Aperçu des produits pour C-JUN (JUN) Kits ELISA

Full name:
Jun Proto-Oncogene Kits ELISA (JUN)
Chez www.anticorps-enligne.fr sont 102 Jun Proto-Oncogene (JUN) Kits ELISA de 15 de différents fournisseurs disponibles. De plus, nous expédions C-JUN Anticorps (876) et C-JUN Protéines (24) et beaucoup plus de produits pour cette protéine. Un total de 1063 C-JUN produits sont actuellement listés.
Synonymes:
AP-1, AP1, B230310J22Rik, Bsk, BSK/DJNK, c-jun, CG2275, CG5680, cJun, D-JNK, d-JRA, d-jun, D-jun/Jra, D-junk, dAP-1, DBSK/JNK, dJNK, DJNK/bsk, dJra, djun, dm-Jun, Dmel\\CG2275, Dmel\\CG5680, fj36h07, jnk, JNK/SAPK, jra, jun, Junc, Junk, l(2)46Ef, l(2)IA109, l(2R)IA109, p39, SAPKa, V, wu:fj36h07, zgc:65863

Plus Kits ELISA pour C-JUN partenaires d'interaction

Xenopus laevis Jun Proto-Oncogene (JUN) interaction partners

  1. AP-1(c-Jun/FosB (Montrer FOSB Kits ELISA)) may play a role in neurogenesis via the induction of FoxD5b expression during early vertebrate development

  2. The cJun transcription factor bound to a variant cAMP response element in the promoter region of tlx3 (Montrer TLX3 Kits ELISA) and modulated transcription and regulated neurotransmitter phenotype via its transactivation domain

Fruit Fly (Drosophila melanogaster) Jun Proto-Oncogene (JUN) interaction partners

  1. Mir (Montrer MYLIP Kits ELISA)-8 modulates Drosophila C virus replication by negative regulation of dJun.

  2. Here we uncover a cell non-autonomous requirement for the Epidermal growth factor receptor (Egfr (Montrer EGFR Kits ELISA)) pathway in the lateral epidermis for sustained dpp (Montrer TGFb Kits ELISA) expression in the LE. Specifically, we demonstrate that Egfr (Montrer EGFR Kits ELISA) pathway activity in the lateral epidermis prevents expression of the gene scarface (scaf), encoding a secreted antagonist of JNK (Montrer MAPK8 Kits ELISA) signaling

  3. Tau and spectraplakin promote synapse formation and maintenance through Jun kinase (Montrer MAPK9 Kits ELISA) and neuronal trafficking.

  4. n addition to significantly increasing the number of JNK (Montrer MAPK8 Kits ELISA) target genes identified so far, our results reveal that the LE is a highly heterogeneous morphogenetic organizer, sculpted through crosstalk between JNK (Montrer MAPK8 Kits ELISA), segmental and AP signalling. This fine-tuning regulatory mechanism is essential to coordinate morphogenesis and dynamics of tissue sealing

  5. malignant transformation of the ras(V12)scrib(1 (Montrer SCRIB Kits ELISA)) tumors requires bZIP protein Fos, the ETS (Montrer ETS1 Kits ELISA)-domain factor Ets21c and the nuclear receptor Ftz-F1 (Montrer NR5A2 Kits ELISA), all acting downstream of Jun-N-terminal kinase (Montrer MAPK8 Kits ELISA).

  6. Diminished MTORC1-dependent JNK (Montrer MAPK8 Kits ELISA) activation underlies the neurodevelopmental defects associated with lysosomal dysfunction.

  7. ROS (Montrer ROS1 Kits ELISA)/JNK (Montrer MAPK8 Kits ELISA)/p38 (Montrer MAPK14 Kits ELISA)/Upd (Montrer UROD Kits ELISA) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  8. Significantly, the JNK (Montrer MAPK8 Kits ELISA) pathway is responsible for the majority of the phenotypes and transcriptional changes downstream of Notch (Montrer NOTCH1 Kits ELISA)-Src (Montrer SRC Kits ELISA) synergy.

