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anti-Mouse (Murine) NFATC4 Anticorps:
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Human Polyclonal NFATC4 Primary Antibody pour IF (p), IHC (p) - ABIN1386186
Xie, Deng, Pan, Ren, Jin, Wang, Wang, Zhu, Guo, Yuan, Shang, Liu: Chronic intermittent hypoxia induces cardiac hypertrophy by impairing autophagy through the adenosine 5'-monophosphate-activated protein kinase pathway. dans Archives of biochemistry and biophysics 2016
Human Polyclonal NFATC4 Primary Antibody pour IHC (p), IHC - ABIN249616
Hoey, Sun, Williamson, Xu: Isolation of two new members of the NF-AT gene family and functional characterization of the NF-AT proteins. dans Immunity 1995
Dog (Canine) Polyclonal NFATC4 Primary Antibody pour ELISA - ABIN450047
Yao, Cho, Bergen, Madden, Choi, Ma, Bode, Dong: Nuclear factor of activated T3 is a negative regulator of Ras-JNK1/2-AP-1 induced cell transformation. dans Cancer research 2007
Ca(2 (Montrer CA2 Anticorps)+)/calcineurin (CaN)/nuclear factor of activated T-cells (NFAT (Montrer NFATC1 Anticorps)) c4 axis is required for neuritin (Montrer NRN1 Anticorps)-induced Kv4.2 (Montrer KCND2 Anticorps) transcriptional expression and potentiation of IA densities in cerebellum granule neurons.
Nfatc4 expression was coordinately upregulated by top hypoxia-activated transcription factors, while NFATc4 target genes were enriched in hypoxic neural stem cells.
results indicate that NFAT (Montrer NFATC1 Anticorps) is a BACE1 (Montrer BACE Anticorps) transcription factor. Our study suggests that inhibition of NFAT (Montrer NFATC1 Anticorps)-mediated BACE1 (Montrer BACE Anticorps) expression may be a valuable drug target for AD therapy
our results suggest that activation of NHE1 (Montrer SLC9A1 Anticorps) induces hypertrophy through the activation of NFAT3/Gata4 (Montrer GATA4 Anticorps) and OPN (Montrer SPP1 Anticorps) expression
L-carnitine reversed the activation of NFAT3 and PPARa (Montrer PPARA Anticorps) caused by carboplatin through altering the phosphorylation of PTEN (Montrer PTEN Anticorps) and AMPK (Montrer PRKAA1 Anticorps).
Disrupting the calcineurin-NFAT (Montrer NFATC1 Anticorps) axis by either genetic or pharmacologic approaches confers resistance to the development of social stress-induced voiding and dysfunction.
The NFATc4-dependent increase in hippocampal neurogenesis after GABAA (Montrer GABRg1 Anticorps) receptor stimulation is required for suppression of anxiety response.
IL-13 (Montrer IL13 Anticorps) specifically induced NFAT3 activation through promoting its dephosphorylation in air-liquid interface cultures of tracheal epithelial cells (mTECs).
Data suggest that expression of Itpr1 (inositol 1,4,5-triphosphate receptor 1 (Montrer ITPR1 Anticorps)) in cerebral cortex neurons is regulated by dopamine D2 receptors; Itpr1 (Montrer ITPR1 Anticorps) is up-regulated via increased binding of NFATc4 and transcription factor AP-1 (Montrer JUN Anticorps) to Itpr1 (Montrer ITPR1 Anticorps) promoter.
these results reveal a novel PI3K-independent role for p85alpha in controlling VEGF (Montrer VEGFA Anticorps) induction during the cellular UVB response by regulating NFAT3 activity.
These results provided evidence supporting the oncogenic potential of NFAT3 and suggested that CDK3 (Montrer CDK3 Anticorps)-mediated phosphorylation of NFAT3 has an important role in skin tumorigenesis.
Data indicate that RNA interference of NFAT (Montrer NFATC1 Anticorps) isoforms NFATc1 (Montrer NFATC1 Anticorps), NFATc2 (Montrer NFAT1 Anticorps), NFATc3 (Montrer NFATC3 Anticorps) and NFATc4 regulate gene expression differentially in human retinal microvascular endothelial cells (HRMEC).
NFAT3 expression plays a role in regulating CXCR4 (Montrer CXCR4 Anticorps) expression.
Suggest nuclear NF-AT3 and NF-AT4 (Montrer NFATC3 Anticorps) participates in atrial structural remodeling, and that PICP and TGF-beta1 (Montrer TGFB1 Anticorps) levels may be sensitive serum biomarkers to estimate atrial structural remodeling with atrial fibrillation.
This is the first study to provide evidence of new and differential roles for NFAT3 and SMAD3 (Montrer SMAD3 Anticorps) in the osteoarthritis process in the regulation of miR (Montrer MLXIP Anticorps)-140 transcription
Expression level of PPP3R1 (Montrer PPP3R1 Anticorps) and GATA4 (Montrer GATA4 Anticorps), and NFATC4 genes for transcription factors did not differ in studied subgroups of patients.
Data indicate that NFATc3 (Montrer NFATC3 Anticorps) undergoes rapid dephosphorylation and nuclear translocation that are essentially complete within 20 min, although NFATc4 remains phosphorylated and localized to the cytosol.
Syndecan-4 (Montrer SDC4 Anticorps) is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT (Montrer NFATC1 Anticorps) pathway
Dendritic spine loss and dendritic branching simplification induced by amyloid-beta peptide exposure are mimicked by constitutively active NFATC4, and abolished when NFATC4 activation is blocked by the genetically encoded inhibitor VIVIT.
The product of this gene is a member of the nuclear factors of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation and an inducible nuclear component. Other members of this family of nuclear factors of activated T cells also participate in the formation of this complex. The product of this gene plays a role in the inducible expression of cytokine genes in T cells, especially in the induction of the IL-2 and IL-4. Alternatively spliced transcript variants encoding different isoforms have been noted for this gene.
cytoplasmic nuclear factor of activated T-cells 4
, nuclear factor of activated T-cells, cytoplasmic, calcineurin-dependent 4
, nuclear factor of activated T-cells, cytoplasmic 4-like
, T-cell transcription factor NFAT3
, nuclear factor of activated T-cells, cytoplasmic 4
, transcription complex subunit NF-ATc4
, T cell transcription factor NFAT3