Aperçu des produits pour anti-S100A9 (S100A9) Anticorps

Full name:
anti-S100 Calcium Binding Protein A9 Anticorps (S100A9)
Chez www.anticorps-enligne.fr sont 273 S100 Calcium Binding Protein A9 (S100A9) Anticorps de 23 de différents fournisseurs disponibles. De plus, nous expédions S100A9 Kits (69) et S100A9 Protéines (34) et beaucoup plus de produits pour cette protéine. Un total de 383 S100A9 produits sont actuellement listés.
Synonymes:
60B8Ag, AW546964, BEE22, Cagb, CFAG, CGLB, GAGB, L1Ag, LIAG, MAC387, MIF, MRP-14, Mrp14, NIF, p14, S100A9

Réactivités les plus populaires pour anti-S100A9 (S100A9) Anticorps

Sélectionnez vos espèces et l'application

anti-Human S100A9 Anticorps:

anti-Rat (Rattus) S100A9 Anticorps:

anti-Mouse (Murine) S100A9 Anticorps:

Tous anti-S100A9 Anticorps disponible

Vous arrivez à notre recherche pré-filtrée.

anti-S100A9 Anticorps mieux référencés

  1. Human Monoclonal S100A9 Primary Antibody pour EIA, FACS - ABIN111889 : Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. dans Journal of clinical pathology 1989 (PubMed)
    Show all 11 references for 111889

  2. Human Monoclonal S100A9 Primary Antibody pour EIA, FACS - ABIN111888 : Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. dans Clinical and experimental immunology 1990 (PubMed)
    Show all 9 references for 111888

  3. Cow (Bovine) Monoclonal S100A9 Primary Antibody pour EIA, FACS - ABIN111887 : Mues, Brisse, Zwadlo, Themann, Bender, Sorg: Phenotyping of macrophages with monoclonal antibodies in endomyocardial biopsies as a new approach to diagnosis of myocarditis. dans European heart journal 1990 (PubMed)
    Show all 8 references for 111887

  4. Human Monoclonal S100A9 Primary Antibody pour EIA, FACS - ABIN111886 : Odink, Cerletti, Brüggen, Clerc, Tarcsay, Zwadlo, Gerhards, Schlegel, Sorg: Two calcium-binding proteins in infiltrate macrophages of rheumatoid arthritis. dans Nature 1987 (PubMed)
    Show all 8 references for 111886

  5. Human Monoclonal S100A9 Primary Antibody pour IHC (fro), FACS - ABIN954655 : Klein, Pino, Johnson, Davies, Nagpal, Thacher, Krasinski, Chandraratna: Identification and functional separation of retinoic acid receptor neutral antagonists and inverse agonists. dans The Journal of biological chemistry 1996 (PubMed)
    Show all 3 references for 954655

  6. Human Monoclonal S100A9 Primary Antibody pour IHC (fro), FACS - ABIN954656 : Nagpal, Thacher, Patel, Friant, Malhotra, Shafer, Krasinski, Asano, Teng, Duvic, Chandraratna: Negative regulation of two hyperproliferative keratinocyte differentiation markers by a retinoic acid receptor-specific retinoid: insight into the mechanism of retinoid action in psoriasis. dans Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1997 (PubMed)
    Show all 3 references for 954656

  7. Human Monoclonal S100A9 Primary Antibody pour IHC (fro), FACS - ABIN954657 : Thacher, Nagpal, Klein, Arefieg, Krasinski, DiSepio, Agarwal, Johnson, Eckert, Chandraratna: Cell type and gene-specific activity of the retinoid inverse agonist AGN 193109: divergent effects from agonist at retinoic acid receptor gamma in human keratinocytes. dans Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1999 (PubMed)
    Show all 3 references for 954657

  8. Human Polyclonal S100A9 Primary Antibody pour IHC, WB - ABIN2786758 : Ghavami, Kerkhoff, Chazin, Kadkhoda, Xiao, Zuse, Hashemi, Eshraghi, Schulze-Osthoff, Klonisch, Los: S100A8/9 induces cell death via a novel, RAGE-independent pathway that involves selective release of Smac/DIABLO and Omi/HtrA2. dans Biochimica et biophysica acta 2008 (PubMed)

Plus d’anticorps contre S100A9 partenaires d’interaction

Human S100 Calcium Binding Protein A9 (S100A9) interaction partners

  1. observations indicate a novel way in which S100A9 may contribute to the pathogenesis of gout, by priming neutrophils to respond to MSUs.

