anti-ADAM Metallopeptidase with Thrombospondin Type 1 Motif, 7 (ADAMTS7) Anticorps

The protein encoded by ADAMTS7 is a member of the ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) family. De plus, nous expédions ADAMTS7 Kits (23) et ADAMTS7 Protéines (9) et beaucoup plus de produits pour cette protéine.

afficher tous les anticorps Gène GeneID UniProt
ADAMTS7 108153  
ADAMTS7 11173 Q9UKP4
ADAMTS7 315879 Q1EHB3
Comment commander chez anticorps-enligne
  • +1 877 302 8632
  • +1 888 205 9894 (toll-free)
  • Commandez enligne
  • orders@anticorps-enligne.fr

Top anti-ADAMTS7 Anticorps sur anticorps-enligne.fr

Showing 10 out of 55 products:

Catalogue No. Reactivité Hôte Conjugué Application Images Quantité Fournisseur Livraison Prix Détails
Humain Lapin Inconjugué WB Western blot analysis of ADAMTS7 expression in HEK293T (A), Raw264.7 (B), H9C2 (C) whole cell lysates. 200 μL Connectez-vous pour afficher 13 to 14 Days
$487.50
Détails
Humain Lapin Inconjugué IHC, ELISA, WB 0.1 mg Connectez-vous pour afficher 2 to 6 Days
$403.70
Détails
Humain Lapin Inconjugué IF (p), IHC (p), WB Formalin-fixed and paraffin embedded human placenta labeled with Anti-ADAMTS7 Polyclonal Antibody, Unconjugated (ABIN668586) at 1:200 followed by conjugation to the secondary antibody and DAB staining 100 μL Connectez-vous pour afficher 3 to 7 Days
$306.90
Détails
Humain Lapin Inconjugué IF (p), IHC (p), WB Formalin-fixed and paraffin embedded rat brain labeled with Anti-ADAMTS7 Polyclonal Antibody, Unconjugated (ABIN672486) at 1:200 followed by conjugation to the secondary antibody and DAB staining. Formalin-fixed and paraffin-embedded rat brain labeled with Anti-ADAMTS7 Polyclonal Antibody, Unconjugated (ABIN672486) 1:200, overnight at 4°C, The secondary antibody was Goat Anti-Rabbit IgG, Cy3 conjugated used at 1:200 dilution for 40 minutes at 37°C. 100 μL Connectez-vous pour afficher 3 to 7 Days
$306.90
Détails
Humain Lapin Inconjugué ICC, IHC, WB Used in DAB staining on fromalin fixed paraffin-embedded Liver tissue 100 μg Connectez-vous pour afficher 15 to 18 Days
$332.00
Détails
Souris Lapin Inconjugué ICC, IHC, WB Figure. DAB staining on IHC-P; Samples: Mouse Uterus Tissue. 100 μg Connectez-vous pour afficher 13 to 16 Days
$342.00
Détails
Rat Lapin Inconjugué ICC, IHC, WB Figure.DAB staining on IHC-P. Samples: Rat Tissue 100 μg Connectez-vous pour afficher 13 to 16 Days
$362.00
Détails
Humain Lapin Cy5 IF (p)   100 μL Connectez-vous pour afficher 14 to 21 Days
$405.90
Détails
Humain Lapin Cy5.5 IF (p)   100 μL Connectez-vous pour afficher 14 to 21 Days
$405.90
Détails
Humain Lapin FITC IF (p)   100 μL Connectez-vous pour afficher 14 to 21 Days
$405.90
Détails

ADAMTS7 Anticorps par réactivité, application, clonalité et conjugué

Attributs Application Hôte Clonalité Conjugué
Mouse (Murine) ,


, , , , , , , , , , , , ,
Human , , , ,
,

, , , , , , , , , , , , ,
Rat (Rattus) ,


, , , , , , , , , , , , ,

Plus d’anticorps contre ADAMTS7 partenaires d’interaction

Mouse (Murine) ADAM Metallopeptidase with Thrombospondin Type 1 Motif, 7 (ADAMTS7) interaction partners

  1. Therefore, these data provided the in vivo evidence, suggesting that ADAMTS-7 may play an important role in the pathogenesis of inflammatory arthritis.

  2. Mice lacking Adamts7, Ldlr, Apoe had less lesion formation in aortas and aortic roots vs controls and less neointimal formation after femoral wire injury. Adamts7 expression was induced by injury and hyperlipidemia.

  3. Adamts-7 deficiency substantially ameliorated neointima formation in mice at days 14 and 28 after injury. ADAMTS-7 inhibited both endothelial cell proliferation and migration.

  4. ADAMTS-7 and TNF-alpha (Montrer TNF Anticorps) form a positive feedback loop in the regulation of cartilage degradation and osteoarthritis progression.

