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Overexpression of ASL (Montrer ADSL Kits ELISA) may be a contributing factor in drug resistance for arginine deprivation therapy.
ASL (Montrer ADSL Kits ELISA)-targeting shRNA-induced growth inhibition is associated with decreased cyclin A2 (Montrer CCNA2 Kits ELISA) expression and Nitric oxide content in colon cancer.
the mechanism induced by ASL (Montrer ADSL Kits ELISA) shRNA which occurred in human breast cancer may be attributed to a decrease in cyclin A2 (Montrer CCNA2 Kits ELISA) and NO.
The clinical and biochemical course in variant forms of ASL (Montrer ADSL Kits ELISA) deficiency is associated with relevant residual levels of ASL (Montrer ADSL Kits ELISA) activity as well as instability of mutant ASL (Montrer ADSL Kits ELISA) proteins.
Point mutation of ASS1 (Montrer ASS1 Kits ELISA), ASL (Montrer ADSL Kits ELISA) and SLC25A13 (Montrer slc25a13 Kits ELISA) is associated with citrullinemia (Montrer ASS1 Kits ELISA).
Data show that in patients with Argininosuccinate lyase deficiency, the ASl (Montrer ADSL Kits ELISA) gene is subject to several mutations, the majority are missense; some more frequent then others.
Our results suggest that ASL (Montrer ADSL Kits ELISA) transcripts can contribute to the highly variable phenotype in ASA (Montrer ARSA Kits ELISA) patients if expressed at high levels.
Cox (Montrer COX8A Kits ELISA) regression analysis showed that ASL (Montrer ADSL Kits ELISA) is an independent prognostic marker for HCC (Montrer FAM126A Kits ELISA). Therefore, reduced ASL (Montrer ADSL Kits ELISA) expression may be a novel maker for poor prognosis in HCC (Montrer FAM126A Kits ELISA) patients
analysis of mutant argininosuccinate lyase in argininosuccinic aciduria
extent of protection of ASL (Montrer ADSL Kits ELISA) and delta-crystallin at different ratios of alphaA-crystallin (Montrer CRYAA Kits ELISA)
Enterocyte-derived ASL (Montrer ADSL Kits ELISA) has a protective role in necrotizing enterocolitis.
This gene encodes a member of the lyase 1 family. The encoded protein forms a cytosolic homotetramer and primarily catalyzes the reversible hydrolytic cleavage of argininosuccinate into arginine and fumarate, an essential step in the liver in detoxifying ammonia via the urea cycle. Mutations in this gene result in the autosomal recessive disorder argininosuccinic aciduria, or argininosuccinic acid lyase deficiency. A nontranscribed pseudogene is also located on the long arm of chromosome 22. Alternatively spliced transcript variants encoding different isoforms have been described.
, delta crystallin II
, delta-2 crystallin