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Rat (Rattus) Angiotensin II Kit ELISA pour Competition ELISA - ABIN416068
Liu, Hu, Wang, Zhang, Ma, Feng, Wang, Wang, Dong, Gao, Zhang, Zhang: Angiotensin-converting enzyme (ACE) 2 overexpression ameliorates glomerular injury in a rat model of diabetic nephropathy: a comparison with ACE inhibition. dans Molecular medicine (Cambridge, Mass.) 2011
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Human Angiotensin II Kit ELISA pour Competition ELISA - ABIN414350
Zhang, Long, Sun, Wang, Li, Wu, Guo, Li, Niu, Li, Liu, Mei et al.: Evidence for the complementary and synergistic effects of the three-alkaloid combination regimen containing berberine, hypaconitine and skimmianine on the ulcerative colitis rats induced by ... dans European journal of pharmacology 2010
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Mouse (Murine) Angiotensin II Kit ELISA pour Competition ELISA - ABIN415355
Wang, Liu, Ye, Zhang, Tang, Chen, Zhang, Lou: Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes. dans European journal of clinical investigation 2009
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NOXs had two time-dependent reactions in response to Ang II (Montrer AGT Kits ELISA) stimulation via MAPK (Montrer MAPK1 Kits ELISA) pathwa
increased Ang-II/Ang-(1 (Montrer ANGPT1 Kits ELISA)-7) ratios may lead to Ang-II (Montrer AGT Kits ELISA) over-activation and aggravate atherosclerosis progression.
urinary angiotensinogen (Montrer AGT Kits ELISA) could be a prognostic marker of the albuminuria reduction effects of olmesartan in patients with metabolic syndrome
AGT (Montrer AGXT Kits ELISA) expression is significantly upregulated in human masticatory mucosa during wound healing
Urinary angiotensinogen reflects intrarenal RAS status, and is of value to assess the severity of acute tubular necrosis .
Data suggest that enhanced survival of normal prostate epithelial cells (PNT1A cell line) in presence of either or both angiotensin II (Ang II) and relaxin 2 (RLN2 (Montrer RLN1 Kits ELISA)) appears to be associated with increased ratio of BCL2 (Montrer BCL2 Kits ELISA)/BAX (Montrer BAX Kits ELISA) messenger RNA expression; thus, Ang II (Montrer AGT Kits ELISA) and/or RLN2 (Montrer RLN1 Kits ELISA) appear to be involved in regulation of apoptosis in prostate epithelium.
increasing the serum levels of AngII increased the risk of acute myocardial infarction (AMI (Montrer CFD Kits ELISA)); the risk of AMI (Montrer CFD Kits ELISA) increased when the serum levels of AngII and KLK1 (Montrer KLK1 Kits ELISA) simultaneously increased; individuals with the combined genotypes of ACE (Montrer ACE Kits ELISA) DD and KLK1 (Montrer KLK1 Kits ELISA) GG showed increased risk of AMI (Montrer CFD Kits ELISA) compared with those with the combined genotypes of ACE (Montrer ACE Kits ELISA) II and KLK1 (Montrer KLK1 Kits ELISA) AA
genetic association study and meta-analysis revealed a significant association between M235T polymorphism of angiotensinogen (Montrer AGT Kits ELISA) and myocardial infarction risk
The aim of this work is to assess the influence of the rs1799752 polymorphism of the angiotensin-converting enzyme (ACE (Montrer ACE Kits ELISA)) gene and the rs699 polymorphism of the angiotensinogen (AGT (Montrer AGT Kits ELISA)) gene on the in-stent restenosis in Polish patients with stable coronary artery disease.
in cultured H9C2 cells and that CyPA (Montrer PPIA Kits ELISA)/CD147 signaling pathway inhibition protects against Ang II (Montrer AGT Kits ELISA)-induced cardiomyocyte hypertrophy.
the beneficial actions of insulin (Montrer INS Kits ELISA) in diabetic nephropathy appear to be mediated, in part, by suppressing renal Nrf2 (Montrer NFE2L2 Kits ELISA) and Agt (Montrer AGXT Kits ELISA) gene transcription and preventing Nrf2 (Montrer NFE2L2 Kits ELISA) stimulation of Agt (Montrer AGXT Kits ELISA) expression via hnRNP F (Montrer HNRNPF Kits ELISA)/K.
Angiotensinogen (Montrer AGT Kits ELISA)-mediated downregulation of aquaporin 1 (Montrer AQP1 Kits ELISA) and Nrf2 (Montrer NFE2L2 Kits ELISA) signalling may play an important role in intrarenal renin (Montrer REN Kits ELISA)-angiotensin system-induced hypertension and kidney injury.
This study suggests that deletion of AT2R decreases the expression of the beneficial ACE2/Ang-(1-7)/MasR.
an inverse correlation was found between Ang-(1 (Montrer ANGPT1 Kits ELISA)-7) level and tau hyperphosphorylation, a pathological hallmark of Alzheimer's disease, in cerebral cortex and hippocampus of SAMP8 mice.
The inhibition of pathological autophagy in the heart in response to chronic Ang II (Montrer AGT Kits ELISA) by Interleukin-10 (Montrer IL10 Kits ELISA), and its implications, has been described.
These results suggest that increased formation of AT1R (Montrer AGTRAP Kits ELISA)-P2Y6R (Montrer P2RY6 Kits ELISA) heterodimers with age may increase the likelihood of hypertension induced by Ang II (Montrer AGT Kits ELISA).
These data suggest dysregulation of renin (Montrer REN Kits ELISA)-angiotensin system in acute pancreatitis as evidenced by altered Ang II/Ang-(1 (Montrer ANGPT1 Kits ELISA)-7) levels induced by the imbalance of ACE (Montrer ACE Kits ELISA)/ACE2 (Montrer ACE2 Kits ELISA) activity.
Results indicate that Ang III (Montrer AGT Kits ELISA) produces nociceptive behavior similar to Ang II (Montrer AGT Kits ELISA), and suggest that the phosphorylation of p38 MAPK (Montrer MAPK14 Kits ELISA) mediated through AT1 (Montrer SLC33A1 Kits ELISA) receptors on spinal astrocytes and neurons contributes to Ang II (Montrer AGT Kits ELISA)- and III-induced nociceptive behavior
SIRT1 (Montrer SIRT1 Kits ELISA) and the axis AII (Montrer ARG2 Kits ELISA)/AT1 (Montrer SLC33A1 Kits ELISA)/NADPH-oxidase (Montrer NOX1 Kits ELISA) regulate each other
Angiotensin (1-7) and Bradykinin may have roles in diabetic nephropathy induced by increased ACE (Montrer ACE Kits ELISA) gene dosage
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll