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findings suggest that G5PR is involved in both the survival of B cells and the differentiation of peritoneal B1a cells into autoantibody-producing plasma cells.
G5PR is required for the B cell receptor-mediated proliferation associated with the prevention of activation-induced cell death in mature B cells.
G5PR regulates the thymocyte unique apoptotic signal involved in JNK (Montrer MAPK8 Kits ELISA)-mediated Caspase-3 (Montrer CASP3 Kits ELISA) activation but not in Bim (Montrer BCL2L11 Kits ELISA) activation.
Knockdown of PP5 (Montrer PPP5C Kits ELISA) and/or PPP2R3C increased P-gp (Montrer ABCB4 Kits ELISA) expression and lowered the sensitivity to vincristine and doxorubicin. Consequently, results indicate that PP5 (Montrer PPP5C Kits ELISA)/PPP2R3C negatively regulates P-gp (Montrer ABCB4 Kits ELISA) expression and function.
G5PR expression is markedly augmented in the centrocytes of germinal centers after immunization of transgenic mice with T cell-dependent antigen.
May regulate MCM3AP phosphorylation through phosphatase recruitment. May play a role in the activation-induced cell death of B-cells (By similarity).
phosphatase subunit gene g4-1
, protein phosphatase subunit G5PR
, serine/threonine-protein phosphatase 2A regulatory subunit B'' subunit gamma
, protein phosphatase 2 (formerly 2A), regulatory subunit B'', gamma
, rhabdomyosarcoma antigen MU-RMS-40.6A
, rhabdomyosarcoma antigen MU-RMS-40.6A/6C
, rhabdomyosarcoma antigen Mu-RMS-40.6C
, serine/threonine-protein phosphatase 2A regulatory subunit B'' gamma subunit