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Human CP Kit ELISA pour Sandwich ELISA - ABIN414431
Li, Zheng, Huang, Lin, Zhao, Liao, Zhao, Lin, Gu: Screening disease-associated proteins from sera of patients with rheumatoid arthritis: a comparative proteomic study. dans Chinese medical journal 2010
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Costantini, Casasole, Eens: Does reproduction protect against oxidative stress? dans The Journal of experimental biology 2014
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Rat (Rattus) CP Kit ELISA pour Competition ELISA - ABIN2683892
Møller, Tümer, Lund, Petersen, Cole, Hanusch, Seidel, Jensen, Horn: Similar splice-site mutations of the ATP7A gene lead to different phenotypes: classical Menkes disease or occipital horn syndrome. dans American journal of human genetics 2000
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Human CP Kit ELISA pour Competition ELISA - ABIN612680
Harris, Takahashi, Miyajima, Serizawa, MacGillivray, Gitlin: Aceruloplasminemia: molecular characterization of this disorder of iron metabolism. dans Proceedings of the National Academy of Sciences of the United States of America 1995
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Human CP Kit ELISA pour Sandwich ELISA - ABIN612681
Aliyazicioğlu, Değer, Karahan, Yildirmiş, Küçüködük: Reference values of cord blood transferrin, ceruloplasmin, alpha-1 antitrypsin, prealbumin, and alpha-2 macroglobulin concentrations in healthy term newborns. dans The Turkish journal of pediatrics 2007
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Human CP Kit ELISA pour Sandwich ELISA - ABIN365071
Lee, Joo, Lim, Park, Lee, Park, Seok, Lee, Kang: Proteomic analysis of serum from patients with major depressive disorder to compare their depressive and remission statuses. dans Psychiatry investigation 2015
In D-galactosamine-sensitized mice CP+Cu(II) increased the LPS (Montrer TLR4 Kits ELISA)-induced lethality from 54 to 100%, while administration of antibodies against MIF (Montrer MIF Kits ELISA) prevented the lethal effect. The enhancement by CP+Cu(II) of the pro-inflammatory signal of MIF (Montrer MIF Kits ELISA) is discussed
mice with mutation of Cp and Heph (Montrer HEPH Kits ELISA), iron accumulates in glia, while neurons have low iron levels. Both neurons and glia degenerate and mice become ataxic unless given an iron chelator.
Data (including data from studies in knockout mice) suggest that ceruloplasmin and hephaestin (Montrer HEPH Kits ELISA) play distinct roles in regulation of gene expression in various regions of the brain and are involved in iron homeostasis.
Evidence supports a regulatory role of both proteins (Ceruloplasmin (CP) and beta-amyloid protein precursor (APP (Montrer APP Kits ELISA))) in defence against iron-induced oxidative damage after TBI, which presents as a tractable therapeutic target.
Genetic interactions between Cp, Mon1a (Montrer MON1A Kits ELISA), and the Slc40a1 (Montrer SLC40A1 Kits ELISA) locus are involved in iron metabolism.
ceruloplasmin should provide a protective shield against inadvertent oxidant production by myeloperoxidase (Montrer MPO Kits ELISA) during inflammation
The mouse ceruloplasmin gene has been mapped to chromosome 3.
Data found an increase in ceruloplasmin levels in the plasma of Npc1 (Montrer NPC1 Kits ELISA) -/- mice compared to Npc1 (Montrer NPC1 Kits ELISA) +/+ mice, and this increase was statistically significant (*p < 0.05).
Cp and Heph (Montrer HEPH Kits ELISA) are necessary for iron export from the retina but are not essential for iron import into the retina.
Mechanistically, ceruloplasmin could specifically interact with the hepatitis B virus middle surface protein.
The present study determined that ARE, CLP (Montrer CALML3 Kits ELISA), CAT, and MPO (Montrer MPO Kits ELISA) levels are different between the pediatric patients with sepsis and healthy controls. ARE level can be a potent biomarker for sepsis in critical patients in intensive care units.
Ceruloplasmin was independently and negatively associated with liver fibrosis in chronic hepatitis b.
Coronary atherosclerosis is distinguished by serum C4 complement up-regulation and ceruloplasmin down-regulation.
Data show that the concentration of ceruloplasmin (ferroxidase; Cp) was significantly higher in Low hemoglobin (Hb) compared to High Hb subsample.
serum ceruloplasmin level was lower in the primary open-angle glaucoma group in comparison to the group with only cataract.
The determination of serum ceruloplasmin in adolescents might be a useful tool to identify patients with the highest risk of future cardiovascular disease.
The main result of this study is that ceruloplasmin specific activity is associated with a decreased risk of developing Alzheimer's disease
pathological cerebrospinal fluid's environment of Parkinson's disease patients promoted the same modifications in the exogenously added ceruloplasmin
PON-1 (Montrer PON1 Kits ELISA) and ferroxidase activities in older patients with mild cognitive impairment, late onset Alzheimer's disease or vascular dementia
The protein encoded by this gene is a metalloprotein that binds most of the copper in plasma and is involved in the peroxidation of Fe(II)transferrin to Fe(III) transferrin. Mutations in this gene cause aceruloplasminemia, which results in iron accumulation and tissue damage, and is associated with diabetes and neurologic abnormalities. Two transcript variants, one protein-coding and the other not protein-coding, have been found for this gene.
, hypothetical protein
, ceruloplasmin (ferroxidase)