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The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. De plus, nous expédions HAVCR1 Kits (80) et HAVCR1 Protéines (36) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 160 products:
Human Monoclonal HAVCR1 Primary Antibody pour CyTOF, FACS - ABIN4899579
Ivie, Fennessey, Sheng, Rubin, McClain: Gene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxin. dans PLoS ONE 2011
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Human Monoclonal HAVCR1 Primary Antibody pour CyTOF, FACS - ABIN4899580
Kuroda, Fujikura, Noyori, Kajihara, Maruyama, Miyamoto, Yoshida, Takada: A polymorphism of the TIM-1 IgV domain: implications for the susceptibility to filovirus infection. dans Biochemical and biophysical research communications 2014
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Mouse (Murine) Polyclonal HAVCR1 Primary Antibody pour ICC, IHC - ABIN1077715
Schindler, Bondeva, Schindler, Claus, Franke, Wolf: Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models. dans Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2016
Data suggest that T-cell immunoglobulin domain and mucin (Montrer SLC13A2 Anticorps) domain containing protein 1 (TIM1 (Montrer TIMELESS Anticorps)) to be under positive natural selection in primates.
Cisplatin enhances kidney injury molecule-1 (Kim-1) gene expression in kidney S3 cells.
By preventing ERK1/2 phosphorylation following renal injury, STAT3 (Montrer STAT3 Anticorps) phosphorylation is decreased, leading to less phosphorylated STAT3 (Montrer STAT3 Anticorps) within the nucleus, and subsequently less KIM-1 mRNA increases post injury
Study used a previously described highly mobile membrane mimetic membrane in combination with a conventional lipid bilayer model to generate a membrane-bound configuration of Tim1 (Montrer ARHGEF5 Anticorps) in silico, identified two possible states for a membrane-bound form of Tim1 (Montrer ARHGEF5 Anticorps).
Our data reveal a previously unknown role for Galpha12 (Montrer GNA12 Anticorps) in regulating efferocytosis and that renal tubular epithelial cells require KIM-1 to mediate this process.
Urinary L-FABP (Montrer FABP1 Anticorps), NGAL (Montrer LCN2 Anticorps), Kim-1 and albumin (Montrer ALB Anticorps) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (Montrer HAND1 Anticorps)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (Montrer LDLR Anticorps) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
data suggest that TIM-1 signaling plays a direct role in Breg maintenance and induction both under physiological conditions (in response to ACs (Montrer Acsl1 Anticorps)) and in response to therapy through TIM-1 ligation
Deletion of the mucin (Montrer SLC13A2 Anticorps) domain impaired KIM-1-mediated phagocytic function, resulting in increased proinflammatory cytokine production, decreased antiinflammatory growth factor secretion by proximal epithelial cells, and an increase in tissue macrophages.
Tim-1 is critical for maintaining self-tolerance by regulating IL-10 (Montrer IL10 Anticorps) production in Bregs
data show that in addition to the phospholipid-binding function of HAVCR1, the enhancement of HCV infection involves other determinants in the IgV domain of HAVCR1. These findings expand the repertoire of molecules that HCV uses for cell entry, adding to the already complex mechanism of HCV infection and pathogenesis.
Review/Meta-analysis: suggest that TIM-1 (Montrer ARHGEF5 Anticorps)-416G[C single nucleotide polymorphism is associated with the risk of asthma for the Asian ethnicity in the codominant (G/G vs. G/C) and dominant (G/G vs. G/C + C/C) genetic models.
Urinary NGAL (Montrer LCN2 Anticorps) is a good predictor of mortality in established pediatric acute kidney injury (AKI) of heterogeneous etiology and outperforms calprotectin (Montrer S100A8 Anticorps) and KIM-1. Urinary calprotectin (Montrer S100A8 Anticorps) and KIM-1 predict renal replacement therapy requirement in pediatric AKI.
A panel consisting of IGFBP1 (Montrer IGFBPI Anticorps), KIM1, GCLC (Montrer GCLC Anticorps) and GSTM1 (Montrer GSTM1 Anticorps) genes could be used in combination for early screening of CKDu, whereas these genes in addition with FN1 (Montrer FN1 Anticorps), IGFBP3 (Montrer IGFBP3 Anticorps) and KLK1 (Montrer KLK1 Anticorps) could be used to monitor progression of CKDu. The regulation of these genes has to be studied on larger populations to validate their efficiency for further clinical use.
Quantification of TIM-3 (Montrer HAVCR2 Anticorps) and KIM-1 mRNA expressions, along with KIM-1 protein measurements in urine and blood could be employed as promising tools for noninvasive diagnosis of allograft dysfunction.
Report detection of drug-induced acute kidney injury in humans by combining the sensitivity of urinary KIM-1 along with urinary miR (Montrer MLXIP Anticorps)-21, -200c, and -423.
measurement of urinary and renal KIM-1 level may be helpful to evaluate severity of renal pathological damage and prognosis in adult Henoch-Schonlein purpura patients with nephritis
Urine KIM-1 level in acute kidney injury patients was significantly higher than that in the healthy controls.
PCNSL is characterized by frequent Tim-1 (Montrer ARHGEF5 Anticorps) expression, and its soluble form in CSF (Montrer CSF2 Anticorps) may become a useful biomarker for PCNSL.
TIM-1 (Montrer ARHGEF5 Anticorps) is a unique marker for the identification of a human IL-10 (Montrer IL10 Anticorps)(+) Breg subpopulation which is partially superimposed with transitional B cells
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, hepatitis A virus cellular receptor 1 homolog
, kidney injury molecule 1
, t cell immunoglobulin and mucin domain-containing protein 1
, t cell membrane protein 1
, T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, rho guanine nucleotide exchange factor 5