Aperçu des produits pour anti-C-JUN (JUN) Anticorps

Full name:
anti-Jun Proto-Oncogene Anticorps (JUN)
Chez www.anticorps-enligne.fr sont 876 Jun Proto-Oncogene (JUN) Anticorps de 34 de différents fournisseurs disponibles. De plus, nous expédions C-JUN Kits (102) et C-JUN Protéines (24) et beaucoup plus de produits pour cette protéine. Un total de 1063 C-JUN produits sont actuellement listés.
AP-1, AP1, B230310J22Rik, Bsk, BSK/DJNK, c-jun, CG2275, CG5680, cJun, D-JNK, d-JRA, d-jun, D-jun/Jra, D-junk, dAP-1, DBSK/JNK, dJNK, DJNK/bsk, dJra, djun, dm-Jun, Dmel\\CG2275, Dmel\\CG5680, fj36h07, jnk, JNK/SAPK, jra, jun, Junc, Junk, l(2)46Ef, l(2)IA109, l(2R)IA109, p39, SAPKa, V, wu:fj36h07, zgc:65863

Tous anti-C-JUN Anticorps disponible

Vous arrivez à notre recherche pré-filtrée.

anti-C-JUN Anticorps mieux référencés

  1. Human Polyclonal C-JUN Primary Antibody pour IHC, ELISA - ABIN1531886 : Binétruy, Smeal, Karin: Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain. dans Nature 1991 (PubMed)
    Show all 4 references for 1531886

  2. Human Polyclonal C-JUN Primary Antibody pour DB, IHC (p) - ABIN389544 : Fang, Kerppola: Ubiquitin-mediated fluorescence complementation reveals that Jun ubiquitinated by Itch/AIP4 is localized to lysosomes. dans Proceedings of the National Academy of Sciences of the United States of America 2004 (PubMed)
    Show all 2 references for 389544

  3. Cow (Bovine) Polyclonal C-JUN Primary Antibody pour ChIP, IHC - ABIN2792650 : Adiseshaiah, Li, Vaz, Kalvakolanu, Reddy: ERK signaling regulates tumor promoter induced c-Jun recruitment at the Fra-1 promoter. dans Biochemical and biophysical research communications 2008 (PubMed)
    Show all 2 references for 2792650

  4. Human Polyclonal C-JUN Primary Antibody pour DB - ABIN389696 : Wang, Li, Qi, Yuan, Ai, Zhu, Cao, Yang, Liu, Wu, Liu: TRIM45, a novel human RBCC/TRIM protein, inhibits transcriptional activities of ElK-1 and AP-1. dans Biochemical and biophysical research communications 2004 (PubMed)
    Show all 2 references for 389696

  5. Human Polyclonal C-JUN Primary Antibody pour FACS, IHC (p) - ABIN650689 : Song, Han, Sabapathy, Lee, Yu, Choi: Expression of a homeostatic regulator, Wip1 (wild-type p53-induced phosphatase), is temporally induced by c-Jun and p53 in response to UV irradiation. dans The Journal of biological chemistry 2010 (PubMed)
    Show all 2 references for 650689

  6. Cow (Bovine) Polyclonal C-JUN Primary Antibody pour WB - ABIN2774668 : Trøen, Nygaard, Jenssen, Ikonomou, Tierens, Matutes, Gruszka-Westwood, Catovsky, Myklebost, Lauritzsen, Hovig, Delabie: Constitutive expression of the AP-1 transcription factors c-jun, junD, junB, and c-fos and the marginal zone B-cell transcription factor Notch2 in splenic marginal zone lymphoma. dans The Journal of molecular diagnostics : JMD 2004 (PubMed)

  7. Cow (Bovine) Polyclonal C-JUN Primary Antibody pour WB - ABIN2780411 : Grau, Punzón, Fresno, Iñiguez: Peroxisome-proliferator-activated receptor alpha agonists inhibit cyclo-oxygenase 2 and vascular endothelial growth factor transcriptional activation in human colorectal carcinoma cells via inhibition of activator protein-1. dans The Biochemical journal 2006 (PubMed)

Plus d’anticorps contre C-JUN partenaires d’interaction

Xenopus laevis Jun Proto-Oncogene (JUN) interaction partners

  1. AP-1(c-Jun/FosB (Montrer FOSB Anticorps)) may play a role in neurogenesis via the induction of FoxD5b expression during early vertebrate development

  2. The cJun transcription factor bound to a variant cAMP response element in the promoter region of tlx3 (Montrer TLX3 Anticorps) and modulated transcription and regulated neurotransmitter phenotype via its transactivation domain

