anti-Flightless I Homolog (Drosophila) (FLII) Anticorps

FLII encodes a protein with a gelsolin-like actin binding domain and an N-terminal leucine-rich repeat-protein protein interaction domain. De plus, nous expédions Flightless I Homolog (Drosophila) Protéines (4) et Flightless I Homolog (Drosophila) Kits (3) et beaucoup plus de produits pour cette protéine.

afficher tous les anticorps Gène GeneID UniProt
FLII 2314 Q13045
FLII 14248 Q9JJ28
FLII 287375  
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Top anti-Flightless I Homolog (Drosophila) Anticorps sur anticorps-enligne.fr

Showing 10 out of 45 products:

Catalogue No. Reactivité Hôte Conjugué Application Images Quantité Livraison Prix Détails
Boeuf (Vache) Lapin Inconjugué IHC, WB WB Suggested Anti-FLII Antibody Titration: 0.2-1 ug/mlELISA Titer: 1:62500Positive Control: OVCAR-3 cell lysate 100 μL 2 to 3 Days
$289.00
Détails
Humain Lapin Inconjugué ELISA, WB Western blot analysis FLII using HepG2 whole cell lysates 100 μL 11 to 12 Days
$390.77
Détails
Cheval Lapin Inconjugué IHC, IHC (p) Anti-FLII / FLI antibody IHC staining of human heart. Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval. Antibody  ABIN1101558 dilution 41552 ug/ml. Anti-FLII / FLI antibody IHC staining of human prostate. Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval. Antibody  ABIN1101558 dilution 41552 ug/ml. 50 μg 11 to 14 Days
$484.00
Détails
Boeuf (Vache) Lapin Inconjugué IHC, IHC (p), WB Human, Heart: Formalin-Fixed Paraffin-Embedded (FFPE) Human, Prostate: Formalin-Fixed Paraffin-Embedded (FFPE) 100 μL 11 to 14 Days
$727.83
Détails
Humain Lapin Inconjugué IF, IHC (p), WB Western blot analysis of Lane 1: RT-4, Lane 2: U-251 MG, Lane 3: Human Plasma, Lane 4: Liver, Lane 5: Tonsil with FLII polyclonal antibody  at 1:250-1:500 dilution. Immunofluorescent staining of human cell line A-431 with FLII polyclonal antibody  at 1-4 ug/mL dilution shows positivity in nucleus but not nucleoli, cytoplasm. 100 μL 11 to 12 Days
$577.33
Détails
Humain Lapin Inconjugué ICC, IHC, WB Figure. Western Blot; Sample: Lane1: Human Lung Tissue; Lane2: Porcine Kidney Tissue. Figure. Western Blot; Sample: Recombinant FLII, Human. 100 μg 13 to 16 Days
$350.00
Détails
Humain Lapin Inconjugué ICC, WB   100 μL 11 to 14 Days
$537.17
Détails
Humain Lapin Inconjugué WB Western blot analysis of extracts of various cell lines, using FLII antibody. 200 μL 12 to 14 Days
$438.90
Détails
Humain Lapin Inconjugué WB FLII antibody used at 1 ug/ml to detect target protein. 50 μg 9 to 11 Days
$449.29
Détails
Souris Lapin FITC ELISA, WB   100 μg 11 to 14 Days
$581.17
Détails

Plus d’anticorps contre Flightless I Homolog (Drosophila) partenaires d’interaction

Human Flightless I Homolog (Drosophila) (FLII) interaction partners

  1. this study shows that Flii alters the inflammatory response and autoantibody profile in an OVA-induced atopic dermatitis skin-like disease

  2. Flightless-I functions as a checkpoint protein for selective autophagy by interacting with p62 to block its recognition of LC3, leading to tumorigenesis in breast cancer.

  3. Data reveal an unknown mechanism by which the transcription coactivator FLII regulates the GR-mediated repression of ERalpha target gene expression in MCF-7 cells.

  4. Low FLII expression is associated with lung carcinoma.

  5. Data show that the ability of Ca(2+) to accentuate the activity of NLRP3 inflammasome is abrogated in Flightless-I (FliI) and leucine-rich repeat FliI-interaction protein 2 (LRRFIP2)-knockdown macrophages.

