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BTLA encodes a member of the immunoglobulin superfamily. De plus, nous expédions BTLA Anticorps (214) et BTLA Protéines (43) et beaucoup plus de produits pour cette protéine.
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The genetic variants of rs76844316 in BTLA influence the susceptibility to severe chronic hepatitis B and might play a protective role against the progression of chronic hepatitis B.
The impairment of Bregs and CD19 (Montrer CD19 Kits ELISA)+/BTLA+ cells could play an important pathogenic role in multiple sclerosis (MS).
Our data portrays BTLA as a molecule with the singular ability to provide both costimulatory and coinhibitory signals to activated CD8 (Montrer CD8A Kits ELISA)(+) T cells, resulting in extended survival, improved tumor control, and the development of a functional recall response.
results indicate that polymorphisms rs1982809 situated in 3' UTR (Montrer UTS2R Kits ELISA) nearby region of BTLA gene might be considered as low-penetrating risk factor for RCC (Montrer XRCC1 Kits ELISA), but results have to be confirmed in further studies
The percentage of circulating BTLA+CD4+ lymphocytes was significantly higher in patients with severe community acquired pneumonia.
Plasma concentrations of soluble BTLA were increased early in sepsis/septic shock and correlated to severity of disease. A baseline concentration >21ng/mL was associated with a poor prognosis.
this study shows that in chronic hepatitis B virus patients, a subset of inefficient interferon-gamma (Montrer IFNG Kits ELISA) producing antigen-specific CD8 (Montrer CD8A Kits ELISA)+ T cells recruited to the liver expressed high BTLA levels
this study shows that BTLA expression is likely associated with positive rather than conventional negative regulation of CD11c (Montrer ITGAX Kits ELISA) antigen-presenting cell stimulatory capacity
rs1982809 BTLA gene polymorphism is associated with mRNA expression level and variations in the BTLA gene might be considered as potentially low-penetrating chronic lymphocytic leukemia risk factor
Study showed a decreased expression of BTLA in ocular Behcet's disease suggesting that it may be involved in the development and recurrence of this disease.
these data indicate that HVEM (Montrer TNFRSF14 Kits ELISA)/BTLA interactions are dispensable for the formation of de novo host antidonor isotype-specific antibodies following skin transplantation
The percentage of circulating BTLA+CD4+ lymphocytes was significantly higher in mice with LPS-induced acute lung inflammation.
Dendritic cells require BTLA and HVEM (Montrer TNFRSF14 Kits ELISA) to actively adjust tolerizing T cell responses under steady-state conditions.
these data indicate that CCR7 (Montrer CCR7 Kits ELISA) and BTLA cooverexpression imparts an intermediate immune phenotype in mmature dendritic cells when compared to that in CCR7 (Montrer CCR7 Kits ELISA)- or BTLA-expressing counterparts that show a more immunocompetent or immunotolerant phenotype
this study shows that miR (Montrer MLXIP Kits ELISA)-155 is involved in the inhibition of BTLA during CD4 (Montrer CD4 Kits ELISA)+ T cell activation
Our findings indicate that BTLA may be involved in the control of inflammatory responses through increasing Foxp3 (Montrer FOXP3 Kits ELISA) expression, rather than attenuating IL-17 (Montrer IL17A Kits ELISA) production, in dextran sulfate sodium-induced colitis.
This study uncovers a BTLA-mediated strategy used by the host that permits Listeria proliferation to enable increasing T cell responses for long-term protection.
results support a model whereby BTLA on innate leukocytes is triggered by HVEM (Montrer TNFRSF14 Kits ELISA) and delivers negative signals into BTLA(+) cells, thereby interfering with the protective immune response to this intestinal parasite.
by coordinating expression of BTLA, RORgammat and IL-7 (Montrer IL7 Kits ELISA) balance suppressive and activation stimuli to regulate gammadelta T cell homeostasis and inflammatory responses
BTLA promotes the pathogenesis of virus-induced fulminant hepatitis by enhancing macrophage viability and function. Targeting BTLA may be a novel strategy for the treatment of FH.
This gene encodes a member of the immunoglobulin superfamily. The encoded protein contains a single immunoglobulin (Ig) domain and is a receptor that relays inhibitory signals to suppress the immune response. Alternative splicing results in multiple transcript variants. Polymorphisms in this gene have been associated with an increased risk of rheumatoid arthritis.
B and T lymphocyte associated
, B and T lymphocyte attenuator
, B- and T-lymphocyte attenuator
, B- and T-lymphocyte-associated protein