Fruit Fly (Drosophila melanogaster) Jun Proto-Oncogene (JUN) interaction partners

  1. Mir (Montrer MYLIP Anticorps)-8 modulates Drosophila C virus replication by negative regulation of dJun.

  2. Here we uncover a cell non-autonomous requirement for the Epidermal growth factor receptor (Egfr (Montrer EGFR Anticorps)) pathway in the lateral epidermis for sustained dpp (Montrer TGFb Anticorps) expression in the LE. Specifically, we demonstrate that Egfr (Montrer EGFR Anticorps) pathway activity in the lateral epidermis prevents expression of the gene scarface (scaf), encoding a secreted antagonist of JNK (Montrer MAPK8 Anticorps) signaling

  3. Tau and spectraplakin promote synapse formation and maintenance through Jun kinase (Montrer MAPK9 Anticorps) and neuronal trafficking.

  4. n addition to significantly increasing the number of JNK (Montrer MAPK8 Anticorps) target genes identified so far, our results reveal that the LE is a highly heterogeneous morphogenetic organizer, sculpted through crosstalk between JNK (Montrer MAPK8 Anticorps), segmental and AP signalling. This fine-tuning regulatory mechanism is essential to coordinate morphogenesis and dynamics of tissue sealing

  5. malignant transformation of the ras(V12)scrib(1 (Montrer SCRIB Anticorps)) tumors requires bZIP protein Fos, the ETS (Montrer ETS1 Anticorps)-domain factor Ets21c and the nuclear receptor Ftz-F1 (Montrer NR5A2 Anticorps), all acting downstream of Jun-N-terminal kinase (Montrer MAPK8 Anticorps).

  6. Diminished MTORC1-dependent JNK (Montrer MAPK8 Anticorps) activation underlies the neurodevelopmental defects associated with lysosomal dysfunction.

  7. ROS (Montrer ROS1 Anticorps)/JNK (Montrer MAPK8 Anticorps)/p38 (Montrer MAPK14 Anticorps)/Upd (Montrer UROD Anticorps) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  8. Significantly, the JNK (Montrer MAPK8 Anticorps) pathway is responsible for the majority of the phenotypes and transcriptional changes downstream of Notch (Montrer NOTCH1 Anticorps)-Src (Montrer SRC Anticorps) synergy.

  9. This study demonstrated that the mechanism by which Bsk (Montrer FRK Anticorps) is required for pruning is through reducing the membrane levels of the adhesion molecule (Montrer NCAM1 Anticorps) Fasciclin II (Montrer NCAM2 Anticorps) (FasII)

  10. Jra recruits the HP1a (Montrer CBX5 Anticorps)/KDM4A (Montrer KDM4A Anticorps) complex to its gene body region upon osmotic stress to reduce H3K36 methylation levels and disrupt H3K36 methylation-dependent histone deacetylation

Pig (Porcine) Jun Proto-Oncogene (JUN) interaction partners

  1. Porcine reproductive and respiratory syndrome virus -activated TAK-1 (Montrer NR2C2 Anticorps) was essential for the activation of JNK (Montrer MAPK8 Anticorps) and NF-kappaB (Montrer NFKB1 Anticorps) pathways and IL-8 (Montrer IL8 Anticorps) expression.

  2. The effects of prostaglandin F2alpha administration on transcription factor AP-1 expression and the expression of downstream genes involved in luteolysis are reported.

Rabbit Jun Proto-Oncogene (JUN) interaction partners

  1. ICAM1 (Montrer ICAM1 Anticorps) and IL10 (Montrer IL10 Anticorps) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 may be responsible for this upregulation.

Mouse (Murine) Jun Proto-Oncogene (JUN) interaction partners

  1. AP1 factors are important regulators of adult taste cell renewal and their downregulation negatively impacts taste maintenance.

  2. these data indicate that MEF2 (Montrer MEF2C Anticorps) and AP-1 confer antagonistic regulation of Hspb7 (Montrer HSPB7 Anticorps) gene expression in skeletal muscle, with implications for autophagy and muscle atrophy.

