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FDXR encodes a mitochondrial flavoprotein that initiates electron transport for cytochromes P450 receiving electrons from NADPH. De plus, nous expédions Ferredoxin Reductase Anticorps (83) et Ferredoxin Reductase Protéines (11) et beaucoup plus de produits pour cette protéine.
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We constructed cDNA including genes of the bovine cholesterol hydroxylase/lyase (CHL (Montrer CHRDL1 Kits ELISA)) system proteins-cytochrome P450scc (CYP11A1 (Montrer CYP11A1 Kits ELISA)), adrenodoxin (Adx (Montrer FDX1 Kits ELISA)) and adrenodoxin reductase (AdR), that are fused into a single ORF using FMDV 2A nucleotide sequences.
S. pombe is either very robust to mitochondrial reactive oxygen species (ROS (Montrer ROS1 Kits ELISA)) production or does not undergo apoptotic cell death in response to mitochondrial ROS (Montrer ROS1 Kits ELISA) at all.
FDXR and p53 (Montrer TP53 Kits ELISA) are mutually regulated and that the FDXR-p53 (Montrer TP53 Kits ELISA) loop is critical for tumor suppression via iron homeostasis.
we identified a novel disease-causing gene FDXR associated with mitochondrial diseases. The biallelic FDXR mutations cause optic atrophy and neuropathy. we found that FDXR levels are significantly lower in the patient fibroblast cells with the homozygous mutations R392W. Fourteen missense or nonsense FDXR mutations were identified in this study and eight of them (I143F, V158M, T211A, I213F, K280*, R315*, C359Y, D374N) clu (Montrer CLU Kits ELISA)
Using surface plasmon resonance, physiologically relevant concentrations of isatin (25-100 muM) were found to increase affinity of interactions between human recombinant ferrochelatase (FECH (Montrer FECH Kits ELISA)) and NADPH (Montrer NQO1 Kits ELISA)-dependent adrenodoxin reductase (ADR).
Mutation in FDXR gene is associated with Sensorial Neuropathies.
NOS-3 overexpression resulted in an increased sensitivity to anti-Fas (Montrer FAS Kits ELISA) induced cell death, independently of AR expression and CatD (Montrer CTSD Kits ELISA) activity.
These results indicated that abundant FDXR expression in these steroidogenic cells was maintained through SF-1 (Montrer NR5A1 Kits ELISA) binding to the intronic enhancer of the FDXR gene
results suggest that both FDX1 (Montrer FDX1 Kits ELISA) and FDX2 (Montrer FDX1L Kits ELISA) and their likely reductase partner, FDXR, contribute to iron-sulfur cluster biogenesis
comparison of catalytic properties between conditions of limiting and saturating adrenodoxin reductase [cytochrome P450scc (Montrer CYP11A1 Kits ELISA)]
The ferredoxin reductase gene is regulated by the p53 (Montrer TP53 Kits ELISA) family and sensitizes cells to oxidative stress-induced (Montrer SQSTM1 Kits ELISA) apoptosis
ADXR rate of hydroxylation was linear with incubation time.
This gene encodes a mitochondrial flavoprotein that initiates electron transport for cytochromes P450 receiving electrons from NADPH. Multiple alternatively spliced transcript variants have been found for this gene.
NADPH:adrenodoxin oxidoreductase, mitochondrial
, adrenodoxin reductase
, ferredoxin--NADP(+) reductase
, ferredoxin reductase
, ferredoxin--NADP reductase
, adrenodoxin-NADP(+) reductase
, ferredoxin reductase L homeolog