  2. S100A9 was an ATRA-responsive gene, and PML (Montrer PML Anticorps)/RARalpha (Montrer RARA Anticorps) was necessary for the ATRA-induced expression of S100A9 in acute promyelocytic leukemia (Montrer PML Anticorps) (APL (Montrer FASL Anticorps)) cells. PML (Montrer PML Anticorps)/RARalpha (Montrer RARA Anticorps) and PU.1 were necessary for the ATRA-induced expression of S100A9 in APL (Montrer FASL Anticorps) cells. Furthermore, S100A9 promoted apoptosis in APL (Montrer FASL Anticorps) cells and affected cell growth.

  3. Salivary S100A9 is a biomarker for oral squamous cell carcinoma in a Hungarian population.

  4. High S100A9 expression is associated with glioblastoma.

  5. This study implicates S100A8 (Montrer S100A8 Anticorps) and S100A9 as important mediators of tumor cell aggressiveness, and highlights the therapeutic potential of S100A8 (Montrer S100A8 Anticorps) and S100A9 for interference of metastasis.

  6. Faecal calprotectin (Montrer S100A8 Anticorps) and CRP (Montrer CRP Anticorps) are predictive of symptomatic relapse and may be valuable in management of paediatric IBD in clinical remission

  7. Data indicate a statistical correlation between fecal calprotectin (Montrer S100A8 Anticorps) (FC) and gastrointestinal graft-versus-host disease (GvHD).

  8. Perinatal alarmins S100A8 (Montrer S100A8 Anticorps) and S100A9 specifically altered MyD88 (Montrer MYD88 Anticorps)-dependent proinflammatory gene programs. S100 programming prevented hyperinflammatory responses without impairing pathogen defense. TRIF (Montrer TRIM69 Anticorps)-adaptor-dependent regulatory genes remained unaffected by perinatal S100 programming and responded strongly to lipopolysaccharide, but were barely expressed.

  9. These results suggest a preponderant role for the protein S100-A9 as an activator of the NF-kappa B (Montrer NFKB1 Anticorps) pathway during Chronic lymphocytic leukemia progression and suggest that the leukemic clone can generate an autoactivation loop through S100-A9 expression, NF-kappa B (Montrer NFKB1 Anticorps) activation, and exosome secretion.

  10. Expression of S100A8 (Montrer S100A8 Anticorps), S100A9 and S100A12 (Montrer S100A12 Anticorps) is modulated by eicosapentaenoic acid and docosahexaenoic acid during Inflammation in adipose tissue and mononuclear cells.

Rabbit S100 Calcium Binding Protein A9 (S100A9) interaction partners

  1. this study shows that S100A9 inhibits B lymphopoiesis by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta (Montrer IL1B Anticorps)

Pig (Porcine) S100 Calcium Binding Protein A9 (S100A9) interaction partners

  1. Study verified porcine calprotectin (S100A8 (Montrer S100A8 Anticorps)/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.

Cow (Bovine) S100 Calcium Binding Protein A9 (S100A9) interaction partners

  1. Data suggest that up-regulation of S100A8 (Montrer S100A8 Anticorps) and S100A9 is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.

Mouse (Murine) S100 Calcium Binding Protein A9 (S100A9) interaction partners

  1. Neutrophil-derived S100A8 (Montrer S100A8 Anticorps)/A9 promotes thrombocytosis in diabetic mice

  2. S100A9 knockdown almost completely abrogated the effects of IRF7 (Montrer IRF7 Anticorps) deletion on granulocytic myeloid-derived suppressor cells development and tumor metastasis.

  3. Perinatal alarmins S100A8 (Montrer S100A8 Anticorps) and S100A9 specifically altered MyD88 (Montrer MYD88 Anticorps)-dependent proinflammatory gene programs. S100 programming prevented hyperinflammatory responses without impairing pathogen defense. TRIF (Montrer RNF138 Anticorps)-adaptor-dependent regulatory genes remained unaffected by perinatal S100 programming and responded strongly to lipopolysaccharide, but were barely expressed.