  6. Together, the data demonstrate the critical requirement of GGAA-microsatellites as EWS/FLI activating response elements in vivo and reveal an unexpected role for the EWS portion of the EWS/FLI fusion in binding to sweet-spot GGAA-microsatellites.

  7. FLII is a component of the ChREBP transcriptional complex and negatively regulates ChREBP function in cancer cells.

  8. Demonstrate an important role for Flii in the development and regulation of the epidermal barrier, which may contribute to the impaired healing and skin fragility of epidermolysis bullosa patients.

  9. FLII is associated with SENP3 and the MLL1/2 complex and FLII is indispensible for H3K4 methylation and proper loading of active RNA polymerase II at this gene locus.

  10. Flightless-I (Drosophila) homolog (FLII) activates TGFbeta1-mediated expression of COL1A2 gene.

  11. Studies suggest that Flii enhances cutaneous squamous cell carcinoma progression by decreasing apoptosis and enhancing tumor cell invasion.

  12. FLII plays a tumor-suppressive role and serves as a crucial determinant of resistance of prostate cancer to endocrine therapies.

  13. These data suggest FLII as a key regulator of ERalpha-mediated transcription through its role in regulating chromatin accessibility for the binding of RNA Polymerase II and possibly other transcriptional coactivators.

  14. Flii is constitutively secreted from macrophages and fibroblasts and is present in human plasma.

  15. Fli-I promotes the GTP-bound active Rho-mediated relief of the autoinhibition of Daam1 and mDia1. Thus, Fli-I is a novel positive regulator of Rho-induced linear actin assembly mediated by DRFs.

  16. performs an essential function in early embryonic development

  17. These data suggest that Flightless-I may facilitate interaction of the p160 coactivator complex with other coactivators or coactivator complexes containing actin or actin-like proteins.

  18. effect of FliI protein on actin remodelling is a vital part of cellular motility, contraction and adhesion. Exact signaling pathways and mechanisms underpinning FliI effects in wound healing are yet to be fully identified[review]

  19. These findings support a novel mechanism whereby cytosolic CaMK-II influences beta-catenin dependent gene expression through Fli-I.

  20. The interactions between MyD88 and multiple positive and negative regulators LRRFIP2, FLAP-1, and Fliih are highly dynamic and time-course dependent in differentially regulating/modulating NF-kappa B signal transduction.

Mouse (Murine) Flightless I Homolog (Drosophila) (FLII) interaction partners

  1. FliI is required for the interaction of Rasgap(120) with G3BP1 to regulate R-ras activity and growth of cell extensions.

  2. Fluorescence resonance energy transfer experiments showed that FliI-NMMIIA interactions require Ca(2+) influx. We conclude that Ca(2+) influx through the TRPV4 channel regulates FliI-NMMIIA interaction, which in turn enables generation of the cell extensions essential for collagen remodeling

  3. Flii genetic expression is enhances tissue regeneration, after claw amputation.

  4. Studies suggest that Flii enhances cutaneous squamous cell carcinoma progression by decreasing apoptosis and enhancing tumor cell invasion.

  5. P-Rex1 stimulates migration through enhancing the interaction between Rac1 and the actin-remodelling protein.

  6. Genes downstream from Flii, including TGF-beta1 and TGF-beta3, showed significantly altered expression confirming a functional effect of the Rhodamine-Flii small interfering RNA on gene expression

  7. FliI interacts with NMMIIA to promote cell extension formation, which enables collagen remodeling in fibroblasts.

  8. FLII functions in PPARgamma activation as a molecular switch to repress transcriptional activity by interrupting formation of the PPARgamma/RXRalpha complex.

  9. LRRFIP2 inhibits NLRP3 inflammasome activation by recruiting the caspase-1 inhibitor Flightless-I, thus outlining a new mechanism for negative regulation of NLRP3 inflammasome.

