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anti-Human BCL2 Anticorps:
anti-Rat (Rattus) BCL2 Anticorps:
anti-Mouse (Murine) BCL2 Anticorps:
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Human Monoclonal BCL2 Primary Antibody pour ICC, IHC (fro) - ABIN3044481
Jiang, Li, Zhou, Wang, Zhang, Wang: Colistin-induced apoptosis in PC12 cells: involvement of the mitochondrial apoptotic and death receptor pathways. dans International journal of molecular medicine 2014
Show all 91 Pubmed References
Human Polyclonal BCL2 Primary Antibody pour ELISA, WB - ABIN3042814
Zong, Li, Lin, Hou, Ma: Lipoxin A4 pretreatment mitigates skeletal muscle ischemia-reperfusion injury in rats. dans American journal of translational research 2017
Show all 79 Pubmed References
Human Polyclonal BCL2 Primary Antibody pour IF, IHC (p) - ABIN388095
Zheng, Zhou, Ye, Li, Bai, Zhang: The conformation change of Bcl-2 is involved in arsenic trioxide-induced apoptosis and inhibition of proliferation in SGC7901 human gastric cancer cells. dans World journal of surgical oncology 2010
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Human Monoclonal BCL2 Primary Antibody pour IHC (fro), FACS - ABIN967312
Batistatou, Merry, Korsmeyer, Greene: Bcl-2 affects survival but not neuronal differentiation of PC12 cells. dans The Journal of neuroscience : the official journal of the Society for Neuroscience 1993
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Mouse (Murine) Monoclonal BCL2 Primary Antibody pour FM, IHC (fro) - ABIN967441
Hockenbery, Nuñez, Milliman, Schreiber, Korsmeyer: Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death. dans Nature 1991
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Human Monoclonal BCL2 Primary Antibody pour IHC (f), IHC (fro) - ABIN967321
Krajewski, Tanaka, Takayama, Schibler, Fenton, Reed: Investigation of the subcellular distribution of the bcl-2 oncoprotein: residence in the nuclear envelope, endoplasmic reticulum, and outer mitochondrial membranes. dans Cancer research 1993
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Human Monoclonal BCL2 Primary Antibody pour IHC (f), IHC (fro) - ABIN967322
Pezzella, Jones, Ralfkiaer, Ersbøll, Gatter, Mason: Evaluation of bcl-2 protein expression and 14;18 translocation as prognostic markers in follicular lymphoma. dans British journal of cancer 1992
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Dog (Canine) Polyclonal BCL2 Primary Antibody pour IHC, IHC (p) - ABIN4283360
Chopra, Ray, Dwivedi, Tiwari, Singh, Singh, Kushwaha, Jahan, Pandey, Gupta, Chaturvedi, Pant, Ray, Gupta: Photoprotective efficiency of PLGA-curcumin nanoparticles versus curcumin through the involvement of ERK/AKT pathway under ambient UV-R exposure in HaCaT cell line. dans Biomaterials 2016
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Human Polyclonal BCL2 Primary Antibody pour IF (p), IHC (p) - ABIN707156
Wang, Ma, Su: NF-?B pathway contributes to cadmium-induced apoptosis of porcine granulosa cells. dans Biological trace element research 2013
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Human Monoclonal BCL2 Primary Antibody pour IP, IHC - ABIN222984
Cordiali-Fei, Ardigo, Mastroianni, Giuliani, D Agosto, Bordignon, Trento, Vento, Berardesca: Serum cytokines and biohumoral immunological characterization of psoriatic patients in long term etanercept treatment. dans International journal of immunopathology and pharmacology 2008
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Th17 cells are resistant to glucocorticoid-induced apoptosis and cytokine suppression, at least in part due to high levels of BCL-2. These findings support a role of Th17 cells in glucocorticoid-resistant inflammatory conditions such as certain endotypes of asthma.
The findings are consistent with rs539846 influencing chronic lymphocytic leukemia (CLL) susceptibility through differential RELA (Montrer NFkBP65 Anticorps) binding, with direct modulation of BMF (Montrer BMF Anticorps) expression impacting on anti-apoptotic BCL2, a hallmark of oncogenic dependency in CLL.
DR_MOMP delivers a system-based biomarker with significant potential as a prognostic tool for stage III CRC (Montrer CALR Anticorps) that significantly improves established histopathological risk factors. BCL-2-dependent signalling critically contributed to treatment responses in consensus molecular subtypes 1 and 3, linking for the first time specific molecular subtypes to apoptosis signalling.