  4. vitro studies on the mechanisms/pathways involved in leukemic cell differentiation revealed that binding of S100A9 to Toll-like receptor 4 (TLR4 (Montrer TLR4 Anticorps)) promotes activation of p38 mitogen-activated protein kinase (Montrer MAPK14 Anticorps), extracellular signal-regulated kinases 1 and 2, and Jun N-terminal kinase (Montrer MAPK8 Anticorps) signaling pathways, leading to myelomonocytic and monocytic AML (Montrer RUNX1 Anticorps) cell differentiation.

  5. these results indicate that CD14 (Montrer CD14 Anticorps) is a co-receptor of TLR4 (Montrer TLR4 Anticorps) in the S100A9-induced cytokine response.

  6. Mechanistic studies showed that in MDSCs, ATF3 (Montrer ATF3 Anticorps) was transrepressed by the GC receptor GR through direct binding to the negative GR-response element. S100A9 is the major transcriptional target of ATF3 (Montrer ATF3 Anticorps) in Granulocytic Myeloid-derived Suppressor Cells.

  7. CHI3L1 (Montrer CHI3L1 Anticorps)-mediated cell proliferation/survival involves partial downregulation of the pro-apoptotic S100A9 protein that is highly expressed during the acute phase of colitis, by binding to the S100A9 receptor, RAGE (Montrer AGER Anticorps) (Receptor for Advanced Glycation End products).

  8. Although MRP-8 (Montrer S100A8 Anticorps)/-14 expression is highly increased in experimental, these proteins do not contribute to the pathogenesis in the effector phase of epidermolysis bullosa acquisita and bullous pemphigoid (Montrer DST Anticorps).

  9. Rps14 (Montrer RPS14 Anticorps) haploinsufficiency in del(5q) myelodysplastic syndrome is linked to activation of the innate immune system and induction of S100A8 (Montrer S100A8 Anticorps)-S100A9 expression, leading to a p53 (Montrer TP53 Anticorps)-dependent erythroid differentiation defect.

  10. MRP8 (Montrer S100A8 Anticorps)/14 serves in an unexpected protective role for the lung in staphylococcal pneumonia.

S100A9 profil antigène

Antigen Summary

The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and altered expression of this protein is associated with the disease cystic fibrosis.

Alternative names and synonyms associated with S100A9

  • S100 calcium binding protein A9 (S100A9) anticorps
  • S100 calcium binding protein A9 (S100a9) anticorps
  • S100 calcium binding protein A9 (calgranulin B) (S100a9) anticorps
  • 60B8Ag anticorps
  • AW546964 anticorps
  • BEE22 anticorps
  • Cagb anticorps
  • CFAG anticorps
  • CGLB anticorps
  • GAGB anticorps
  • L1Ag anticorps
  • LIAG anticorps
  • MAC387 anticorps
  • MIF anticorps
  • MRP-14 anticorps
  • Mrp14 anticorps
  • NIF anticorps
  • p14 anticorps
  • S100A9 anticorps

Protein level used designations for S100A9

S100 calcium binding protein A9 , MRP-14 , S100 calcium-binding protein A9 (calgranulin B) , calgranulin B , calgranulin-B , calprotectin L1H subunit , leukocyte L1 complex heavy chain , migration inhibitory factor-related protein 14 , protein S100-A9 , S100 calcium binding protein A9 (calgranulin B) , intracellular calcium-binding protein (MRP14) , myeloid-related protein 14 , p14 , S100 calcium-binding protein A9 , RNA-binding region containing protein 2-like , calprotectin , BEE22 , neutrophil cytosolic 23 kDa protein , p23 , protein MRP-126

GENE ID SPECIES
457998 Pan troglodytes
714690 Macaca mulatta
100294530 Equus caballus
6280 Homo sapiens
94195 Rattus norvegicus
100008704 Oryctolagus cuniculus
100735238 Cavia porcellus
100127489 Sus scrofa
532569 Bos taurus
490463 Canis lupus familiaris
426356 Gallus gallus
20202 Mus musculus
Fournisseurs de qualité sélectionnés pour anti-S100A9 (S100A9) Anticorps
Avez-vous cherché autre chose?