  5. ADAMTS7B has a domain organization with a total of eight thrombospondin type 1 repeats in its ancillary domain. Of these, seven are arranged in two distinct clusters that are separated by a mucin (Montrer SLC13A2 Anticorps) domain

  6. Findings demonstrate that ADAMTS-7, a direct target of PTHrP signaling, negatively regulates endochondral bone formation by associating with and inactivating GEP (Montrer GRN Anticorps) chondrogenic growth factor.

Human ADAM Metallopeptidase with Thrombospondin Type 1 Motif, 7 (ADAMTS7) interaction partners

  1. The findings suggest that upregulation of ADAMTS-7 and down regulation of COMP (Montrer COMP Anticorps) are associated with human AA.

  2. The native overfunctional ADAMTS7 allele (A) may accelerate VSMC migration and lead to neointimal thickening, atherosclerosis progression and acute plaque events.

  3. miR (Montrer MLXIP Anticorps)-105/Runx2 (Montrer RUNX2 Anticorps) axis mediates FGF2 (Montrer FGF2 Anticorps)-induced ADAMTS (Montrer ADAMTS13 Anticorps) expression in osteoarthritis cartilage.

  4. Allelic variation that associates with reduced ADAMTS7 expression confers stronger coronary heart disease protection in never-smokers than in ever-smokers.

  5. During inflammatory conditions, AP-1 (Montrer FOSB Anticorps) and Sp1 (Montrer PSG1 Anticorps) sustained the expression of ADAMTS7, and ADAMTS7 sustained the expression of catabolic genes in nucleus pulposus cells

  6. ADAMTS7 and LPA (Montrer APOA Anticorps) single nucleotide polymorphisms are related to a 24-h ambulatory systolic-diastolic pressure regression index.

  7. Expression of miR (Montrer MLXIP Anticorps)-26a and miR (Montrer MLXIP Anticorps)-29a was significantly down regulated in leukoplakia and cancer tissues but up regulated in lichen planus tissues. Expression of target genes such as, ADAMTS7, ATP1B1 (Montrer ATP1B1 Anticorps), COL4A2 (Montrer COL4a2 Anticorps), CPEB3 (Montrer CPEB3 Anticorps), CDK6 (Montrer CDK6 Anticorps), DNMT3a (Montrer DNMT3A Anticorps) and PI3KR1 was significantly down regulated in at least two of three disease types with respect to normal tissues.

  8. Our results indicate the presence of ADAMTS-7 in human NP cells and imply its potential role in disc degeneration.

  9. The main contribution of this study is the proposal of a pharmacophore for ADAMTS7.

  10. The significant associations observed between this coding variant in ADAMTS7 and the risk of CAD (Montrer CAD Anticorps) development.

ADAMTS7 profil antigène

Profil protéine

The protein encoded by this gene is a member of the ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) family. Members of this family share several distinct protein modules, including a propeptide region, a metalloproteinase domain, a disintegrin-like domain, and a thrombospondin type 1 (TS) motif. Individual members of this family differ in the number of C-terminal TS motifs, and some have unique C-terminal domains. The protein encoded by this gene contains two C-terminal TS motifs.

Gene names and symbols associated with ADAMTS7

  • ADAM metallopeptidase with thrombospondin type 1 motif 7 (ADAMTS7) anticorps
  • a disintegrin-like and metallopeptidase (reprolysin type) with thrombospondin type 1 motif, 7 (Adamts7) anticorps
  • ADAM metallopeptidase with thrombospondin type 1 motif, 7 (Adamts7) anticorps
  • ADAM metallopeptidase with thrombospondin type 1 motif 7 (Adamts7) anticorps
  • ADAM-TS7 anticorps
  • ADAM-TS 7 anticorps
  • ADAMTS-7 anticorps
  • ADAMTS7B anticorps
  • COMPase anticorps

Protein level used designations for ADAMTS7

ADAM metallopeptidase with thrombospondin type 1 motif, 7 , A disintegrin and metalloproteinase with thrombospondin motifs 7 , ADAM-TS 7 , ADAMTS-7 , COMPase , a disintegrin-like and metalloprotease (reprolysin type) with thrombospondin type 1 motif, 7 , metalloprotease , a disintegrin and metalloprotease with thrombospondin motifs-7 preproprotein , ADAM-TS7 , a disintegrin-like and metallopeptidase (reprolysin type) with thrombospondin type 1 motif, 7

GENE ID SPECIES
100026396 Monodelphis domestica
108153 Mus musculus
11173 Homo sapiens
100340872 Oryctolagus cuniculus
315879 Rattus norvegicus
504694 Bos taurus
100728771 Cavia porcellus
100154398 Sus scrofa
Fournisseurs de qualité sélectionnés pour anti-ADAMTS7 (ADAMTS7) Anticorps
Avez-vous cherché autre chose?