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The protein encoded by GRPP is actually a preproprotein that is cleaved into four distinct mature peptides.
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Study concludes that in healthy subjects, glucagon-like peptide-1 (GLP-1 (Montrer GCG Anticorps)) increases cardiac output acutely due to a GLP-1 (Montrer GCG Anticorps)-induced vasodilation in adipose tissue and skeletal muscle together with an increase in cardiac work.
Chenodeoxycholic acid stimulates glucagon-like peptide-1 (Montrer GCG Anticorps) secretion in patients after Roux-en-Y gastric bypass.
The results demonstrate that glucagon-like peptide-1 (Montrer GCG Anticorps) and insulin (Montrer INS Anticorps) synergistically and additively activate vagal afferent neurons.
DPP-4 (Montrer DPP4 Anticorps) activity and GLP (Montrer RCBTB1 Anticorps)-1total levels were higher in patients with microvascular complications associated with T2DM. Contrary to expectations, no negative correlation was seen between GLP-1 (Montrer GCG Anticorps) and DDP (Montrer TIMM8A Anticorps)-4 levels. This result suggests the possible inefficacy of DDP (Montrer TIMM8A Anticorps)-4 activity as a marker to predict in vivo degradation of endogenous GLP-1 (Montrer GCG Anticorps).
Data suggest that cAMP acts as amplifier of insulin (Montrer INS Anticorps) secretion triggered by Ca2 (Montrer CA2 Anticorps)+ elevation in beta-cells; both messengers are also positive modulators of glucagon (Montrer GCG Anticorps) release from alpha-cells, but in this case cAMP signaling may be the important regulator and Ca2 (Montrer CA2 Anticorps)+ signaling has a more permissive role. [REVIEW]
This study provides evidence that, in HepG2 cells, GLP-1 (Montrer GCG Anticorps) may affect cholesterol homeostasis by regulating the expression of miR (Montrer MLXIP Anticorps)-758 and ABCA1 (Montrer ABCA1 Anticorps).
This study reports the transition dipole strengths and frequencies of the amyloid beta-sheet amide I mode for the aggregated proteins amyloid-beta1-40, calcitonin (Montrer CALCA Anticorps), alpha-synuclein, and glucagon (Montrer GCG Anticorps).
genetic association studies in population in China: Data confirm that an SNP in an intron of SLC47A1 (Montrer SLC47A1 Anticorps) (rs2289669) is associated with hypoglycemic response to metformin in patients with newly diagnosed type 2 diabetes; differential increases in basal GLP1 (Montrer GCG Anticorps) plasma levels are also related to this SNP. (SLC47A1 (Montrer SLC47A1 Anticorps) = solute carrier family 47 member 1 (Montrer SLC47A1 Anticorps); GLP1 (Montrer GCG Anticorps) = glucagon-like peptide-1 (Montrer GCG Anticorps))
GLP-2 (Montrer GCG Anticorps) augmented BRIN BD11 (Montrer DEFB110 Anticorps) beta-cell proliferation, but was less efficacious in 1.1B4 cells. These data highlight the involvement of GLP-2 receptor (Montrer GLP2R Anticorps) signalling in the adaptations to pancreatic islet cell stress.
Everolimus down-regulates the systemic levels of gastrin (Montrer GAST Anticorps) and glucagon (Montrer GCG Anticorps) in patients with pancreatic neuroendocrine tumors.
Chronic stress accelerates DPP4-mediated GLP-1 degradation and alters plasma adiponectin, accelerating vascular senescence and impairing ischemia-induced neovascularization.
Data suggest that metabolism of glutamine (Montrer GFPT1 Anticorps) and related analogs by Gdh (Montrer UGDH Anticorps) in intestinal L-cells explains why Glp1 (Montrer GCG Anticorps) secretion, but not that of insulin (Montrer INS Anticorps) by pancreatic beta-cells, is activated by these secretagogues. (Gdh (Montrer UGDH Anticorps) = glutamate (Montrer GRIN1 Anticorps) dehydrogenase; Glp1 (Montrer GCG Anticorps) = glucagon-like peptide 1 (Montrer GCG Anticorps))
Glucokinase (Montrer GCK Anticorps) governs an alpha-cell metabolic pathway that suppresses secretion at or above normoglycemic levels; abnormal suppression of glucagon (Montrer GCG Anticorps) secretion deregulates hepatic glucose metabolism and, over time, induces a pre-diabetic phenotype.
in colonic crypt cultures, the GLP-1 (Montrer GCG Anticorps) secretion induced by such Gq + Gs GPR40 (Montrer FFAR1 Anticorps) agonists is indeed inhibited by blockers of both Gq and Gs and is eliminated by combining these.
Enteric GLP-1 (Montrer GCG Anticorps) activates NO production by enteric neurons that is impaired in type 2 diabetes. Gut (Montrer GUSB Anticorps) microbiota dysbiosis induces enteric neuropathy. Gut (Montrer GUSB Anticorps) microbiota dysbiosis is responsible for the GLP-1 (Montrer GCG Anticorps) resistance.
of glucagon-like peptide-1 (Montrer GCG Anticorps) in vagotomized mice may prevent VLDL overproduction and insulin (Montrer INS Anticorps) resistance induced by high-fat diet.
beta-cell function, plasma active GLP-1 (Montrer GCG Anticorps) levels, the GLP-1R (Montrer GLP1R Anticorps) pathway in beta cells and L cell differentiation, were investigated.
CCK (Montrer CCK Anticorps)/GLP-1 (Montrer GCG Anticorps) play contributory roles in anorexia induction by trichothecenes T-2 toxin, HT-2 toxin, diacetoxyscirpenol and neosolaniol.
The role of syntaxin 1A (Montrer STX1A Anticorps) in GLP1 (Montrer GCG Anticorps) release from intestinal cells as a response to external stimuli is reported.
GCG (Montrer GCG Anticorps) neurons likely stimulate separate populations of downstream cells to produce a change in food intake and glucose homeostasis and that these effects depend on the metabolic state of the animal.
The protein encoded by this gene is actually a preproprotein that is cleaved into four distinct mature peptides. One of these, glucagon, is a pancreatic hormone that counteracts the glucose-lowering action of insulin by stimulating glycogenolysis and gluconeogenesis. Glucagon is a ligand for a specific G-protein linked receptor whose signalling pathway controls cell proliferation. Two of the other peptides are secreted from gut endocrine cells and promote nutrient absorption through distinct mechanisms. Finally, the fourth peptide is similar to glicentin, an active enteroglucagon.
, glucagon-like peptide 1
, glucagon-like peptide 2
, glucagon-like peptide I