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Human Polyclonal STX16 Primary Antibody pour ICC, IHC - ABIN1742232
Sadakata, Shinoda, Sekine, Saruta, Itakura, Takahashi, Furuichi et al.: Interaction of calcium-dependent activator protein for secretion 1 (CAPS1) with the class II ADP-ribosylation factor small GTPases is required for dense-core vesicle trafficking in the trans-Golgi ... dans The Journal of biological chemistry 2010
Show all 11 Pubmed References
syntaxin 16 may selectively regulate Golgi dynamics
Syntaxin 16 regulates lumen formation during kidney epithelial morphogenesis.
Syntaxin 16 may thus play a role in neurite outgrowth and perhaps other specific dendritic anterograde/retrograde traffic.
we here present a patient with PHP1b (Montrer GNAS Anticorps) caused by a recurrent STX16 deletion, presenting with macrosomia, early onset obesity, and macrocephaly without other AHO (Montrer GNAS Anticorps) symptoms. we reemphasize STX16 deletions and PHP1b (Montrer GNAS Anticorps) as a rare cause for early onset obesity and macrosomia.
STX16 microdeletion was identified in male monozygotic twins (with pseudohypoparathyroidism type 1B leading to growth hormone deficiency) and mother/grandmother (not father/grandfather or sister [their triplet with separate placenta]). [CASE STUDY]
syntaxin 16 is a key regulator of cytokinesis.
Data indicate that depletion of VAMP4 (Montrer VAMP4 Anticorps), syntaxin 6 (Montrer STX6 Anticorps), syntaxin 16, and Vti1a (Montrer VTI1A Anticorps) disrupted the Golgi ribbon structure.
A patient with familial pseudohypoparathyroidism type Ib and his asymptomatic brother were found to have methylation defect at GNAS (Montrer GNAS Anticorps) (guanine nucleotide-binding protein (Montrer COASY Anticorps) G) and microdeletion involving exons 4-6 of neighboring gene STX16. [CASE REPORT]
De novo 3-kb STX16 deletions, reported only once previously, are infrequent but should be excluded in all cases of Pseudohypoparathyroidism-Ib, even when the family history is negative for an inherited form of this disorder.
Results suggest that STX16 mediates recycling of CFTR (Montrer CFTR Anticorps) and constitutes an important component of CFTR (Montrer CFTR Anticorps) trafficking machinery in intestinal epithelial cells.
the region of overlap between the two microdeletions likely harbors a cis (Montrer CISH Anticorps)-acting imprinting control element that is necessary for establishing and methylation at GNAS (Montrer GNAS Anticorps) exon A/B, thus allowing normal G alpha(s (Montrer GNAS Anticorps)) expression in the proximal renal tubules. I
function of syntaxin 16 was specifically required for, and restricted to, the retrograde pathway
This gene encodes a protein that is a member of the syntaxin or t-SNARE (target-SNAP receptor) family. These proteins are found on cell membranes and serve as the targets for V-SNARES (vesicle-SNAP receptors) permitting specific synaptic vesicle docking and fusion. A microdeletion in the region of chromosome 20 where this gene is located has been associated with pseudohypoparathyroidism type Ib. Multiple transcript variants have been found for this gene. Read-through transcription also exists between this gene and the neighboring downstream aminopeptidase-like 1 (NPEPL1) gene.
, syntaxin 16
, syntaxin 16 a
, Syntaxin 16