  3. results suggest that fibroblasts, c-Jun, and IGF-1 (Montrer IGF1 Anticorps) play key roles in mediating stromal-epithelial interactions that are required for the therapeutic effects of finasteride in benign prostate epithelial cells

  4. These findings highlight a key role of the TLR4 (Montrer TLR4 Anticorps)-NOS1 (Montrer NOS1 Anticorps)-AP1 signaling axis in regulating macrophage polarization

  5. Data suggest that Sf1 and c-jun interact and cooperate to activate the Fdx1 promoter in MA-10 (tumorigenic cell line) and TM3 (non-tumorigenic cell line) Leydig cells; such activation requires different regulatory elements located between -124 and -306 bp of Fdx1 promoter and involves recruitment of Sf1 to this region. (Sf1 = splicing factor 1; c-jun = proto-oncogene c-jun; Fdx1 = ferredoxin 1)

  6. In this study, we discovered that selective upregulation of p39 (Montrer ATP6V0D1 Anticorps) is the underlying mechanism that accommodates the increased functional requirement of Cdk5 (Montrer CDK5 Anticorps) activation during neuronal differentiation. In addition, we demonstrated that p39 (Montrer ATP6V0D1 Anticorps) selectively directs Cdk5 (Montrer CDK5 Anticorps) to phosphorylate protein substrates essential for axonal development, dendritic spine formation, and synaptogenesis. Moreover, our studies suggest opposing roles o

  7. NF-kappaB (Montrer NFKB1 Anticorps) and c-Jun coregulate lipopolysaccharide-induced Fra-1 (Montrer FOSL1 Anticorps) transcription.

  8. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  9. this study shows that c-Jun regulates the activation state of macrophages and promotes arthritis via differentially regulating cyclooxygenase-2 (Montrer PTGS2 Anticorps) and arginase-1 (Montrer ARG1 Anticorps) levels

  10. PLIO treatment inhibited nuclear factorkappaB (NFkappaB) nuclear translocation in B16F10 cells. In addition, PLIO treatment inhibited the phosphorylation of c-Jun Nterminal kinases and AKT (Montrer AKT1 Anticorps)

Human Jun Proto-Oncogene (JUN) interaction partners

  1. Our results indicate that assessing AP1 (Montrer FOSB Anticorps) and PEA3 (Montrer ETV4 Anticorps) transcription factor status might be a good indicator of OAC status. However, we could not detect any associations with disease stage or patient treatment regime. This suggests that the PEA3 (Montrer ETV4 Anticorps)-AP1 (Montrer FOSB Anticorps) regulatory module more likely contributes more generally to the cancer phenotype. In keeping with this observation, depletion of ETV1 (Montrer ETV1 Anticorps) and/or ETV4 (Montrer ETV4 Anticorps) causes an OAC cell growth defect

  2. shRNA-mediated inhibition of JUN decreases AML (Montrer RUNX1 Anticorps) cell survival and propagation in vivo. These data uncover a previously unrecognized role of JUN as a regulator of the unfolded protein response

  3. These findings demonstrate an essential role for the ERK pathway together with c-JUN and c-FOS in the differentiation activity of LukS-PV.

  4. The present study defines the minimal TIM-3 (Montrer HAVCR2 Anticorps) promoter region and demonstrates its interaction with c-Jun during TIM-3 (Montrer HAVCR2 Anticorps) transcription in CD4 (Montrer CD4 Anticorps)(+) T cells.

  5. Taken together, our data demonstrate that JNK (Montrer MAPK8 Anticorps) regulates triple-negative breast cancer (TNBC)tumorigenesis by promoting CSC phenotype through Notch1 (Montrer NOTCH1 Anticorps) signaling via activation of c-Jun and indicate that JNK/c-Jun/Notch1 (Montrer NOTCH1 Anticorps) signaling is a potential therapeutic target for TNBC

  6. Regulation of osteosarcoma cell lung metastasis by the c-Fos/AP-1 target FGFR1

  7. c-jun promoted FOXK1 (Montrer FOXK2 Anticorps)-mediated proliferation and metastasis via orthotopic implantation.

  8. Data provide evidence that AP-1 (Montrer FOSB Anticorps) is a key determinant of endocrine resistance of breast cancer cells by mediating a global shift in the estrogen receptor (Montrer ESR1 Anticorps) transcriptional program.

  9. Comparison of how AP-1 (Jun/Jun dimer) and Epstein-Barr virus Zta recognize methyl groups within their cognate response elements

  10. this study revealed the functional and mechanistic links between CDK5 (Montrer CDK5 Anticorps) and the oncogenic ERK5 (Montrer MAPK7 Anticorps)-AP-1 (Montrer FOSB Anticorps) signaling pathway in the pathogenesis of colorectal cancer.

Zebrafish Jun Proto-Oncogene (JUN) interaction partners

  1. These results support a role for trim69 (Montrer TRIM69 Anticorps) in the development of the zebrafish brain through ap-1 pathway.

  2. CPEB-1 (Montrer CPEB1 Anticorps) control of c-Jun mRNA translation regulates GH gene expression and resulting downstream signaling events (e.g., synaptic plasticity) in the mouse hippocampus.

