Amyloid beta (Abeta) Kits ELISA

Abeta encodes a cell surface receptor and transmembrane precursor protein that is cleaved by secretases to form a number of peptides. De plus, nous expédions beta Amyloid Anticorps (184) et beta Amyloid Protéines (3) et beaucoup plus de produits pour cette protéine.

list all ELISA KIts Gène GeneID UniProt
Abeta 351 P05067
Anti-Rat Abeta Abeta 54226 P08592
Anti-Souris Abeta Abeta 11820 P12023
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Catalogue No. Reactivité Sensibilité Gamme Images Quantité Fournisseur Livraison Prix Détails
Humain 10 pg/mL 312-20000 pg/mL Representative Standard Curve 96 Tests Connectez-vous pour afficher 15 to 16 Days

Plus Kits ELISA pour beta Amyloid partenaires d'interaction

Human Amyloid beta (Abeta) interaction partners

  1. The findings reveal a novel mechanism whereby GSK3beta stimulates amyloidogenic processing of APP by phosphorylation of FE65 at threonine 579.

  2. A Toxic Conformer of Abeta42 with a Turn at 22-23 is a Novel Therapeutic Target for Alzheimer's Disease.

  3. Alzheimer's disease-associated TREM2 variants bind Abeta with equivalent affinity but show loss of function in terms of signaling and Abeta internalization.

  4. PKCdelta knockdown reduces BACE1 expression, BACE1-mediated APP processing, and Abeta production.

  5. recombination of APP in human neurons, occurring mosaically as thousands of variant 'genomic cDNAs' (gencDNAs); gencDNAs lacked introns and ranged from full-length cDNA copies of expressed, brain-specific RNA splice variants to myriad smaller forms that contained intra-exonic junctions, insertions, deletions, and/or single nucleotide variations

  6. results confirm the presence of Abeta seeds in archived c-hGH vials and are consistent with the hypothesized iatrogenic human transmission of Abeta pathology; this experimental confirmation has implications for both the prevention and the treatment of Alzheimer's disease, and prompt a review of the risk of iatrogenic transmission of Abeta seeds by medical and surgical procedures long recognized to pose a risk of prion ...

  7. Fpn and Heph co-localize, and FRET analysis indicated that the two proteins form an iron-efflux complex; in contrast, none of the full-length, cellular APP proteins exhibited Fpn co-localization or FRET

  8. we show that F-actin depolymerization in spines leads to a breakdown of the nano-organization of outwardly radiating F-actin rods in cortical neurons from APPswe/PS1DeltaE9 mice

  9. It was evident that on average only a single histidine residue coordinates Cu(II) in monomeric ABETA(1-42) at pH 6.1, in addition to 3 other oxygen or nitrogen ligands. Cu(II) coordination in ABETA(1-42) at pH 7.4 is similarly 4-coordinate with oxygen and nitrogen ligands, although an average of 2 histidine residues appear to coordinate at this pH.

  10. High carboxypeptidase A4 (CPA4) expression in triplenegative breast cancer (TNBC) cases is associated with low expression of E-cadherin and with the expression of cancer stem cell markers. Patients with TNBC and high levels of CPA4 expression have poorer prognosis compared with those with low CPA4 expression. Viability and migration are reduced, but E-cadherin expression is upregulated in CPA4-suppressed TNBC cells.

  11. Overexpression of AbetaPP in Muller cells (MC) induced strong antioxidant and anti-ER stress (PERK downregulation and GADD34 upregulation) responses accompanied by activation of the prosurvival branch of the unfolded protein response. It was also associated with upregulation of major genes involved in MC-controlled retinal homeostasis (KCNJ10, GS, and RLBP1) and protection against HNE-induced apoptosis.

  12. plasma amyloid beta protein level may predicti cognitive decline in patients suffering from chronic kidney disease.

  13. BIN1 rs744373 SNP is associated with increased tau but not beta-amyloid pathology, suggesting that alterations in BIN1 may contribute to memory deficits via increased tau pathology

  14. the presence of ApoEepsilon4 allele might also have a possible effect on the amyloid beta phagocytosis efficiency of the macrophages.

  15. Using Western blotting, kinetic assays, and microfluidic analyses, the study shows that FXIIIa covalently cross-links ABETA40 into dimers and oligomers (k cat/Km = 1.5 x 10(5) m(-1)s(-1)), as well as to fibrin, platelet proteins, and blood clots under flow in vitro ABETA40 also increased the stiffness of platelet-rich plasma clots in the presence of FXIIIa.

  16. As indicated by a comparison between experiment and computation, the number of adjacent beta-strands per peptide molecule is two for ABETA40 oligomers and two or more for ABETA42 oligomers. These results are well explained by regular, antiparallel beta-sheets or beta-barrels.

  17. Study shows attenuated pain-like behaviour in a transgenic mouse model of Alzheimer's disease (bearing mutant human amyloid precursor protein (APP) (695-aa isoform)) due to alterations in the opioidergic system and central plasticity mechanisms of persistent pain.

  18. The primary conclusion is that, under copper-limiting conditions, the M1 site in domain E2 dominates the properties of the entire APP ectodomain with comparable picomolar affinities for both oxidation states Cu(I) and Cu(II).

  19. These results demonstrated the contributory role of APP cleavage on its oncogenic roles in breast cancer. ADAM10 was the key alpha-secretase. APP and ADAM10 co-expression was associated with worse survival in non-luminal breast cancers.

