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Rat (Rattus) Angiotensin II Kit ELISA pour Competition ELISA - ABIN416068
Liu, Hu, Wang, Zhang, Ma, Feng, Wang, Wang, Dong, Gao, Zhang, Zhang: Angiotensin-converting enzyme (ACE) 2 overexpression ameliorates glomerular injury in a rat model of diabetic nephropathy: a comparison with ACE inhibition. dans Molecular medicine (Cambridge, Mass.) 2011
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Human Angiotensin II Kit ELISA pour Competition ELISA - ABIN414350
Zhang, Long, Sun, Wang, Li, Wu, Guo, Li, Niu, Li, Liu, Mei et al.: Evidence for the complementary and synergistic effects of the three-alkaloid combination regimen containing berberine, hypaconitine and skimmianine on the ulcerative colitis rats induced by ... dans European journal of pharmacology 2010
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Mouse (Murine) Angiotensin II Kit ELISA pour Competition ELISA - ABIN415355
Wang, Liu, Ye, Zhang, Tang, Chen, Zhang, Lou: Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes. dans European journal of clinical investigation 2009
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Rat (Rattus) Angiotensin II Kit ELISA pour Sandwich ELISA - ABIN368044
Simic, Hermann, Shaw, Bigler, Stalder, Dörries, Besler, Lüscher, Ruschitzka: Torcetrapib impairs endothelial function in hypertension. dans European heart journal 2011
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Mouse (Murine) Angiotensin II Kit ELISA pour Sandwich ELISA - ABIN366511
Than, Leow, Chen: Control of adipogenesis by the autocrine interplays between angiotensin 1-7/Mas receptor and angiotensin II/AT1 receptor signaling pathways. dans The Journal of biological chemistry 2013
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Human Angiotensin II Kit ELISA pour Sandwich ELISA - ABIN366510
Mao, Cao, Lan, He, Chen, Wang, Hu, Lv, Wang, Yan: Therapeutic effect of forest bathing on human hypertension in the elderly. dans Journal of cardiology 2012
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Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (Montrer AGT Kits ELISA) and renin (Montrer REN Kits ELISA) excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Quaternary interactions and supercoiling modulate the cooperative DNA binding of AGT (Montrer AGXT Kits ELISA).
results show that SNPs in the Hap (Montrer SAFB Kits ELISA)-I of the hAGT gene promote high-fat diet-induced binding of transcription factors GR, CEBP-beta (Montrer CEBPB Kits ELISA) and STAT3 (Montrer STAT3 Kits ELISA), which lead to elevated expression of the hAGT gene in hepatic and adipose tissues
Angiotensinogen (Montrer AGT Kits ELISA) import and subsequent trafficking to the mitochondria occurs in proximal kidney tubules.
Transgenic mice expressing human AGT (Montrer AGXT Kits ELISA) in the subfornical organ AGT (Montrer AGXT Kits ELISA) and possibly ANG I/ANG II (Montrer AGT Kits ELISA) into the cerebral ventricles.
AngII could induce pulmonary injury by triggering endothelial barrier injury, and such process may be related to the dephosphorylation of Y685-VE-cadherin (Montrer CDH5 Kits ELISA) and the endothelial skeletal rearrangement
Renin-angiotensin system transgenic mouse model suggests that renal injury in preeclampsia may be mediated through local VEGF.
endoplasmic reticulum stress induces apoptosis in human alveolar epithelial cells through mediation of unfolded protein response pathways, which in turn regulate the autocrine ANGII/ANG1 (Montrer ANGPT1 Kits ELISA)-7 system.
Angiotensin II stimulates PYY secretion, in turn inhibiting epithelial anion fluxes, thereby reducing net fluid secretion into the colonic lumen.
NOXs had two time-dependent reactions in response to Ang II (Montrer AGT Kits ELISA) stimulation via MAPK (Montrer MAPK1 Kits ELISA) pathwa
this study demonstrated that Ang II (Montrer AGT Kits ELISA) could increase TRPC6 (Montrer TRPC6 Kits ELISA) induced Ca(2 (Montrer CA2 Kits ELISA)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (Montrer AGT Kits ELISA)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (Montrer AGTRAP Kits ELISA).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (Montrer AGTRAP Kits ELISA)) in the inflammation induced by Aah (Montrer ASPH Kits ELISA) venom, in the heart and the aorta.
expression of spinal ACE (Montrer ACE Kits ELISA) increased in streptozotocin-induced diabetic mice, which in turn led to an increase in Ang II (Montrer AGT Kits ELISA) levels and tactile allodynia.
the beneficial actions of insulin (Montrer INS Kits ELISA) in diabetic nephropathy appear to be mediated, in part, by suppressing renal Nrf2 (Montrer NFE2L2 Kits ELISA) and Agt (Montrer AGXT Kits ELISA) gene transcription and preventing Nrf2 (Montrer NFE2L2 Kits ELISA) stimulation of Agt (Montrer AGXT Kits ELISA) expression via hnRNP F (Montrer HNRNPF Kits ELISA)/K.
Angiotensinogen (Montrer AGT Kits ELISA)-mediated downregulation of aquaporin 1 (Montrer AQP1 Kits ELISA) and Nrf2 (Montrer NFE2L2 Kits ELISA) signalling may play an important role in intrarenal renin (Montrer REN Kits ELISA)-angiotensin system-induced hypertension and kidney injury.
This study suggests that deletion of AT2R decreases the expression of the beneficial ACE2/Ang-(1-7)/MasR.
an inverse correlation was found between Ang-(1 (Montrer ANGPT1 Kits ELISA)-7) level and tau hyperphosphorylation, a pathological hallmark of Alzheimer's disease, in cerebral cortex and hippocampus of SAMP8 mice.
The inhibition of pathological autophagy in the heart in response to chronic Ang II (Montrer AGT Kits ELISA) by Interleukin-10 (Montrer IL10 Kits ELISA), and its implications, has been described.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll