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showed that patients with SSc (Montrer CYP11A1 Kits ELISA) or SLE have AAb against EphB2, a protein involved in angiogenesis, and THEX1 (Montrer ERI1 Kits ELISA), a 3'-5' exoribonuclease involved in histone mRNA degradation. We have further identified a peptide from EphB2 as a specific and sensitive tool for SLE diagnosis
show that expression of EPHB2 and SNAIL1 (Montrer SNAI1 Kits ELISA) - an inducer of epithelial-mesenchymal transition (EMT (Montrer ITK Kits ELISA)) - is anti-correlated in colorectal cancer cell lines and tumors
Tiam2 (Montrer TIAM2 Kits ELISA)/Rac (Montrer AKT1 Kits ELISA) are key components of EphB2 trans-endocytosis and are important for cell repulsion.
High expression of junctional adhesion molecule-A (Montrer F11R Kits ELISA) and EphB2 can predict poor overall survival and high mortality rate, and EphB2 is an independent prognostic biomarker in lung adenocarcinoma patients.
Data show that activation of EphB2 receptor kinase arrests tau protein hyperphosphorylation through phosphatidylinositol 3-kinase (PI3K (Montrer PIK3CA Kits ELISA))/Akt (Montrer AKT1 Kits ELISA) protein-mediated glycogen synthase kinase-3beta (GSK-3beta (Montrer GSK3b Kits ELISA)) inhibition.
Expression of the Receptor Tyrosine Kinase (Montrer RET Kits ELISA) EphB2 on Dendritic Cells Is Modulated by Toll (Montrer TLR4 Kits ELISA)-Like Receptor Ligation but Is Not Required for T Cell Activation
Myosin 1 functions as an effector of EphB2/ephrinB signaling, controls cell morphology, and thereby cell repulsion.
EphB2 activation is required for ependymoma development as well as it inhibits differentiation and promotes proliferation of the transformed cell.
EphB2/ephrin-B1 (Montrer EFNB1 Kits ELISA) were invoked in dental pulp stem cells with TNF-alpha (Montrer TNF Kits ELISA) treatment via the JNK (Montrer MAPK8 Kits ELISA)-dependent pathway, but not NF-kB, p38 MAPK (Montrer MAPK14 Kits ELISA) or MEK (Montrer MAP2K1 Kits ELISA) signalling.
The results show an intricate interplay between p53 (Montrer TP53 Kits ELISA) and TGF-beta3 (Montrer TGFB3 Kits ELISA) whereby p53 (Montrer TP53 Kits ELISA) inhibits the TGF-beta3 (Montrer TGFB3 Kits ELISA)-induced expression of genes, e.g., EPHB2, to impede tumor cell invasion and migration
ephrin B3 (Montrer EFNB3 Kits ELISA)/EphB2 are obvious candidates for driving the Syk (Montrer SYK Kits ELISA)-dependent repulsive response.
The results of this study indicated that the decrease in spine density in the mPFC was associated with susceptibility to stress, and EphB2 downregulation in the mPFC increased the vulnerability to stress.
We here identify that EphB2 receptor tyrosine kinase (Montrer ERBB3 Kits ELISA), which is specifically expressed in glutamatergic neurons, is required for the innate fear responses in the neonatal brain.
Here, we identified the interaction sites of the EphB2 FN domain with ADDLs for the first time to develop a small (10 aa) peptide (Pep63) capable of blocking the EphB2-ADDL (Montrer ADD3 Kits ELISA) interaction.
EphB4 (Montrer EPHB4 Kits ELISA) plays an irreplaceable role in bone regeneration in an inflammatory microenvironment, whereas the functional loss of ephrinB2 (Montrer EFNB2 Kits ELISA) can be effectively compensated, most possibly by other ephrins with similar chemical structures
Authors suggest that aging is accompanied by the upregulation of miR-204 in the hippocampus, which downregulates EphB2 and results in reduced surface and total NR1 expression.
Findings suggest that a combination of forward and reverse EphB1 (Montrer EPHB1 Kits ELISA)/2 receptor-mediated signaling contribute to posterior branch of the anterior commissure and corpus callosum axon guidance
Results demonstrate that EphB2 reverse signaling plays a unique and requisite role in inhibiting the development of opiate-dependent tolerance in vivo
During re-epithelialization ephrin-B1 (Montrer EFNB1 Kits ELISA) and its receptor EphB2 are both upregulated in vivo, just for the duration of repair.
EphB2 prevents amyloid-beta-induced depletion of cell surface GluN1 (Montrer GRIN1 Kits ELISA) requiring the PDZ (Montrer INADL Kits ELISA)-binding motif of EphB2.
Ephrin receptors and their ligands, the ephrins, mediate numerous developmental processes, particularly in the nervous system. Based on their structures and sequence relationships, ephrins are divided into the ephrin-A (EFNA) class, which are anchored to the membrane by a glycosylphosphatidylinositol linkage, and the ephrin-B (EFNB) class, which are transmembrane proteins. The Eph family of receptors are divided into 2 groups based on the similarity of their extracellular domain sequences and their affinities for binding ephrin-A and ephrin-B ligands. Ephrin receptors make up the largest subgroup of the receptor tyrosine kinase (RTK) family. The protein encoded by this gene is a receptor for ephrin-B family members.
ephrin receptor EphB2
, EPH receptor B2
, ephrin type-B receptor 2-like
, EPH-like kinase 5
, developmentally-regulated Eph-related tyrosine kinase
, elk-related tyrosine kinase
, eph tyrosine kinase 3
, ephrin type-B receptor 2
, protein-tyrosine kinase HEK5
, renal carcinoma antigen NY-REN-47
, tyrosine-protein kinase TYRO5
, tyrosine-protein kinase receptor EPH-3
, neural kinase
, nuk receptor tyrosine kinase
, tyrosine-protein kinase receptor SEK-3
, embryo kinase 5 protein CEK5