Results show that expression level of mTOR (Montrer FRAP1 Anticorps) and BCL2 are regulated by miR497 and provide evidence for their role in the development of TMZ-resistance phenotype of glioma cells.
Data show that MYC (Montrer MYC Anticorps), BCL2 and BCL6 (Montrer BCL6 Anticorps) translocation had no prognostic impact with respect to progression free survival (PFS) or overall survival (OS).
Interaction of the N-Terminal Tandem Domains of hnRNP (Montrer HNRNPC Anticorps) LL with the BCL2 Promoter i-Motif DNA Sequence
MYC (Montrer MYC Anticorps)/BCL2 co-expression is associated with a poorer prognosis and is independent of cell-of-origin classification in diffuse large B-cell lymphoma in the CNS.
These findings provide new insights into MCL-1 (Montrer MCL1 Anticorps) ligands, and the interplay between DRP-1 (Montrer CRMP1 Anticorps) and the anti-apoptotic BCL-2 family members in the regulation of apoptosis
Our findings suggest that the molecular machinery and mechanisms for executing Bcl-2-mediated apoptosis as observed in mammals are evolutionary ancient, with early regulation of apoptotic machineries closely resembling their modern counterparts in mammals rather than Caenorhabditis elegans or drosophila.
Clinical utility for selective inhibition of specific anti-apoptotic Bcl-2 family proteins has recently been realized with the Food and Drug Administration (FDA) approval of venetoclax (formerly ABT-199/GDC (Montrer SLC25A16 Anticorps)-0199) in relapsed chronic lymphocytic leukemia (CLL) with 17p deletion.
E2 protects skeletal myoblasts against apoptosis induced by H2 O2 modulating p53 (Montrer TP53 Anticorps) and FoxO (Montrer FOXO3 Anticorps) transcription factors and then their target genes Bcl-2, Bim (Montrer BCL2L11 Anticorps), Puma (Montrer BBC3 Anticorps), PERP (Montrer PERP Anticorps), and MDM2 (Montrer MDM2 Anticorps), without affecting Noxa (Montrer PMAIP1 Anticorps) gene.
Bcl-2 expression has a significant impact on the mineralogical content of enamel crystals of tooth structure
The results suggest that CARP (Montrer ANKRD1 Anticorps) can protect against hypoxia-reperfusion induced cardiomyocyte apoptosis, possibly through increasing anti-apoptosis Bcl2 gene expression.
This study provides evidence from transgenic mouse models that Crebbp (Montrer CREBBP Anticorps) deletion results in deficits in B-cell development and can cooperate with Bcl2 overexpression to promote B-cell lymphoma.
an essential role for the binding of BCL2 to BIM (Montrer BCL2L11 Anticorps) in the survival of noncycling NK cells.
LIGHT signalling pathway combined with IFN-gamma (Montrer IFNG Anticorps) induces beta cells apoptosis via an NF-kappaB (Montrer NFKB1 Anticorps)/Bcl2-dependent mitochondrial pathway.
Bcl-2 was highly expressed in tubulointerstitial infiltrates in (NZB x NZW)F1 (NZB/NZW) lupus-prone mice.
The abnormal expression of epidermal cytokeratins suggests that Ha-Ras and Bcl-2 suppress the terminal differentiation and sustain the stem cell-like features in epidermal keratinocytes
the augmentation of endogenous MTA1 (Montrer MTA1 Anticorps) expression during neuronal ischemic injury acts additionally to an endocrinous cascade orchestrating intimate interactions between ERalpha (Montrer ESR1 Anticorps) and BCL2 pathways.
These results suggest a role for mitochondrial p53 (Montrer TP53 Anticorps) activity in promoting hair cell death due to aminoglycosides, likely upstream of Bax (Montrer BAX Anticorps) and Bcl2.
Zebrafish gene expressions of P53 (Montrer TP53 Anticorps), Bcl-2, Bax (Montrer BAX Anticorps) and caspase-3 (Montrer CASP3 Anticorps) were elevated after exposure with microcystin-LR under different ambient temperatures.
Analysis using clinical biopsy specimens revealed autophagy and increased levels of BCL2, S1P1 (Montrer S1PR1 Anticorps), and ICAM1 (Montrer ICAM1 Anticorps) in human T-lymphoblastic lymphoma compared with T-lymphoblastic leukemia.
