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Aperçu des produits pour C-JUN (JUN) Kits ELISA

Full name:
Jun Proto-Oncogene Kits ELISA (JUN)
Chez sont 99 Jun Proto-Oncogene (JUN) Kits ELISA de 14 de différents fournisseurs disponibles. De plus, nous expédions C-JUN Anticorps (836) et C-JUN Protéines (23) et beaucoup plus de produits pour cette protéine. Un total de 1019 C-JUN produits sont actuellement listés.
AP-1, AP1, B230310J22Rik, Bsk, BSK/DJNK, c-jun, CG2275, CG5680, cJun, D-JNK, d-JRA, d-jun, D-jun/Jra, D-junk, dAP-1, DBSK/JNK, dJNK, DJNK/bsk, dJra, djun, dm-Jun, Dmel\\CG2275, Dmel\\CG5680, fj36h07, jnk, JNK/SAPK, jra, jun, Junc, Junk, l(2)46Ef, l(2)IA109, l(2R)IA109, p39, SAPKa, V, wu:fj36h07, zgc:65863

Plus Kits ELISA pour C-JUN partenaires d'interaction

Xenopus laevis Jun Proto-Oncogene (JUN) interaction partners

  1. AP-1(c-Jun/FosB (Montrer FOSB Kits ELISA)) may play a role in neurogenesis via the induction of FoxD5b expression during early vertebrate development

  2. The cJun transcription factor bound to a variant cAMP response element in the promoter region of tlx3 (Montrer TLX3 Kits ELISA) and modulated transcription and regulated neurotransmitter phenotype via its transactivation domain

Fruit Fly (Drosophila melanogaster) Jun Proto-Oncogene (JUN) interaction partners

  1. Tau and spectraplakin promote synapse formation and maintenance through Jun kinase (Montrer MAPK9 Kits ELISA) and neuronal trafficking.

  2. n addition to significantly increasing the number of JNK (Montrer MAPK8 Kits ELISA) target genes identified so far, our results reveal that the LE is a highly heterogeneous morphogenetic organizer, sculpted through crosstalk between JNK (Montrer MAPK8 Kits ELISA), segmental and AP signalling. This fine-tuning regulatory mechanism is essential to coordinate morphogenesis and dynamics of tissue sealing

  3. malignant transformation of the ras(V12)scrib(1 (Montrer SCRIB Kits ELISA)) tumors requires bZIP protein Fos, the ETS (Montrer ETS1 Kits ELISA)-domain factor Ets21c and the nuclear receptor Ftz-F1 (Montrer NR5A2 Kits ELISA), all acting downstream of Jun-N-terminal kinase (Montrer MAPK8 Kits ELISA).

  4. Diminished MTORC1-dependent JNK (Montrer MAPK8 Kits ELISA) activation underlies the neurodevelopmental defects associated with lysosomal dysfunction.

  5. ROS (Montrer ROS1 Kits ELISA)/JNK (Montrer MAPK8 Kits ELISA)/p38 (Montrer MAPK14 Kits ELISA)/Upd (Montrer UROD Kits ELISA) stress responsive module restores tissue homeostasis. This module is not only activated after cell death induction but also after physical damage and reveals one of the earliest responses for imaginal disc regeneration.

  6. Significantly, the JNK (Montrer MAPK8 Kits ELISA) pathway is responsible for the majority of the phenotypes and transcriptional changes downstream of Notch (Montrer NOTCH1 Kits ELISA)-Src (Montrer SRC Kits ELISA) synergy.

  7. This study demonstrated that the mechanism by which Bsk (Montrer FRK Kits ELISA) is required for pruning is through reducing the membrane levels of the adhesion molecule (Montrer NCAM1 Kits ELISA) Fasciclin II (Montrer NCAM2 Kits ELISA) (FasII)

  8. Jra recruits the HP1a (Montrer CBX5 Kits ELISA)/KDM4A (Montrer KDM4A Kits ELISA) complex to its gene body region upon osmotic stress to reduce H3K36 methylation levels and disrupt H3K36 methylation-dependent histone deacetylation

  9. Study solves the crystal structure of unphosphorylated DJNK in complex with adenylyl imidodiphosphate (AMP (Montrer AMPH Kits ELISA)-PNP (Montrer NP Kits ELISA)) and magnesium.

