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Human Aquaporin 4 Kit ELISA pour Sandwich ELISA - ABIN414404
Blocher, Eckert, Elster, Wiefek, Eiffert, Schmidt: Aquaporins AQP1 and AQP4 in the cerebrospinal fluid of bacterial meningitis patients. dans Neuroscience letters 2011
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Rat (Rattus) Aquaporin 4 Kit ELISA pour Sandwich ELISA - ABIN416092
Liu, Zhang, Zhao, Chang, Li, Yu, Zhang, Zhang: Melanocortin MC4 receptor agonists alleviate brain damage in abdominal compartment syndrome in the rat. dans Neuropeptides 2015
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Ordered disorder of the astrocytic dystrophin (Montrer DMD Kits ELISA)-associated protein complex in the norm and pathology.
the AQP4 could play a role in the regulation of water balance and ion transport in the sensory cells of zebrafish, bringing new data for the utilizing of this experimental model in the biology of sensory system.
These results suggest that AQP4 is damaged first and decrease of EAAT-2 (Montrer SLC1A2 Kits ELISA) may follow in pathogenesis of cortical degeneration. This is the first demonstration of decrease of AQP4 and its association with EAAT-2 (Montrer SLC1A2 Kits ELISA) decrease in AIDS brain, suggesting a role in the pathogenesis of HIV-associated neurocognitive disorders.
Comparative molecular dynamics study of neuromyelitis optica-immunoglobulin G binding to AQP4 extracellular domains has been presented.
Osmotic water permeabilities of aquaporins AQP4, AQP5 (Montrer AQP5 Kits ELISA), and GlpF from near-equilibrium simulations have been presented.
A clear correlation between AQP4 expression and ADCmax values in grade I meningioma was identified.
Peritumoral brain edema in patients with meningiomas may depend on AQP4 expression grades and not on tumor grade, tumor volume, Ki-67 (Montrer MKI67 Kits ELISA) expression, and cell count.
AQP4 antibodies were higher in neuromyelitis optica Chinese Han patients with circulating auto-antibodies.
Retinal nerve fiber layer may be better preserved in MOG-IgG versus AQP4-IgG optic neuritis.
AQP4-autoantibody serostatus correlated with poor visual outcome in neuromyelitis optica (Meta-analysis)
Data show that aquaporin-4 (AQP4) and myelin-oligodendrocyte glycoprotein (MOG) autoantibodies double positive neuromyelitis optica spectrum disorder (NMOSD) patients had a multiphase disease course with a high annual relapse rate.
In this study, altered AQP4 expression was associated with aging brains.
Results suggest that pain is an important factor in the quality of life experienced by both AQP4-Ab positive and negative transverse myelitis patients and suggest an even greater association than disability
The results of this study that AQP4 and GLT1 do not have a strong physical interaction between them and are, instead, differentially regulated.
Study suggests that astaxanthin may exert neuroprotection following traumatic brain injury by ameliorating AQP4/NKCC1 (Montrer SLC12A2 Kits ELISA)-mediated cerebral edema, and NKCC1 (Montrer SLC12A2 Kits ELISA) inactivation inhibitions the upregulation of AQP4 after traumatic brain injury.
this study identified and fine mapped the major T-cell epitope of AQP4 and observed that in a transfer model, AQP4-specific T cells alone induced an encephalomyelitic syndrome, however, only in the additional presence of anti-AQP4 antibodies, central nervous system lesions are reminiscent of neuromyelitis optica
Study found that aquaporin-4 deficiency facilitated fear memory extinction and NMDA receptors-dependent long-term depression in the CA3 (Montrer CA3 Kits ELISA)-CA1 (Montrer CA1 Kits ELISA) hippocampal pathway.
Study demonstrated evidence of beta-dystroglycan cleavage by matrix metalloproteinase-2 (Montrer MMP2 Kits ELISA)/-9 in permanent middle cerebral artery occlusion mouse brains; this cleavage was implicated in aquaporin-4 redistribution and brain edema in cerebral ischemia.
This study demonstrated that AQP4 dorsal hippocampal protein expression was significantly downregulated at 1day post epilepsy and was followed by a gradual return to baseline levels with a significant increase in ipsilateral protein levels by 30days post epilepsy.
The major findings in this study were (1) a higher AQP4 immunogold labeling density over parenchymal astrocytic membranes in human subjects than in mice, and (2) a lower degree of AQP4 immunogold polarization to perivascular astrocytic endfoot membranes in humans compared with mice. In the human individuals, endfoot AQP4 polarization did not differ between capillary and arteriolar vessel segments.
TRPV4 (Montrer TRPV4 Kits ELISA)-AQP4 interactions 'turbocharge' astroglial sensitivity to small osmotic gradients
Elevation of AQP4 and selective cytokines in experimental autoimmune encephalitis mice provides some potential biomarkers in optic neuritis and demyelinating diseases.
This study provided the AQP4 modulates astrocyte-to-microglia communication in neuroinflammation.
AQP4 plays an important role in mediating brain edema in hypoxic-ischemic encephalopathy.
constitutive recycling of AQP2 (Montrer AQP2 Kits ELISA) does not require phosphorylation at any C-terminal sites
In conclusion, HPO (Montrer GFER Kits ELISA) can decrease AQP4 expression in brain tissue of rabbits with cerebral hemorrhage to suppress the progression of brain edema and promote repairing of injured tissue.
AQP4 may play an important role in brain edema after severe scald (Montrer RDH11 Kits ELISA).
In the guinea pig AQP4 is localised to enteric glial cells.
This gene encodes a member of the aquaporin family of intrinsic membrane proteins that function as water-selective channels in the plasma membranes of many cells. The encoded protein is the predominant aquaporin found in brain. Two alternatively spliced transcript variants encoding distinct isoforms have been found for this gene.
, aquaporin 4
, aquaporin type4
, mercurial-insensitive water channel
, aquaporin 4.M23
, mercurial insensitive water channel
, water channel