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anti-Rat (Rattus) SNW1 Anticorps:
anti-Human SNW1 Anticorps:
anti-Mouse (Murine) SNW1 Anticorps:
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Human Polyclonal SNW1 Primary Antibody pour ICC, IF - ABIN4354973
Lackner, Durocher, Karlseder: A siRNA-based screen for genes involved in chromosome end protection. dans PLoS ONE 2011
c-Abl (Montrer ABL1 Anticorps) promotes TGF-beta (Montrer TGFB1 Anticorps)-induced SKIP/Smad3 (Montrer SMAD3 Anticorps) interaction.
Upon step 1 catalysis, Cwc25 (Montrer CWC25 Anticorps) contacts with the branch-site region, and enhanced crosslinks of Prp8 (Montrer PRPF8 Anticorps) and Prp45 with nucleotides surrounding the branch-site were observed.
Rab1A (Montrer RAB1A Anticorps) regulates anterograde melanosome transport by recruiting kinesin-1 to melanosomes through interaction with SKIP
Results show that SNW1 directly associates with EFTUD2 (Montrer EFTUD2 Anticorps) and SNRNP200 (Montrer SNRNP200 Anticorps) and that disruption of SNW1 association with these proteins promotes the apoptosis of breast cancer cells.
A transcriptome-wide analysis revealed that SNW1 or PRPF8 (Montrer PRPF8 Anticorps) depletion affects the splicing of specific introns in a subset of pre-mRNAs, including pre-mRNAs encoding the cohesion protein sororin (Montrer CDCA5 Anticorps) and the APC (Montrer APC Anticorps)/C subunit APC2.
High SKIP expression was detected in clinical HCC (Montrer FAM126A Anticorps) samples.
SKIP increased 5alpha-dihydrotestosterone (DHT) induced N-terminal/C-terminal AR interaction from 12-fold to almost 300-fold in a two-hybrid assay, and enhanced AR ligand-independent AF-1 (Montrer EFNA5 Anticorps) transactivation.
Arl8 (Montrer ARL5B Anticorps) and SKIP are required for lysosomes to distribute away from the microtubule-organizing center. We identify two kinesin light chain binding motifs in SKIP that are required for lysosomes to accumulate kinesin-1 and redistribute to the cell periphery.
SKIP is required for epithelial mesenchymal transition and invasiveness induced by TGF-beta1 (Montrer TGFB1 Anticorps) in transformed cells.
This suggests that transcription of stress response genes, unlike, e.g., the SNW1-sensitive mitosis-specific genes, can proceed uncoupled from regulators that normally function under physiological conditions.
interaction with vitamin D receptor (Montrer VDR Anticorps) helix H10 (Montrer H1F0 Anticorps) residues
This gene, a member of the SNW gene family, encodes a coactivator that enhances transcription from some Pol II promoters. This coactivator can bind to the ligand-binding domain of the vitamin D receptor and to retinoid receptors to enhance vitamin D-, retinoic acid-, estrogen-, and glucocorticoid-mediated gene expression. It can also function as a splicing factor by interacting with poly(A)-binding protein 2 to directly control the expression of muscle-specific genes at the transcriptional level. Finally, the protein may be involved in oncogenesis since it interacts with a region of SKI oncoproteins that is required for transforming activity.
SKI interacting protein
, nuclear protein SkiP
, nuclear protein skip
, SKI-interacting protein
, SNW domain-containing protein 1
, homolog of Drosophila BX42
, nuclear receptor coactivator NCoA-62
, nuclear receptor coactivator, 62-kD
, ski-interacting protein