follicle stimulating hormone receptor
, follicle-stimulating hormone receptor
, gonadotropic hormone I
, follicle-stimulating hormone receptor-like
, FSH receptor
, follitropin receptor