  20. The neuronal specific alpha3 (alpha3)-subunit of the plasma membrane enzyme Na, K-ATPase (NKA) is a new binding partner of sAPPalpha.

Mouse (Murine) Amyloid beta (Abeta) interaction partners

  1. NRBF2 plays an important role in regulating degradation of APP-C-terminal fragments through modulating autophagy.

  2. These results provide evidence that both central circadian rhythms and local clock function influence Abeta dynamics and plaque formation.

  3. These data identified APP and APLP2 as modulators of normal myelination and demyelination/remyelination conditions.

  4. these findings suggest that endogenous Abeta is involved in memory forgetting in the normal brain.

  5. pterostilbene attenuated the neuroinflammatory response induced by Abeta1-42 in microglia through inhibiting the NLRP3/caspase-1 inflammasome pathway, indicating that pterostilbene might be an effective therapy for AD.

  6. Abeta40 and Abeta42 demonstrated distinct distribution kinetics in plasma and brain compartments, and insulin differentially modulated their distribution.

  7. APP/GBR complex formation links presynaptic GBR trafficking to Abeta formation.

  8. APP knockout increases synaptic GluA1, PSD-95 and synaptophysin expression and reduces drebrin expression.

  9. data indicate that Drp1 is a direct target of Cdk5, and Cdk5-mediated phosphorylation of Drp1 at Serine 579 regulates Abeta1-42 induced mitochondrial fission and neuronal toxicity.

  10. miR98 reduced the production of Abeta and improved oxidative stress and mitochondrial dysfunction through activation of the Notch signaling pathway by binding to HEY2 in Alzheimer's disease mice.

  11. Heme and Hb suppress immune activity of primary mouse astrocytes by reducing expression of several proinflammatory cytokines (e.g. RANTES (regulated on activation normal T cell expressed and secreted)) and the scavenger receptor CD36 and reducing internalization of Abeta(1-42) by astrocytes.

  12. extracellular cholesterol concentration in serum under conditions of Npc1 deficiency can influence intracellular cholesterol content/distribution and lysosomal efficacy, triggering the accumulation of toxic APP-cleaved products, eventually leading to cell death.

  13. Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer's disease.

  14. This study provides a novel mechanism underlying aggregation of Abeta peptides via BC1 induction of APP mRNA translation.

  15. This study uncovered two clear phases in the life of APP23 mice: developmental and aging. Development displays similarities to young carriers of familial Alzheimer's disease (AD) mutations. All gene expression differences between APP23 and control mice correlate with aging. Age-related expression changes appear exacerbated/accelerated in APP23 mice.

  16. These results provide evidence for an emerging role of BAG-1M in the regulation of BACE1 expression and AD pathogenesis and that targeting the BAG-1M-NF-kappaB complex may provide a mechanism for inhibiting Abeta production and plaque formation.

  17. Conformational changes in a mouse model of Alzheimer's disease-linked amyloid beta and APP take place before the amyloids plaques can be seen.

  18. These results support the proposition that Aβ release during thrombosis serves as part of a natural defense against infection.

  19. These data suggest a novel regulatory function of juxta- and intra-membrane domains on the metabolism and function of APP.

  20. Inflammasome-derived cytokine IL18 suppresses amyloid-induced seizures in Alzheimer-prone mice.

beta Amyloid (Abeta) profil antigène

Antigen Summary

This gene encodes a cell surface receptor and transmembrane precursor protein that is cleaved by secretases to form a number of peptides. Some of these peptides are secreted and can bind to the acetyltransferase complex APBB1/TIP60 to promote transcriptional activation, while others form the protein basis of the amyloid plaques found in the brains of patients with Alzheimer disease. Mutations in this gene have been implicated in autosomal dominant Alzheimer disease and cerebroarterial amyloidosis (cerebral amyloid angiopathy). Multiple transcript variants encoding several different isoforms have been found for this gene.

Gene names and symbols associated with Amyloid beta (Abeta) Kits ELISA

  • amyloid beta precursor protein (APP) anticorps
  • amyloid beta precursor protein (app) anticorps
  • amyloid beta precursor protein (App) anticorps
  • amyloid beta (A4) precursor protein (App) anticorps
  • aaa anticorps
  • Abeta anticorps
  • Abpp anticorps
  • ad1 anticorps
  • Adap anticorps
  • Ag anticorps
  • appi anticorps
  • betaApp anticorps
  • ctfgamma anticorps
  • Cvap anticorps
  • E030013M08Rik anticorps
  • PN-II anticorps
  • pn2 anticorps

Protein level used designations for Amyloid beta (Abeta) Kits ELISA

alzheimer disease amyloid protein , amyloid beta A4 protein , beta-amyloid peptide , cerebral vascular amyloid peptide , peptidase nexin-II , preA4 , protease nexin-II , amyloid beta (A4) precursor protein (peptidase nexin-2, Alzheimer disease) , amyloid beta (A4) precursor protein (protease nexin-II, Alzheimer disease) , beta-amyloid precursor protein , alzheimer disease amyloid A4 protein homolog , amyloid beta (A4) precursor protein (peptidase nexin-II, Alzheimer disease) , ABPP , AG , amyloid A4 , amyloidogenic glycoprotein , appican , protease nexin II

351 Homo sapiens
448208 Xenopus (Silurana) tropicalis
100427716 Macaca mulatta
54226 Rattus norvegicus
11820 Mus musculus
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