  10. PERK/ATF4 activated the JNK pathway through Rac1 and Slpr activation in apoptotic cells.

Pig (Porcine) Jun Proto-Oncogene (JUN) interaction partners

  1. The effects of prostaglandin F2alpha administration on transcription factor AP-1 expression and the expression of downstream genes involved in luteolysis are reported.

Rabbit Jun Proto-Oncogene (JUN) interaction partners

  1. ICAM1 (Montrer ICAM1 Kits ELISA) and IL10 (Montrer IL10 Kits ELISA) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 may be responsible for this upregulation.

Mouse (Murine) Jun Proto-Oncogene (JUN) interaction partners

  1. NF-kappaB (Montrer NFKB1 Kits ELISA) and c-Jun coregulate lipopolysaccharide-induced Fra-1 (Montrer FOSL1 Kits ELISA) transcription.

  2. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  3. this study shows that c-Jun regulates the activation state of macrophages and promotes arthritis via differentially regulating cyclooxygenase-2 (Montrer PTGS2 Kits ELISA) and arginase-1 (Montrer ARG1 Kits ELISA) levels

  4. PLIO treatment inhibited nuclear factorkappaB (NFkappaB) nuclear translocation in B16F10 cells. In addition, PLIO treatment inhibited the phosphorylation of c-Jun Nterminal kinases and AKT (Montrer AKT1 Kits ELISA)

  5. Anxa5 (Montrer ANXA5 Kits ELISA) mediates the in vitro malignant behaviours of murine hepatocarcinoma Hca-F cells via ERK2 (Montrer MAPK1 Kits ELISA)/c-Jun/p-c-Jun(Ser73) and ERK2 (Montrer MAPK1 Kits ELISA)/E-cadherin (Montrer CDH1 Kits ELISA) pathways.

  6. These findings indicate that c-Jun has important functions during HBV-associated tumorigenesis by promoting hepatocyte proliferation as well as progression of dysplasia

  7. The TLR4 (Montrer TLR4 Kits ELISA) pathway may play an important role in regulating AP-1 activation

  8. These observations suggest that the antiinflammatory properties of crebanine may stem from the inhibition of proinflammatory mediators via suppression of the NF-kappaB (Montrer NFKB1 Kits ELISA), AP-1, MAPKs, and Akt (Montrer AKT1 Kits ELISA) signaling pathways.

  9. In a model of heavy metal poisoning, copper contamination of drinking water (as can occur with corrosion in aging plumbing systems) up-regulates binding of transcription factor AP-1 in neurons of brain, even in low concentrations.

  10. Transplant-induced reactivation of murine cytomegalovirus immediate early gene expression is associated with recruitment of NF-kappaB (Montrer NFKB1 Kits ELISA) and AP-1 to the major immediate early promoter.

Human Jun Proto-Oncogene (JUN) interaction partners

  1. The authors find that PRR7 inhibits the ubiquitination of c-Jun by E3 ligase SCF (Montrer KITLG Kits ELISA)(FBW) (7) (FBW7 (Montrer FBXW7 Kits ELISA)), increases c-Jun-dependent transcriptional activity, and promotes neuronal death.

  2. melatonin synergistically augmented the sorafenib-induced apoptosis after 48 hours incubation, which was in accordance with the activation of caspase-3 (Montrer CASP3 Kits ELISA) and the JNK/c-jun pathway.

  3. BATF/JUN-B and BATF/C-JUN complexes play important roles in OA cartilage destruction through regulating anabolic and catabolic gene expression in chondrocytes.