Real-time RT-PCR revealed acetaminophen treatment of zebrafish embryos decreased the expression of cox2 (Montrer COX2 Anticorps) and bcl2, but increased p53 (Montrer TP53 Anticorps) expression.
Leukemia onset was dramatically accelerated in transgenic fish overexpressing human Notch1 (Montrer NOTCH1 Anticorps) and fish bcl-2, indicating synergy between the Notch (Montrer NOTCH1 Anticorps) pathway and the bcl2-mediated antiapoptotic pathway.
Over-expression of STC-1 (Montrer STC1 Anticorps) up-regulated Bcl-2 protein expression and slightly down-regulated caspase-3 (Montrer CASP3 Anticorps) production in the damaged cells. Findings from this study suggest that STC-1 (Montrer STC1 Anticorps) plays a protective role in intestinal cells through an antioxidant mechanism.
BoHV-5 replication apparently modulates BCL-2 expression and gene transcription, enhancing production of virus progeny, but does not increase Bax (Montrer BAX Anticorps) expression.
G3139, a BCL-2 antisesnse oligonucleotide is a promising candidate for treatment of patients with imatinib-resistant Ph-positive leukemia.
role of vascular endothelial growth factor receptor-1 (Montrer FLT1 Anticorps) in Bcl-2 expression
apoptosis-inducing factor (Montrer AIFM1 Anticorps) was expressed in luminal alveolar cells and, in concert with a change in bax (Montrer BAX Anticorps) protein to bcl-2 protein ratio, might contribute to signalling of a change in the dynamic balance of the cell population as lactation progresses
mRNA expression of Bax (Montrer BAX Anticorps), Bcl-2, caspase-3 (Montrer CASP3 Anticorps) and-7 cannot be used as a reliable apoptosis detection method.
These results propose an additional mechanism whereby resveratrol may exert its cardioprotective effects and suggest a key role for Bcl-2 in the resveratrol anti-apoptotic action, especially in disrupting peroxynitrite-triggered mitochondrial pathway.
bcl-2 and bax (Montrer BAX Anticorps) were expressed strongly in denervated guinea-pig facial muscle. [bcl-2; bax (Montrer BAX Anticorps)]
excessive apoptosis does occur in acetabular cartilage with developmental dislocations of the hip, and is positively correlated with high caspase-3 (Montrer CASP3 Anticorps) expression as well as low Bcl-2 expression.
These results indicate that RI-PostC can ameliorate myocardial ischemia-reperfusion injury and increase the Bcl-2/Bax (Montrer BAX Anticorps) ratio through a mechanism involving protein kinase C (Montrer PKC Anticorps).
Cinobufagin can remarkably inhibit the proliferation and induce the apoptosis of lens epithelial cells by increasing expression of bax (Montrer BAX Anticorps) and decreasing expression of bcl2.
Elevated local temperature of the testis buried in the inguinal pocket increases the apoptosis of spermatogenic cells, and the spermatogenic cell apoptosis is highly correlated with the decreased expression of Bcl-2 and increased expression of Bax (Montrer BAX Anticorps).
Our data indicate that exposure to rabbits to Pb(Ac)(2) caused a significant increase of apoptosis protein p53 (Montrer TP53 Anticorps) and decrease in the antiapoptotic BCl2 proteins.
Isoflurane delayed preconditioning can inhibit the apoptosis of myocardium by up-regulating the expression of Bcl-2 and down-regulating the activation of caspase-3 (Montrer CASP3 Anticorps).
Data show that the damaged electron transport chain leads to bcl-2 depletion and MPT opening.
This gene encodes an integral outer mitochondrial membrane protein that blocks the apoptotic death of some cells such as lymphocytes. Constitutive expression of BCL2, such as in the case of translocation of BCL2 to Ig heavy chain locus, is thought to be the cause of follicular lymphoma. Two transcript variants, produced by alternate splicing, differ in their C-terminal ends.
apoptosis regulator Bcl-2
, protein phosphatase 1, regulatory subunit 50
, B cell lymphoma 2 associated oncogene
, B-cell leukemia/lymphoma 2
, Bcl2-like protein
, BCL2B-cell CLL/lymphoma 2
, B-cell lymphoma protein 2
, B-cell CLL/lymphoma 2
, LOW QUALITY PROTEIN: apoptosis regulator Bcl-2
, Apoptosis regulator Bcl-2