Use your antibodies-online credentials, if available.
Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
BTLA encodes a member of the immunoglobulin superfamily. De plus, nous expédions BTLA Anticorps (209) et BTLA Protéines (46) et beaucoup plus de produits pour cette protéine.
Showing 4 out of 8 products:
Data suggest that both HVEM (Montrer TNFRSF14 Kits ELISA) and UL144 bind a common epitope of BTLA, whether engaged in trans or in cis; these studies were conducted in cell lines representing B-lymphocytes, T-lymphocytes, and natural killer cells. (HVEM (Montrer TNFRSF14 Kits ELISA) = human herpes virus entry mediator (Montrer TNFRSF14 Kits ELISA); UL144 = membrane glycoprotein (Montrer CD86 Kits ELISA) UL144 of Human herpesvirus 5; BTLA = human B- and T-lymphocyte attenuator)
our data suggested that the BTLA/HVEM (Montrer TNFRSF14 Kits ELISA) pathway contributes to peripheral T cell suppression in hepatocellular carcinoma patients
data provide the first evidence that increased BTLA predicts poor prognosis in patients with diffuse large B-cell lymphoma (DLBCL), and blockade of BTLA with other checkpoints may potentially represent a new strategy for immunotherapy of DLBCL.
The genetic variants of rs76844316 in BTLA influence the susceptibility to severe chronic hepatitis B and might play a protective role against the progression of chronic hepatitis B.
The impairment of Bregs and CD19 (Montrer CD19 Kits ELISA)+/BTLA+ cells could play an important pathogenic role in multiple sclerosis (MS).
Our data portrays BTLA as a molecule with the singular ability to provide both costimulatory and coinhibitory signals to activated CD8 (Montrer CD8A Kits ELISA)(+) T cells, resulting in extended survival, improved tumor control, and the development of a functional recall response.
results indicate that polymorphisms rs1982809 situated in 3' UTR (Montrer UTS2R Kits ELISA) nearby region of BTLA gene might be considered as low-penetrating risk factor for RCC (Montrer XRCC1 Kits ELISA), but results have to be confirmed in further studies
The percentage of circulating BTLA+CD4+ lymphocytes was significantly higher in patients with severe community acquired pneumonia.
Plasma concentrations of soluble BTLA were increased early in sepsis/septic shock and correlated to severity of disease. A baseline concentration >21ng/mL was associated with a poor prognosis.
this study shows that in chronic hepatitis B virus patients, a subset of inefficient interferon-gamma (Montrer IFNG Kits ELISA) producing antigen-specific CD8 (Montrer CD8A Kits ELISA)+ T cells recruited to the liver expressed high BTLA levels
these data support a role of BTLA on type I NKT (Montrer CTSL1 Kits ELISA) cells in limiting anti-tumor immunity.
these data indicate that HVEM (Montrer TNFRSF14 Kits ELISA)/BTLA interactions are dispensable for the formation of de novo host antidonor isotype-specific antibodies following skin transplantation
The percentage of circulating BTLA+CD4+ lymphocytes was significantly higher in mice with LPS-induced acute lung inflammation.
Dendritic cells require BTLA and HVEM (Montrer TNFRSF14 Kits ELISA) to actively adjust tolerizing T cell responses under steady-state conditions.
these data indicate that CCR7 (Montrer CCR7 Kits ELISA) and BTLA cooverexpression imparts an intermediate immune phenotype in mmature dendritic cells when compared to that in CCR7 (Montrer CCR7 Kits ELISA)- or BTLA-expressing counterparts that show a more immunocompetent or immunotolerant phenotype
this study shows that miR (Montrer MLXIP Kits ELISA)-155 is involved in the inhibition of BTLA during CD4 (Montrer CD4 Kits ELISA)+ T cell activation
Our findings indicate that BTLA may be involved in the control of inflammatory responses through increasing Foxp3 (Montrer FOXP3 Kits ELISA) expression, rather than attenuating IL-17 (Montrer IL17A Kits ELISA) production, in dextran sulfate sodium-induced colitis.
This study uncovers a BTLA-mediated strategy used by the host that permits Listeria proliferation to enable increasing T cell responses for long-term protection.
results support a model whereby BTLA on innate leukocytes is triggered by HVEM (Montrer TNFRSF14 Kits ELISA) and delivers negative signals into BTLA(+) cells, thereby interfering with the protective immune response to this intestinal parasite.
by coordinating expression of BTLA, RORgammat and IL-7 (Montrer IL7 Kits ELISA) balance suppressive and activation stimuli to regulate gammadelta T cell homeostasis and inflammatory responses
This gene encodes a member of the immunoglobulin superfamily. The encoded protein contains a single immunoglobulin (Ig) domain and is a receptor that relays inhibitory signals to suppress the immune response. Alternative splicing results in multiple transcript variants. Polymorphisms in this gene have been associated with an increased risk of rheumatoid arthritis.
B and T lymphocyte associated
, B and T lymphocyte attenuator
, B- and T-lymphocyte attenuator
, B- and T-lymphocyte-associated protein