  9. This study demonstrated that the mechanism by which Bsk (Montrer FRK Kits ELISA) is required for pruning is through reducing the membrane levels of the adhesion molecule (Montrer NCAM1 Kits ELISA) Fasciclin II (Montrer NCAM2 Kits ELISA) (FasII)

  10. Jra recruits the HP1a (Montrer CBX5 Kits ELISA)/KDM4A (Montrer KDM4A Kits ELISA) complex to its gene body region upon osmotic stress to reduce H3K36 methylation levels and disrupt H3K36 methylation-dependent histone deacetylation

Pig (Porcine) Jun Proto-Oncogene (JUN) interaction partners

  1. Porcine reproductive and respiratory syndrome virus -activated TAK-1 (Montrer NR2C2 Kits ELISA) was essential for the activation of JNK (Montrer MAPK8 Kits ELISA) and NF-kappaB (Montrer NFKB1 Kits ELISA) pathways and IL-8 (Montrer IL8 Kits ELISA) expression.

  2. The effects of prostaglandin F2alpha administration on transcription factor AP-1 expression and the expression of downstream genes involved in luteolysis are reported.

Rabbit Jun Proto-Oncogene (JUN) interaction partners

  1. ICAM1 (Montrer ICAM1 Kits ELISA) and IL10 (Montrer IL10 Kits ELISA) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 may be responsible for this upregulation.

Mouse (Murine) Jun Proto-Oncogene (JUN) interaction partners

  1. AP1 factors are important regulators of adult taste cell renewal and their downregulation negatively impacts taste maintenance.

  2. these data indicate that MEF2 (Montrer MEF2C Kits ELISA) and AP-1 confer antagonistic regulation of Hspb7 (Montrer HSPB7 Kits ELISA) gene expression in skeletal muscle, with implications for autophagy and muscle atrophy.

  3. results suggest that fibroblasts, c-Jun, and IGF-1 (Montrer IGF1 Kits ELISA) play key roles in mediating stromal-epithelial interactions that are required for the therapeutic effects of finasteride in benign prostate epithelial cells

  4. These findings highlight a key role of the TLR4 (Montrer TLR4 Kits ELISA)-NOS1 (Montrer NOS1 Kits ELISA)-AP1 signaling axis in regulating macrophage polarization

  5. Data suggest that Sf1 and c-jun interact and cooperate to activate the Fdx1 promoter in MA-10 (tumorigenic cell line) and TM3 (non-tumorigenic cell line) Leydig cells; such activation requires different regulatory elements located between -124 and -306 bp of Fdx1 promoter and involves recruitment of Sf1 to this region. (Sf1 = splicing factor 1; c-jun = proto-oncogene c-jun; Fdx1 = ferredoxin 1)

  6. In this study, we discovered that selective upregulation of p39 (Montrer ATP6V0D1 Kits ELISA) is the underlying mechanism that accommodates the increased functional requirement of Cdk5 (Montrer CDK5 Kits ELISA) activation during neuronal differentiation. In addition, we demonstrated that p39 (Montrer ATP6V0D1 Kits ELISA) selectively directs Cdk5 (Montrer CDK5 Kits ELISA) to phosphorylate protein substrates essential for axonal development, dendritic spine formation, and synaptogenesis. Moreover, our studies suggest opposing roles o

  7. NF-kappaB (Montrer NFKB1 Kits ELISA) and c-Jun coregulate lipopolysaccharide-induced Fra-1 (Montrer FOSL1 Kits ELISA) transcription.

