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IFI6 was first identified as one of the many genes induced by interferon. De plus, nous expédions IFI6 Kits (7) et IFI6 Protéines (3) et beaucoup plus de produits pour cette protéine.
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Consequent to its localisation on inner-mitochondrial membrane, mtROS were higher in G1P3-expressing cells (MCF-7G1P3). This study identified that G1P3-induced mtROS have a direct role in migratory structure formation and nuclear gene expression to promote breast cancer cell metastasis.
Data suggest that p7 is a critical immune evasion protein that suppresses the antiviral interferon function by counteracting the function of IFI6-16.
In insulitic islets from living patients with recent-onset T1D, most of the overexpressed ISGs, including GBP1 (Montrer GBP1 Anticorps), TLR3 (Montrer TLR3 Anticorps), OAS1 (Montrer OAS1 Anticorps), EIF2AK2 (Montrer EIF2AK2 Anticorps), HLA-E (Montrer HLAE Anticorps), IFI6, and STAT1 (Montrer STAT1 Anticorps), showed higher expression in the islet core compared with the peri (Montrer PLIN1 Anticorps)-islet area containing the surrounding immune cells
In conclusion, over-expression of IFI6 promotes hepatitis C virus RNA replication and rescues the interferon alpha-mediated anti-hepatitis C virus activity.
IFI6 inhibits HCV entry by impairing EGFR (Montrer EGFR Anticorps) mediated CD81 (Montrer CD81 Anticorps)/CLDN1 (Montrer CLDN1 Anticorps) interactions. This may be relevant to other virus entry processes employing EGFR (Montrer EGFR Anticorps).
Here we elucidate G1P3, a survival protein induced by interferons (IFNs), as a target of estrogen signaling and a contributor to poor outcomes in estrogen receptor (Montrer ESR1 Anticorps)-positive (ER(+)) breast cancer.
PRINS regulates G1P3, a gene with anti-apoptotic effects in keratinocytes. siRNA-mediated inhibition of PRINS gene resulted in altered cell morphology and gene expression alterations.
G1P3 protein may have function as a cell survival protein by inhibiting mitochondrial-mediated apoptosis
Respiratory syncytial virus upregulated the mRNA expression of chemokines CC and CXC and interfered with type alpha/beta interferon-inducible gene expression by upregulation of MG11 (Montrer SAMHD1 Anticorps) and downregulation of G1P3.
IFN-alpha2b (Montrer ADRA2B Anticorps) induced the expression of G1P3 and antagonized the TRAIL induced apoptosis in myeloma suggesting that either the deregulated or the induced expression of G1P3 could lead to apoptosis resistance in tumor cells.
This gene was first identified as one of the many genes induced by interferon. The encoded protein may play a critical role in the regulation of apoptosis. A minisatellite that consists of 26 repeats of a 12 nucleotide repeating element resembling the mammalian splice donor consensus sequence begins near the end of the second exon. Alternatively spliced transcript variants that encode different isoforms by using the two downstream repeat units as splice donor sites have been described.
interferon alpha-inducible protein 6
, interferon, alpha-inducible protein clone IFI-6-16
, interferon-induced protein 6-16