  10. increasing the level of Flii in diabetic mouse wounds led to increased TLR4 and NF- kappa B production. Treatment of murine diabetic wounds with neutralising antibodies to Flii led to an improvement in healing with decreased expression of TLR4

  11. Using a mouse model of epidermolysis bullosa acquisita, the effect of "mopping up" Flii using Flii-neutralizing antibodies before, during, and after blister formation was determined.

  12. Flii is constitutively secreted from macrophages and fibroblasts and is present in human plasma.

  13. FliI regulates cell migration through its localization to focal adhesions and its ability to cap actin filaments, which collectively affect focal adhesion maturation.

  14. Data show that mice with elevated Flii expression exhibit impaired wound healing.

  15. Overexpression of Flii produced severe blistering post-induction of EBA, while decreased Flii reduced blister severity, elevated integrin expression, and improved ColVII production.

  16. show that Flii modulation of focal adhesions and filamentous actin stress fibers is Rac1-dependent

  17. Flii appears to have a positive role in the regeneration of hair follicles, contrary to its negative influence on wound healing in skin

  18. performs an essential function in early embryonic development

  19. Directly interferes with the formation of the TLR4-MyD88 signaling complex.

  20. FliI is a contributing factor to impaired healing and strategies aimed at decreasing FliI levels in elderly skin may improve wound repair.

Cow (Bovine) Flightless I Homolog (Drosophila) (FLII) interaction partners

  1. These findings strongly demonstrate that the three single nucleotide polymorphisms of FLII gene could be utilized as molecular markers for future assisted selection in cattle breeding program.

Zebrafish Flightless I Homolog (Drosophila) (FLII) interaction partners

  1. Embryos defective in flightless I homolog (flii), which encodes for an actin-regulating protein, exhibit normal coilings at 24 hours post fertilization (hpf) that is followed by significantly slower burst swimming at 48 hpf.

Caenorhabditis elegans (C. elegans) Flightless I Homolog (Drosophila) (FLII) interaction partners

  1. fli-1 plays an important role in regulating the actin-dependent events during C. elegans development.

  2. FLI-1 controls germ line morphogenesis and rachis organization

Flightless I Homolog (Drosophila) (FLII) profil antigène

Profil protéine

This gene encodes a protein with a gelsolin-like actin binding domain and an N-terminal leucine-rich repeat-protein protein interaction domain. The protein is similar to a Drosophila protein involved in early embryogenesis and the structural organization of indirect flight muscle. The gene is located within the Smith-Magenis syndrome region on chromosome 17.

Gene names and symbols associated with FLII

  • FLII, actin remodeling protein (FLII) anticorps
  • flightless I actin binding protein (Flii) anticorps
  • FLII, actin remodeling protein (Flii) anticorps
  • flightless I actin binding protein (flii) anticorps
  • protein flightless-1 homolog (LOC585336) anticorps
  • FLII, actin remodeling protein (flii) anticorps
  • protein flightless-1 homolog (LOC100640615) anticorps
  • FLII, actin remodeling protein S homeolog (flii.S) anticorps
  • Protein flightless-1 homolog (fli-1) anticorps
  • 3632430F08Rik anticorps
  • DKFZp459O043 anticorps
  • FLI anticorps
  • Fli1 anticorps
  • flightless anticorps
  • Fliih anticorps
  • FLIL anticorps
  • im:7141769 anticorps

Protein level used designations for FLII

protein flightless-1 homolog , flightless I homolog (Drosophila) , flightless-I homolog , flightless I homolog

GENE ID SPECIES
2314 Homo sapiens
14248 Mus musculus
287375 Rattus norvegicus
416515 Gallus gallus
454486 Pan troglodytes
479521 Canis lupus familiaris
514446 Bos taurus
560281 Danio rerio
585336 Strongylocentrotus purpuratus
700471 Macaca mulatta
100051793 Equus caballus
100173989 Pongo abelii
100398338 Callithrix jacchus
100475330 Ailuropoda melanoleuca
100543687 Meleagris gallopavo
100553018 Anolis carolinensis
100600354 Nomascus leucogenys
100619473 Monodelphis domestica
100640615 Amphimedon queenslandica
444748 Xenopus laevis
176215 Caenorhabditis elegans
100728597 Cavia porcellus
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