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MAD1 (Montrer MXD1 Kits ELISA) and Proto-Oncogene (Montrer RAB1A Kits ELISA) Proteins c-myc (Montrer MYC Kits ELISA) reciprocally regulate ribosomal DNA transcription, providing a mechanism for coordination of ribosome biogenesis and cell growth
MAD1 (Montrer MXD1 Kits ELISA) is present in mouse oocytes at all stages during the first meiosis and that it participates in spindle checkpoint during meiosis
Data show that the loss of Trrap (Montrer TRRAP Kits ELISA) leads to chromosome missegregation, mitotic exit failure and compromised mitotic checkpoints, which are caused by defective Trrap (Montrer TRRAP Kits ELISA)-mediated transcription of the mitotic checkpoint (Montrer BUB3 Kits ELISA) proteins Mad1 (Montrer MXD1 Kits ELISA) and Mad2 (Montrer MXI1 Kits ELISA).
identified mouse Telomeric Repeat Binding Factor 1 (Trf1 (Montrer TERF1 Kits ELISA)) as a protein that interacts directly with the spindle checkpoint protein Mad1 (Montrer MXD1 Kits ELISA) and the mitotic kinase Nek2 (Montrer NEK2 Kits ELISA)
Together these data demonstrate that the MYC (Montrer MYC Kits ELISA)-antagonist MAD1 (Montrer MXD1 Kits ELISA) and cyclin-dependent kinase inhibitor p27(Kip1 (Montrer CDKN1B Kits ELISA)) cooperate to regulate the self-renewal and differentiation of HSCs in a context-dependent manner.
the simultaneous knockdown of p18 (Montrer CDKN2C Kits ELISA), p27 (Montrer CDKN1B Kits ELISA) and MAD1 (Montrer MXD1 Kits ELISA) with a medium of only stem cell factor (Montrer KITLG Kits ELISA) can induce long-term culture-initiating cell expansion despite the loss of engraftment ability
This study showed that MAD1L1 as a susceptibility gene for both of these genetically overlapping disorders is associated with a decreased bottom-up responsiveness of the mesolimbic reward system and related cortical regions involved in the salience network as well as with reduced top-down control processes.
Results show that low DNA methylation (Montrer HELLS Kits ELISA) levels of LINC00682, MAD1L1, and LINE-2 was strongly correlated with hepatocellular carcinomas recurrence, patient disease-free survival, and/or overall survival.
MAD1L1 positive expression may be associated with tumour progression and metastasis in small-cell lung cancer (SCLC) and may thus serve as a new biomarker for prognosis in these patients.
In this review, we highlight a novel Mad1 role in chromosome alignment, which is the first conserved mechanism that links the spindle assembly checkpoint and kinesin-mediated chromosome gliding.
MAD1L1 Arg558His and MAD2L1 (Montrer MAD2L1 Kits ELISA) Leu84Met interaction with smoking increase the risk of colorectal cancer
MAD1L1 rs12666575 polymorphism may play a protective role against schizophrenia (SCZ) in the Chinese population. rs12666575 may be associated with general psychopathology and thought disturbance in SCZ patients.
We also show that replication perturbations result in relocalization of MAD1 (Montrer MXD1 Kits ELISA)/MAD2 (Montrer MAD2L1 Kits ELISA) in human cells, suggesting that the role of SAC (Montrer ADCY10 Kits ELISA) in DNA repair is conserved.
Mad1 (Montrer MXD1 Kits ELISA) has a role in secretion and cell migration.
MAD1 (Montrer MXD1 Kits ELISA) kinetochore localization dictates the spindle assembly checkpoint in metaphase.
This article reviews Mad1 (Montrer MXD1 Kits ELISA) and Mad2 (Montrer MAD2L1 Kits ELISA) - structural and functional relationship with implication in genetic diseases, specifically in cancer. [review]
MAD1L1 is a component of the mitotic spindle-assembly checkpoint that prevents the onset of anaphase until all chromosome are properly aligned at the metaphase plate. MAD1L1 functions as a homodimer and interacts with MAD2L1. MAD1L1 may play a role in cell cycle control and tumor suppression. Three transcript variants encoding the same protein have been found for this gene.
MAD1-like protein 1
, mitotic arrest deficient 1-like 1
, mitotic arrest deficient 1-like protein 1
, mitotic spindle assembly checkpoint protein MAD1
, mitotic checkpoint MAD1 protein homolog
, mitotic-arrest deficient 1, yeast, homolog-like 1
, tax-binding protein 181
, tumor protein p53 inducible protein 9