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Mouse (Murine) IL9 Kit ELISA pour Sandwich ELISA - ABIN625153
Jimeno, Leceta, Martínez, Gutiérrez-Cañas, Pérez-García, Carrión, Gomariz, Juarranz: Effect of VIP on the balance between cytokines and master regulators of activated helper T cells. dans Immunology and cell biology 2012
Group 2 innate lymphoid cells utilize the IRF4 (Montrer IRF4 Kits ELISA)-IL-9 module to coordinate epithelial cell maintenance of lung homeostasis.
Our results suggest that The Th9/IL-9 is involved in the pathogenesis of UC.
IL-9 exerted pro-atherosclerotic effects in ApoE (Montrer APOE Kits ELISA)-/- mice at least partially by inducing VCAM-1 (Montrer VCAM1 Kits ELISA) expression, which mediated inflammatory cell infiltration into atherosclerotic lesions.
IL-9 is dispensable for mucosal mast cell development but is necessary for their effective expansion to promote intestinal mastocytosis and susceptibility to experimental food allergy in an IL-9-dependent autocrine manner.
IL-9 production following chlamydial lung infection is redundant for host defense against the intracellular bacteria
IL-25 (Montrer IL25 Kits ELISA)-stimulated dendritic cells rapidly induced mediators, such as the chemokine (Montrer CCL1 Kits ELISA) CCL17 (Montrer CCL17 Kits ELISA), which, in turn, attracted IL-9-producing T cells
Data show that the expression of interleukin-9 (IL-9) was higher, but interleukin-10 (IL-10 (Montrer IL10 Kits ELISA)) was lower in asthma group, and that Th9 cells might play an important role in the pathogenesis of asthma.
TH9 cells are a major source of IL-9 in models of allergic inflammation and play an important role in mast cell accumulation and activation.
IRF4 (Montrer IRF4 Kits ELISA) binds to the promoters of IL1b (Montrer IL1B Kits ELISA) and IL9, suggesting that tick-derived sialostatin L suppresses mast cell-derived IL-9 preferentially by inhibiting IRF4 (Montrer IRF4 Kits ELISA).
these data identify TL1A (Montrer TNFSF15 Kits ELISA)-DR3 (Montrer TNFRSF25 Kits ELISA) interactions as a novel pathway that promotes Th9 differentiation and pathogenicity. TL1A (Montrer TNFSF15 Kits ELISA) may be a potential therapeutic target in diseases dependent on IL-9.
IL-9 promotes proliferation and metastasis in pancreatic cancer cells; this effect may partly involve regulation of the miR-200a/b-catenin axis.
in PsA patients gammadelta T cells activation is driven prevalently by IL-9/IL-9R interaction, and not only by IL-23/IL-23R. Together these findings indicate gammadelta T cells and IL-9 as new players in the pathogenesis of PsA.
Th9 cells produce IL9 that may participate in pathogenesis of Takayasu's arteritis
IL-9 serum leves are elevated in patients with systemic lupus erythematosus, rheumatoid arthritis and systemic sclerosis but their clinical significance is unknown. [review]
Th9 cell numbers and IL-9 levels are correlated with OA patient symptoms and joint functionality
IL-9 is functionally active, and is a pro-growth/survival factor for the localized pathologic T cells in the synovium of inflammatory arthritis.
Asthmatic children showed an increase in plasma Il9 levels post Mycoplasma pneumoniae infection.
Interleukin-9 Is Associated with Elevated Anti-Double-Stranded DNA Antibodies in Lupus-Prone Mice
Data demonstrated increased serum levels of IL-9 in systemic lupus erythematosus and rheumatoid arthritis patients but no association with clinical and laboratory parameters was found.
show the regulation of the IL-9-STIM1 (Montrer STIM1 Kits ELISA)-ERK (Montrer EPHB2 Kits ELISA)-IL-8 (Montrer IL8 Kits ELISA) axis in keratinocyte, and how the axis might play an important role in the pathophysiology of atopic dermatitis
The protein encoded by this gene is a cytokine that acts as a regulator of a variety of hematopoietic cells. This cytokine stimulates cell proliferation and prevents apoptosis. It functions through the interleukin 9 receptor (IL9R), which activates different signal transducer and activator (STAT) proteins and thus connects this cytokine to various biological processes. The gene encoding this cytokine has been identified as a candidate gene for asthma. Genetic studies on a mouse model of asthma demonstrated that this cytokine is a determining factor in the pathogenesis of bronchial hyperresponsiveness.
, T-cell growth factor P40
, cytokine P40
, T-cell growth factor p40
, homolog of mouse T cell and mast cell growth factor 40
, p40 T-cell and mast cell growth factor
, p40 cytokine