  4. the current studies provide evidence showing that c-Jun directly binds to a consensus binding sequence within the GNA12 (Montrer GNA12 Kits ELISA)-5' regulatory region, thereby regulating GNA12 (Montrer GNA12 Kits ELISA) transcription.

  5. we demonstrated that miR (Montrer MLXIP Kits ELISA)-22 promoted monocyte/macrophage differentiation, and MECOM (EVI1 (Montrer MECOM Kits ELISA)) mRNA is a direct target of miR (Montrer MLXIP Kits ELISA)-22 and MECOM (EVI1 (Montrer MECOM Kits ELISA)) functions as a negative regulator in the differentiation.The miR (Montrer MLXIP Kits ELISA)-22-mediated MECOM (Montrer MECOM Kits ELISA) degradation increased c-Jun but decreased GATA2 (Montrer GATA2 Kits ELISA) expression, which results in increased interaction between c-Jun and PU.1

  6. Gene expression profiling and pathway analysis followed by pharmacological screening identified activated ERK (Montrer EPHB2 Kits ELISA) and JNK (Montrer MAPK8 Kits ELISA) signaling as key drivers of neurodegeneration in mutant SOD1 (Montrer SOD1 Kits ELISA) motor neurons. Genetic correction of SOD1 (Montrer SOD1 Kits ELISA) mutant iPSCs Reveals ERK (Montrer EPHB2 Kits ELISA) and JNK (Montrer MAPK8 Kits ELISA) Activated AP1 (Montrer FOSB Kits ELISA) as a Driver of Neurodegeneration in Amyotrophic Lateral Sclerosis.

  7. the absence of 14-3-3sigma leads to the nuclear accumulation and stabilization of c-Jun, suggesting that 14-3-3sigma regulates the subcellular localization of c-Jun.

  8. Suggest UCA1 and C-JUN mRNA as promising diagnostic and prognostic markers in hepatocellular carcinoma.

  9. During inflammatory conditions, AP-1 (Montrer FOSB Kits ELISA) and Sp1 (Montrer PSG1 Kits ELISA) sustained the expression of ADAMTS7 (Montrer ADAMTS7 Kits ELISA), and ADAMTS7 (Montrer ADAMTS7 Kits ELISA) sustained the expression of catabolic genes in nucleus pulposus cells

  10. our data strongly suggest that AQCA-mediated suppression of inflammatory responses could be managed by a direct interference of signaling cascades including IRAK (Montrer IRAK1 Kits ELISA) and Syk (Montrer SYK Kits ELISA), linked to the activation of NF-kappaB (Montrer NFKB1 Kits ELISA) and AP-1 (Montrer FOSB Kits ELISA).

Zebrafish Jun Proto-Oncogene (JUN) interaction partners

  1. These results support a role for trim69 (Montrer TRIM69 Kits ELISA) in the development of the zebrafish brain through ap-1 pathway.

  2. CPEB-1 (Montrer CPEB1 Kits ELISA) control of c-Jun mRNA translation regulates GH gene expression and resulting downstream signaling events (e.g., synaptic plasticity) in the mouse hippocampus.

Cow (Bovine) Jun Proto-Oncogene (JUN) interaction partners

  1. The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 DNA-binding activity and expression of several transcriptions factors in breast cancer cells.

  2. dynamic compression stimulates cell proliferation and proteoglycan (Montrer Vcan Kits ELISA) synthesis in the presence of IL-1beta (Montrer IL1B Kits ELISA) and/or inhibitors of the MAPKs and NFkappaB and AP-1 signalling pathways

C-JUN (JUN) profil antigène

Antigen Summary

This gene is the putative transforming gene of avian sarcoma virus 17. It encodes a protein which is highly similar to the viral protein, and which interacts directly with specific target DNA sequences to regulate gene expression. This gene is intronless and is mapped to 1p32-p31, a chromosomal region involved in both translocations and deletions in human malignancies.

Alternative names and synonyms associated with C-JUN (JUN)

  • jun oncogene (jun) Elisa Kit
  • basket (bsk) Elisa Kit
  • C-JUN protein (C-JUN) Elisa Kit
  • c-Jun protein (C-JUN) Elisa Kit
  • c-jun transcription factor (C-JUN) Elisa Kit
  • cyclin-dependent kinase 5, regulatory subunit 2 (p39) (Cdk5r2) Elisa Kit
  • Jun oncogene (Jun) Elisa Kit
  • jun proto-oncogene (JUN) Elisa Kit
  • jun proto-oncogene (jun) Elisa Kit
  • jun proto-oncogene (Jun) Elisa Kit
  • Jun-related antigen (Jra) Elisa Kit
  • AP-1 Elisa Kit
  • AP1 Elisa Kit
  • B230310J22Rik Elisa Kit
  • Bsk Elisa Kit
  • BSK/DJNK Elisa Kit
  • c-jun Elisa Kit
  • CG2275 Elisa Kit
  • CG5680 Elisa Kit
  • cJun Elisa Kit
  • D-JNK Elisa Kit
  • d-JRA Elisa Kit
  • d-jun Elisa Kit
  • D-jun/Jra Elisa Kit
  • D-junk Elisa Kit
  • dAP-1 Elisa Kit
  • DBSK/JNK Elisa Kit
  • dJNK Elisa Kit
  • DJNK/bsk Elisa Kit
  • dJra Elisa Kit
  • djun Elisa Kit
  • dm-Jun Elisa Kit
  • Dmel\\CG2275 Elisa Kit
  • Dmel\\CG5680 Elisa Kit
  • fj36h07 Elisa Kit
  • jnk Elisa Kit
  • JNK/SAPK Elisa Kit
  • jra Elisa Kit
  • jun Elisa Kit
  • Junc Elisa Kit
  • Junk Elisa Kit
  • l(2)46Ef Elisa Kit
  • l(2)IA109 Elisa Kit
  • l(2R)IA109 Elisa Kit
  • p39 Elisa Kit
  • SAPKa Elisa Kit
  • V Elisa Kit
  • wu:fj36h07 Elisa Kit
  • zgc:65863 Elisa Kit

Protein level used designations for Jun Proto-Oncogene (JUN) Kits ELISA

v-jun sarcoma virus 17 oncogene homolog , CG5680-PB , CG5680-PE , CG5680-PF , JNK , JUN kinase , Jun N-terminal kinase , Jun NH2-terminal kinase , Jun-N-terminal kinase , Jun-kinase , bsk-PB , bsk-PE , bsk-PF , c-Jun N-terminal kinase , c-Jun aminoterminal kinase , c-Jun-N-terminal kinase , drosophila JNK , AP1 , V-jun avian sarcoma virus 17 oncogene homolog , activator protein 1 , proto-oncogene c-jun , transcription factor AP-1 , CDK5 activator 2 , cyclin-dependent kinase 5 activator 2 , cyclin-dependent kinase 5 regulatory subunit 2 , p39I , AH119 , immediate early , jun A , Jun activation domain binding protein , enhancer-binding protein AP1 , jun oncogene , p39 , proto-oncogene c-Jun , v-jun avian sarcoma virus 17 oncogene homolog , Avian sarcoma virus 17 (v-jun) oncogene homolog , Jun oncogene , v-jun sarcoma virus 17 oncogene , CG2275-PA , CG2275-PB , CG2275-PC , Jra-PA , Jra-PB , Jra-PC , Jun related antigen , complementation group V , lethal(2)1A109 , lethal(2)IA109

379050 Xenopus laevis
44801 Drosophila melanogaster
396913 Sus scrofa
443219 Ovis aries
100008856 Oryctolagus cuniculus
12570 Mus musculus
16476 Mus musculus
3725 Homo sapiens
335916 Danio rerio
24516 Rattus norvegicus
399400 Xenopus laevis
424673 Gallus gallus
609429 Canis lupus familiaris
280831 Bos taurus
100170668 Ovis aries
36057 Drosophila melanogaster
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