anti-Anti-Mullerian Hormone Receptor, Type II (AMHR2) Anticorps

Cow (Bovine) Anti-Mullerian Hormone Receptor, Type II (AMHR2) interaction partners

1. Results from these studies indicate that AMH (Montrer AMH Anticorps) signaling via AMHR2 plays a role in both regulating granulosa cell proliferation and preventing granulosa cells from 5- to 8-mm follicles from undergoing premature differentiation before follicle selection.

2. These findings indicate the followings: AMH (Montrer AMH Anticorps) mRNA levels decrease in both dominant and secondary follicles during follicular deviation; granulosa cells from heathy follicles express more AMH (Montrer AMH Anticorps) mRNA compared to subordinate follicles undergoing atresia and FSH (Montrer BRD2 Anticorps) stimulates AMH (Montrer AMH Anticorps) and AMHR2 mRNA expression in granulosa cells of co-dominant follicles.

Rhesus Monkey Anti-Mullerian Hormone Receptor, Type II (AMHR2) interaction partners

1. AMH (Montrer AMH Anticorps), co-expressed with AMHR2, was produced heterogeneously by preantral follicles in macaques with levels correlated positively with follicle growth and oocyte maturation. AMH (Montrer AMH Anticorps) may serve as a biomarker for primate follicular development in vitro.

Horse (Equine) Anti-Mullerian Hormone Receptor, Type II (AMHR2) interaction partners

1. Sertoli cell maturation during puberty in the stallion was accompanied by a reduced expression of anti-Mullerian hormone (Montrer AMH Anticorps) and its receptor, arrest of cell proliferation, increased expression of androgen receptor (Montrer AR Anticorps)

Human Anti-Mullerian Hormone Receptor, Type II (AMHR2) interaction partners

1. AMHR2 single nucleotide polymorphism is not associated with Endometriosis-associated infertility.

2. In granulosa cells from women with PCOS, the regulation of AMH (Montrer AMH Anticorps) and AMHR2 expression is altered in a way that promotes the overexpression of the AMH (Montrer AMH Anticorps)/AMHR2 system, and could contribute to the follicular arrest observed in these patients.

3. genetic variants of AMH (Montrer AMH Anticorps) or AMHR2 were not found to be associated with a higher risk for polycystic ovaries syndrome.

4. AMHRII 1749C > T and -482A > G genetic variants are associated with the ovarian response to standard gonadotropin stimulation, affecting mainly the follicular growth in IVF (Montrer SCN5A Anticorps).

5. Genotyping of the AMH (Montrer AMH Anticorps) c.146G>T and AMHR2 -482A>G polymorphisms does not provide additional useful information as a predictor of ovarian reserve or ovarian response and treatment outcomes.

6. The aim of this study was to investigate the density and distribution of single nucleotide polymorphisms (SNPs) anti-Mullerian hormone (AMH (Montrer AMH Anticorps)) and AMHRII receptors in cryptorchid patients.

7. a result of VEGF (Montrer VEGFA Anticorps) misregulation, AMHR2 overexpression increases AMH (Montrer AMH Anticorps) binding, which may attenuate follicular or oocyte maturation.

8. Neither AMH (Montrer AMH Anticorps) nor AMHR2 polymorphisms were related to age, BMI, hormone levels or ovarian parameters in the follicular phase in women of late reproductive stage.

9. There is no association between single nucleotide polymorphisms in the AMH/AMHR2 signaling pathway and early ovarian hyperstimulation syndrome in Han Chinese women

10. A significant subset of GnRH neurons express the AMH receptor.

Mouse (Murine) Anti-Mullerian Hormone Receptor, Type II (AMHR2) interaction partners

1. lncRNA-Amhr2 plays a role in Amhr2 gene activation in ovarian granulosa cells by enhancing promoter activity, providing insight into Amhr2 gene regulation underlying the AMH (Montrer AMH Anticorps) signaling in the female reproductive system.

2. A significant subset of GnRH neurons express the AMH receptor and AMH potently activates the GnRH neuron firing.

3. physiological function(s) of type 2 anti-Mullerian hormone (Montrer AMH Anticorps) receptor (Montrer NR4A1 Anticorps)

4. Wt1 (Montrer WT1 Anticorps) is an activator of Amhr2.