  8. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  9. this study shows that c-Jun regulates the activation state of macrophages and promotes arthritis via differentially regulating cyclooxygenase-2 (Montrer PTGS2 Kits ELISA) and arginase-1 (Montrer ARG1 Kits ELISA) levels

  10. PLIO treatment inhibited nuclear factorkappaB (NFkappaB) nuclear translocation in B16F10 cells. In addition, PLIO treatment inhibited the phosphorylation of c-Jun Nterminal kinases and AKT (Montrer AKT1 Kits ELISA)

Human Jun Proto-Oncogene (JUN) interaction partners

  1. Our results indicate that assessing AP1 (Montrer FOSB Kits ELISA) and PEA3 (Montrer ETV4 Kits ELISA) transcription factor status might be a good indicator of OAC status. However, we could not detect any associations with disease stage or patient treatment regime. This suggests that the PEA3 (Montrer ETV4 Kits ELISA)-AP1 (Montrer FOSB Kits ELISA) regulatory module more likely contributes more generally to the cancer phenotype. In keeping with this observation, depletion of ETV1 (Montrer ETV1 Kits ELISA) and/or ETV4 (Montrer ETV4 Kits ELISA) causes an OAC cell growth defect

  2. shRNA-mediated inhibition of JUN decreases AML (Montrer RUNX1 Kits ELISA) cell survival and propagation in vivo. These data uncover a previously unrecognized role of JUN as a regulator of the unfolded protein response

  3. These findings demonstrate an essential role for the ERK pathway together with c-JUN and c-FOS in the differentiation activity of LukS-PV.

  4. The present study defines the minimal TIM-3 (Montrer HAVCR2 Kits ELISA) promoter region and demonstrates its interaction with c-Jun during TIM-3 (Montrer HAVCR2 Kits ELISA) transcription in CD4 (Montrer CD4 Kits ELISA)(+) T cells.

  5. Taken together, our data demonstrate that JNK (Montrer MAPK8 Kits ELISA) regulates triple-negative breast cancer (TNBC)tumorigenesis by promoting CSC phenotype through Notch1 (Montrer NOTCH1 Kits ELISA) signaling via activation of c-Jun and indicate that JNK/c-Jun/Notch1 (Montrer NOTCH1 Kits ELISA) signaling is a potential therapeutic target for TNBC

  6. Regulation of osteosarcoma cell lung metastasis by the c-Fos/AP-1 target FGFR1

  7. c-jun promoted FOXK1 (Montrer FOXK2 Kits ELISA)-mediated proliferation and metastasis via orthotopic implantation.

  8. Data provide evidence that AP-1 (Montrer FOSB Kits ELISA) is a key determinant of endocrine resistance of breast cancer cells by mediating a global shift in the estrogen receptor (Montrer ESR1 Kits ELISA) transcriptional program.

  9. Comparison of how AP-1 (Jun/Jun dimer) and Epstein-Barr virus Zta recognize methyl groups within their cognate response elements

  10. this study revealed the functional and mechanistic links between CDK5 (Montrer CDK5 Kits ELISA) and the oncogenic ERK5 (Montrer MAPK7 Kits ELISA)-AP-1 (Montrer FOSB Kits ELISA) signaling pathway in the pathogenesis of colorectal cancer.

Zebrafish Jun Proto-Oncogene (JUN) interaction partners

  1. These results support a role for trim69 (Montrer TRIM69 Kits ELISA) in the development of the zebrafish brain through ap-1 pathway.

  2. CPEB-1 (Montrer CPEB1 Kits ELISA) control of c-Jun mRNA translation regulates GH gene expression and resulting downstream signaling events (e.g., synaptic plasticity) in the mouse hippocampus.

Cow (Bovine) Jun Proto-Oncogene (JUN) interaction partners

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

  2. dynamic compression stimulates cell proliferation and proteoglycan (Montrer Vcan Kits ELISA) synthesis in the presence of IL-1beta (Montrer IL1B Kits ELISA) and/or inhibitors of the MAPKs and NFkappaB and AP-1 signalling pathways

C-JUN (JUN) profil antigène

Antigen Summary

This gene is the putative transforming gene of avian sarcoma virus 17. It encodes a protein which is highly similar to the viral protein, and which interacts directly with specific target DNA sequences to regulate gene expression. This gene is intronless and is mapped to 1p32-p31, a chromosomal region involved in both translocations and deletions in human malignancies.