Cow (Bovine) Jun Proto-Oncogene (JUN) interaction partners

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

  2. dynamic compression stimulates cell proliferation and proteoglycan (Montrer Vcan Anticorps) synthesis in the presence of IL-1beta (Montrer IL1B Anticorps) and/or inhibitors of the MAPKs and NFkappaB and AP-1 signalling pathways

C-JUN (JUN) profil antigène

Antigen Summary

This gene is the putative transforming gene of avian sarcoma virus 17. It encodes a protein which is highly similar to the viral protein, and which interacts directly with specific target DNA sequences to regulate gene expression. This gene is intronless and is mapped to 1p32-p31, a chromosomal region involved in both translocations and deletions in human malignancies.

Alternative names and synonyms associated with C-JUN (JUN)

  • jun oncogene (jun) anticorps
  • basket (bsk) anticorps
  • C-JUN protein (C-JUN) anticorps
  • c-Jun protein (C-JUN) anticorps
  • c-jun transcription factor (C-JUN) anticorps
  • cyclin-dependent kinase 5, regulatory subunit 2 (p39) (Cdk5r2) anticorps
  • Jun oncogene (Jun) anticorps
  • jun proto-oncogene (JUN) anticorps
  • jun proto-oncogene (jun) anticorps
  • jun proto-oncogene (Jun) anticorps
  • Jun-related antigen (Jra) anticorps
  • AP-1 anticorps
  • AP1 anticorps
  • B230310J22Rik anticorps
  • Bsk anticorps
  • BSK/DJNK anticorps
  • c-jun anticorps
  • CG2275 anticorps
  • CG5680 anticorps
  • cJun anticorps
  • D-JNK anticorps
  • d-JRA anticorps
  • d-jun anticorps
  • D-jun/Jra anticorps
  • D-junk anticorps
  • dAP-1 anticorps
  • DBSK/JNK anticorps
  • dJNK anticorps
  • DJNK/bsk anticorps
  • dJra anticorps
  • djun anticorps
  • dm-Jun anticorps
  • Dmel\\CG2275 anticorps
  • Dmel\\CG5680 anticorps
  • fj36h07 anticorps
  • jnk anticorps
  • JNK/SAPK anticorps
  • jra anticorps
  • jun anticorps
  • Junc anticorps
  • Junk anticorps
  • l(2)46Ef anticorps
  • l(2)IA109 anticorps
  • l(2R)IA109 anticorps
  • p39 anticorps
  • SAPKa anticorps
  • V anticorps
  • wu:fj36h07 anticorps
  • zgc:65863 anticorps

Protein level used designations for anti-Jun Proto-Oncogene (JUN) Anticorps

v-jun sarcoma virus 17 oncogene homolog , CG5680-PB , CG5680-PE , CG5680-PF , JNK , JUN kinase , Jun N-terminal kinase , Jun NH2-terminal kinase , Jun-N-terminal kinase , Jun-kinase , bsk-PB , bsk-PE , bsk-PF , c-Jun N-terminal kinase , c-Jun aminoterminal kinase , c-Jun-N-terminal kinase , drosophila JNK , AP1 , V-jun avian sarcoma virus 17 oncogene homolog , activator protein 1 , proto-oncogene c-jun , transcription factor AP-1 , CDK5 activator 2 , cyclin-dependent kinase 5 activator 2 , cyclin-dependent kinase 5 regulatory subunit 2 , p39I , AH119 , immediate early , jun A , Jun activation domain binding protein , enhancer-binding protein AP1 , jun oncogene , p39 , proto-oncogene c-Jun , v-jun avian sarcoma virus 17 oncogene homolog , Avian sarcoma virus 17 (v-jun) oncogene homolog , Jun oncogene , v-jun sarcoma virus 17 oncogene , CG2275-PA , CG2275-PB , CG2275-PC , Jra-PA , Jra-PB , Jra-PC , Jun related antigen , complementation group V , lethal(2)1A109 , lethal(2)IA109

379050 Xenopus laevis
44801 Drosophila melanogaster
396913 Sus scrofa
443219 Ovis aries
100008856 Oryctolagus cuniculus
12570 Mus musculus
16476 Mus musculus
3725 Homo sapiens
335916 Danio rerio
24516 Rattus norvegicus
399400 Xenopus laevis
424673 Gallus gallus
609429 Canis lupus familiaris
280831 Bos taurus
100170668 Ovis aries
36057 Drosophila melanogaster
Fournisseurs de qualité sélectionnés pour anti-C-JUN (JUN) Anticorps
Avez-vous cherché autre chose?