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APOC2 encodes a lipid-binding protein belonging to the apolipoprotein gene family. De plus, nous expédions Apolipoprotein C-II Kits (53) et Apolipoprotein C-II Protéines (28) et beaucoup plus de produits pour cette protéine.
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the apoc2 mutant zebrafish is a robust and versatile animal model to study hypertriglyceridemia.
Triglyceride-raising variant alleles of the APOC2 encoding apo (Montrer C9orf3 Anticorps) C-II, associated with clinical Cardiovascular endpoints.
The results demonstrate the important role of both intra- and inter-subunit charge interactions in stabilizing apoC-II amyloid fibrils, a process that may be a key factor in determining the general ability of proteins to form amyloid fibrils.
The results highlight the importance of charge-pair interactions within the apoC-II fibril core
Conformational rearrangement of apoC-II at lipoprotein surfaces promotes interaction with LPL (Montrer LCP1 Anticorps).
Large deletion in APOC2 caused by Alu-Alu homologous recombination is associated with with apolipoprotein C-II deficiency.
No APOC2 mutations were identified in a cohort of patients with diabetic lipemia.
Six apolipoproteins (APOA1 (Montrer APOA1 Anticorps), APOA2 (Montrer APOA2 Anticorps), APOB (Montrer APOB Anticorps), APOC2, APOC3 (Montrer APOC3 Anticorps), and APOE (Montrer APOE Anticorps)) were able to differentiate bladder cancer from hernia. SAA4 (Montrer SAA4 Anticorps) was significantly increased in bladder cancer subgroups, whereas ProEGF was significantly decreased in bladder cancer subgroups.
STAT1 (Montrer STAT1 Anticorps) bound on multienhancer 2 cooperates with RXRalpha (Montrer RXRA Anticorps) located on apoCII promoter and upregulates apoCII expression only in macrophages.
Mutations in GPIHBP1 (Montrer GPIHBP1 Anticorps) are rare but the associated clinical phenotype of hypertriglyceridaemia is severe
These results support a predictive change in the ratio of plasma ApoCIII (Montrer APOC3 Anticorps) to ApoCII in pregnancies complicated by severe preeclampsia.
A novel mouse model of apoC-II deficiency was created with an apoC-II peptide that reverses the hypertriglyceridemia.
Data show that apoC-II and LPL (Montrer LPL Anticorps) mRNAs correlate temporally and geographically with surfactant lipid synthesis in preparation for birth and suggest that fatty acid recruitment from the circulation by apoC-II-activated LPL (Montrer LPL Anticorps) is modulated by apoC-II secretion.
TR4 (Montrer NR2C2 Anticorps) can also regulate apolipoprotein E (Montrer APOE Anticorps), C-I, and C-II gene expression via the TR4 (Montrer NR2C2 Anticorps) response element within the hepatic control region
This gene encodes a lipid-binding protein belonging to the apolipoprotein gene family. The protein is secreted in plasma where it is a component of very low density lipoprotein. This protein activates the enzyme lipoprotein lipase, which hydrolyzes triglycerides and thus provides free fatty acids for cells. Mutations in this gene cause hyperlipoproteinemia type IB, characterized by hypertriglyceridemia, xanthomas, and increased risk of pancreatitis and early atherosclerosis. This gene is present in a cluster with other related apolipoprotein genes on chromosome 19. Naturally occurring read-through transcription exists between this gene and the neighboring upstream apolipoprotein C-IV (APOC4) gene.
, apolipoprotein C2