Alternative names and synonyms associated with C-JUN (JUN)

  • jun oncogene (jun) Elisa Kit
  • basket (bsk) Elisa Kit
  • C-JUN protein (C-JUN) Elisa Kit
  • c-Jun protein (C-JUN) Elisa Kit
  • c-jun transcription factor (C-JUN) Elisa Kit
  • cyclin-dependent kinase 5, regulatory subunit 2 (p39) (Cdk5r2) Elisa Kit
  • Jun oncogene (Jun) Elisa Kit
  • jun proto-oncogene (JUN) Elisa Kit
  • jun proto-oncogene (jun) Elisa Kit
  • jun proto-oncogene (Jun) Elisa Kit
  • Jun-related antigen (Jra) Elisa Kit
  • AP-1 Elisa Kit
  • AP1 Elisa Kit
  • B230310J22Rik Elisa Kit
  • Bsk Elisa Kit
  • BSK/DJNK Elisa Kit
  • c-jun Elisa Kit
  • CG2275 Elisa Kit
  • CG5680 Elisa Kit
  • cJun Elisa Kit
  • D-JNK Elisa Kit
  • d-JRA Elisa Kit
  • d-jun Elisa Kit
  • D-jun/Jra Elisa Kit
  • D-junk Elisa Kit
  • dAP-1 Elisa Kit
  • DBSK/JNK Elisa Kit
  • dJNK Elisa Kit
  • DJNK/bsk Elisa Kit
  • dJra Elisa Kit
  • djun Elisa Kit
  • dm-Jun Elisa Kit
  • Dmel\\CG2275 Elisa Kit
  • Dmel\\CG5680 Elisa Kit
  • fj36h07 Elisa Kit
  • jnk Elisa Kit
  • JNK/SAPK Elisa Kit
  • jra Elisa Kit
  • jun Elisa Kit
  • Junc Elisa Kit
  • Junk Elisa Kit
  • l(2)46Ef Elisa Kit
  • l(2)IA109 Elisa Kit
  • l(2R)IA109 Elisa Kit
  • p39 Elisa Kit
  • SAPKa Elisa Kit
  • V Elisa Kit
  • wu:fj36h07 Elisa Kit
  • zgc:65863 Elisa Kit

Protein level used designations for Jun Proto-Oncogene (JUN) Kits ELISA

v-jun sarcoma virus 17 oncogene homolog , CG5680-PB , CG5680-PE , CG5680-PF , JNK , JUN kinase , Jun N-terminal kinase , Jun NH2-terminal kinase , Jun-N-terminal kinase , Jun-kinase , bsk-PB , bsk-PE , bsk-PF , c-Jun N-terminal kinase , c-Jun aminoterminal kinase , c-Jun-N-terminal kinase , drosophila JNK , AP1 , V-jun avian sarcoma virus 17 oncogene homolog , activator protein 1 , proto-oncogene c-jun , transcription factor AP-1 , CDK5 activator 2 , cyclin-dependent kinase 5 activator 2 , cyclin-dependent kinase 5 regulatory subunit 2 , p39I , AH119 , immediate early , jun A , Jun activation domain binding protein , enhancer-binding protein AP1 , jun oncogene , p39 , proto-oncogene c-Jun , v-jun avian sarcoma virus 17 oncogene homolog , Avian sarcoma virus 17 (v-jun) oncogene homolog , Jun oncogene , v-jun sarcoma virus 17 oncogene , CG2275-PA , CG2275-PB , CG2275-PC , Jra-PA , Jra-PB , Jra-PC , Jun related antigen , complementation group V , lethal(2)1A109 , lethal(2)IA109

GENE ID SPECIES
379050 Xenopus laevis
44801 Drosophila melanogaster
396913 Sus scrofa
443219 Ovis aries
100008856 Oryctolagus cuniculus
12570 Mus musculus
16476 Mus musculus
3725 Homo sapiens
335916 Danio rerio
24516 Rattus norvegicus
399400 Xenopus laevis
424673 Gallus gallus
609429 Canis lupus familiaris
280831 Bos taurus
100170668 Ovis aries
36057 Drosophila melanogaster
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