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The protein encoded by CTNNBIP1 binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. De plus, nous expédions CTNNBIP1 Anticorps (49) et CTNNBIP1 Protéines (15) et beaucoup plus de produits pour cette protéine.
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miR (Montrer MLXIP Kits ELISA)-215-5p is a negative regulator of adipocyte differentiation through post-transcriptional regulation of FNDC3B (Montrer FNDC3B Kits ELISA) and CTNNBIP1 during early adipogenesis
miR (Montrer MLXIP Kits ELISA)-29b plays a pivotal role in fetal mouse neurogenesis by regulating ICAT-mediated Wnt (Montrer WNT2 Kits ELISA)/beta-catenin (Montrer CTNNB1 Kits ELISA) signaling.
Mechanistic studies revealed that CTNNBIP1 suppresses Wnt (Montrer WNT2 Kits ELISA)/beta-catenin (Montrer CTNNB1 Kits ELISA) signaling and that miR (Montrer MLXIP Kits ELISA)-215 promotes beta-catenin (Montrer CTNNB1 Kits ELISA) activation and upregulates alpha-SMA (Montrer SMN1 Kits ELISA) and fibronectin (Montrer FN1 Kits ELISA) expression in TGF-beta1 (Montrer TGFB1 Kits ELISA)-treated MMCs by targeting CTNNBIP1
Overexpression of Icat induces G(2) arrest and cell death in tumor cell mutants for adenomatous polyposis coli (Montrer APC Kits ELISA), beta-catenin (Montrer CTNNB1 Kits ELISA), or Axin (Montrer AXIN1 Kits ELISA).
ICAT plays an important role in the anteriorization of neural cells by inhibiting the posteriorizing activity of Wnt (Montrer WNT2 Kits ELISA) signaling
These results suggest that the loss of ICAT gene function causes the arrest of UB branching and the apoptotic death of MM cells, resulting in renal agenesis.
Inhibition of beta-catenin (Montrer CTNNB1 Kits ELISA) signaling in articular chondrocytes causes increased cell apoptosis and articular cartilage destruction in Col2a1 (Montrer COL2A1 Kits ELISA)-ICAT- transgenic mice.
Data demonstrated that ICAT was highly expressed in cervical cancer tissues and had a role in control of epithelial-mesenchymal transition (EMT) in cervical cancer cells. Its overexpression promoted cell proliferation, migration, invasion and resulted in EMT by disrupting the stability of E-cadherin/betacatenin complex.
Low PLD1 expression and high ICAT expression were significantly associated with increased survival in colorectal cancer patients.
The present work aims to investigate the relationship between the expression of AEG-1 (Montrer MTDH Kits ELISA)(astrocyte elevated gene-1), b-FGF(basic-fibroblast growth factor (Montrer FGF2 Kits ELISA)), beta-catenin (Montrer CTNNB1 Kits ELISA), Ki-67 (Montrer MKI67 Kits ELISA), TNF-alpha (tumor necrosis factor (Montrer TNF Kits ELISA)-alfa) other prognostic parameters in DC (Ductal Carcinomas) and ductal intraepithelial neoplasm. We found a relationship between these factors.
Overexpression of ICAT promoted Caski cells' proliferation, arrested the cell cycle in the S phase and enhanced cell migration. Conclusion Overexpression of ICAT can promote the proliferation and migration of Caski cervical cancer cells.
Somatic mutation of beta-catenin (CTNNB1 (Montrer CTNNB1 Kits ELISA)) is known to be crucial for Wilms tumor development in up to 15% of cases.
CTNNBIP1 expression correlated with longer overall survival in LAC (Montrer LCT Kits ELISA) patients. This study reveals that miR (Montrer MLXIP Kits ELISA)-214 plays a critical role in CSLC self-renewal and stemness by targeting CTNNBIP1.
Data show that microRNA miR (Montrer MLXIP Kits ELISA)-215 activates beta-catenin (Montrer CTNNB1 Kits ELISA) pathways by decreasing catenin beta interacting protein 1 (CTNNBIP1)expression in gliomas.
Simultaneous silencing of beta-catenin (Montrer CTNNB1 Kits ELISA) and STAT3 (Montrer STAT3 Kits ELISA) synergistically induces apoptosis and inhibits cell proliferation in HepG2 liver cancer cells.
Finally, pro-incubation with idebenone inhibited mitochondrial dysfunction induced by oxLDL through the mitochondrial-dependent apoptotic pathway and GSK3beta/beta-catenin (Montrer CTNNB1 Kits ELISA) signalling pathways.
A potent beta-catenin (Montrer CTNNB1 Kits ELISA) inhibitor, ICAT/CTNNBIP1 was a direct target of miR (Montrer MLXIP Kits ELISA)-424-5p.
The protein encoded by this gene binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. The encoded protein is a negative regulator of the Wnt signaling pathway. Two transcript variants encoding the same protein have been found for this gene.
, beta-catenin-interacting protein 1
, catenin, beta interacting protein 1 a
, catenin beta interacting protein 1 b
, catenin, beta interacting protein 1
, inhibitor of beta-catenin and Tcf-4
, beta-catenin-interacting protein ICAT