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Human TEK Kit ELISA pour Sandwich ELISA - ABIN625097
Zhang, Lin, Jiang, Xu, Luo, Mo, Li, Chen: Extensive serum biomarker analysis in patients with ST segment elevation myocardial infarction (STEMI). dans Cytokine 2015
Human TEK Kit ELISA pour Sandwich ELISA - ABIN411357
Zheng, Zhu, Bai, Wu, Jia, Hu: Exercise improves recovery after ischemic brain injury by inducing the expression of angiopoietin-1 and Tie-2 in rats. dans The Tohoku journal of experimental medicine 2011
Show all 6 Pubmed References
These results indicate that the Angpt-Tie2 system is essential for SC integrity. The impairment of this system underlies POAG-associated pathogenesis, supporting the possibility that Tie2 agonists could be a therapeutic option for glaucoma.
TEK mutations have a role in primary congenital glaucoma with variable expressivity
ANG2 (Montrer ANGPT2 Kits ELISA) activation of Tie2 supports stable enlargement of normal nonleaky vessels, but reduction of Tie1 (Montrer TIE1 Kits ELISA) in inflammation leads to ANG2 (Montrer ANGPT2 Kits ELISA) antagonism of Tie2 and initiates a positive feedback loop wherein FOXO1 (Montrer FOXO1 Kits ELISA)-driven ANG2 (Montrer ANGPT2 Kits ELISA) expression promotes vascular remodeling and leakage
Hydroxysafflor yellow A promotes angiogenesis in ischemic hindlimb via the angiopoi (Montrer ANGPT1 Kits ELISA)etin 1/ Tie-2 signaling pathway.
TBMS1 further stimulated the proteasomal degradation of vascular endothelial growth factor receptor-2 (VEGFR2 (Montrer KDR Kits ELISA)) and Tie2 in endothelial cells, which down-regulated AKT (Montrer AKT1 Kits ELISA)/mTOR (Montrer FRAP1 Kits ELISA) signaling.
results implicate common genetic variation at the TIE2 locus as a determinant of vascular leak-related clinical outcomes from common infections, suggesting new tools to identify individuals at unusual risk for deleterious complications of infection
the results suggest that an increased level of TNF-alpha (Montrer TNF Kits ELISA) together with concomitant upregulation of Tie2/Angiopoietin signaling have critical roles in severe dengue infection.
Data support an interactive model of Tie1 (Montrer TIE1 Kits ELISA) and Tie2 function, in which dynamically regulated Tie1 (Montrer TIE1 Kits ELISA) versus Tie2 expression determines the net positive or negative effect of Tie1 (Montrer TIE1 Kits ELISA) on Tie2 signaling.
These results suggest that Tie2 signaling induces alpha4beta1 integrin activation on bone marrow-mast cell progenitor for adhesion to VCAM-1 (Montrer VCAM1 Kits ELISA).
Report accelerated lung regeneration by platelet-rich plasma extract through Lrp5 (Montrer LRP5 Kits ELISA)/Tie2 pathway.
Results show that TIE2 phosphorylates caveolin-1 (Montrer CAV1 Kits ELISA) at Tyr14, and associates with caveolin-1 (Montrer CAV1 Kits ELISA) in caveolae. Also, its nuclear translocation is caveolin-1 (Montrer CAV1 Kits ELISA) dependent.
Tie1 (Montrer TIE1 Kits ELISA) directly interacts with Tie2 to promote ANG (Montrer ANG Kits ELISA)-induced vascular responses under noninflammatory conditions, whereas in inflammation, Tie1 (Montrer TIE1 Kits ELISA) cleavage contributes to loss of ANG2 (Montrer ANGPT2 Kits ELISA) agonist activity and vascular stability
ANG-1 (Montrer ANGPT1 Kits ELISA), ANG-2 (Montrer ANGPT2 Kits ELISA) and TIE-2 levels were significantly increased in placenta of non-complicated ART pregnancies compared to placentas from spontaneous conception.
Our data suggest that interaction of TEK and CYP1B1 (Montrer CYP1B1 Kits ELISA) contributes to primary congenital glaucoma pathogenesis and argue that TEK-CYP1B1 (Montrer CYP1B1 Kits ELISA) may perform overlapping as well as distinct functions in manifesting the disease etiology.
High Tie-2 expression is associated with Primary Myelofibrosis.
In this study, we found that angiopoietins and Tie receptors were highly expressed in cervical cancer cells. Tie-2 expression in tumor cells predicted poorer prognosis.Our data support that dual inhibition of Ang-1 (Montrer ANGPT1 Kits ELISA) and Ang-2 (Montrer ANGPT2 Kits ELISA) may be an alternative target for anti-angiogenic adjuvant therapy in advanced or recurrent cervical squamous cell cancer.
Data show that fibulin-5 (Montrer FBLN5 Kits ELISA) strongly binds to the endothelial cell surface reducing endothelial cell viability and interfering with the signaling pathways of the Ang-1 (Montrer ANGPT1 Kits ELISA)/TIE-2 receptor axis.
Blue rubber bleb nevus syndrome cased by somatic mutation of TEK.
Angiopoietin-1 receptor Tie2 distinguishes multipotent differentiation capability in bovine coccygeal nucleus pulposus cells.
observations demonstrate that Tie2 is an important regulator of tip cell behaviors
Tie-1 (Montrer TIE1 Kits ELISA) and Tie-2 are not required for early heart development, yet they have redundant roles for the maintenance of endocardial-myocardial connection in later stages.
The TEK receptor tyrosine kinase is expressed almost exclusively in endothelial cells in mice, rats, and humans. This receptor possesses a unique extracellular domain containing 2 immunoglobulin-like loops separated by 3 epidermal growth factor-like repeats that are connected to 3 fibronectin type III-like repeats. The ligand for the receptor is angiopoietin-1. Defects in TEK are associated with inherited venous malformations\; the TEK signaling pathway appears to be critical for endothelial cell-smooth muscle cell communication in venous morphogenesis. TEK is closely related to the TIE receptor tyrosine kinase.
growth factor receptor Tie2/Tek
, TEK tyrosine kinase, endothelial (venous malformations, multiple cutaneous and mucosal)
, Angiopoietin-1 receptor
, TEK tyrosine kinase, endothelial
, angiopoietin-1 receptor-like
, angiopoietin-1 receptor
, endothelial tyrosine kinase
, p140 TEK
, tunica interna endothelial cell kinase
, tyrosine kinase with Ig and EGF homology domains-2
, tyrosine-protein kinase receptor TEK
, tyrosine-protein kinase receptor TIE-2
, soluble TIE2 variant 1
, soluble TIE2 variant 2
, protein-tyrosine kinase Tie2
, endothelial-specific receptor tyrosine kinase
, tyrosine kinases that contain immunoglobulin-like loops and epidermal growth factor-similar domains 2
, endothelium-specific receptor tyrosine kinase 2
, tyrosine-protein kinase receptor Tie-2