Matrilin 3 (MATN3) Kits ELISA

MATN3 encodes a member of von Willebrand factor A domain containing protein family. De plus, nous expédions Matrilin 3 Anticorps (69) et Matrilin 3 Protéines (4) et beaucoup plus de produits pour cette protéine.

list all ELISA KIts Gène GeneID UniProt
MATN3 4148 O15232
MATN3 17182 O35701
Anti-Rat MATN3 MATN3 313954  
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Top Matrilin 3 Kits ELISA sur anticorps-enligne.fr

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Catalogue No. Reactivité Sensibilité Gamme Images Quantité Livraison Prix Détails
Humain 2 pg/mL n/a   96 Tests 11 to 16 Days
$673.75
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Lapin
  96 Tests 26 to 36 Days
$899.80
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Lapin
  96 Tests 31 to 41 Days
$728.20
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Souris
  96 Tests 21 to 31 Days
$796.95
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Humain
  96 Tests 21 to 31 Days
$546.70
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Plus Kits ELISA pour Matrilin 3 partenaires d'interaction

Human Matrilin 3 (MATN3) interaction partners

  1. Our data highlights the importance of detection and careful characterization of intragenic duplication CNVs, presenting them as a novel and very rare genetic mechanism in IFT81-related Jeune syndrome and MATN3-related MED.

  2. miR-448 contributed to the progression of osteoarthritis by directly targeting matrilin-3.

  3. Study confirmed that MATN3 protein was highly expressed in GAC patients, and MATN3 overexpression could be used as an independent predictor of poor prognosis in GAC patients.

  4. our results revealed miR-483-5p directly targeted to the cartilage matrix protein matrilin 3 (Matn3) and tissue inhibitor of metalloproteinase 2 (Timp2) to stimulate chondrocyte hypertrophy, extracellular matrix degradation, and cartilage angiogenesis, and it consequently initiated and accelerated the development of OA.

  5. The results of the study indicate a potential role for the MATN3 rs28598872 polymorphism in the pathogenesis of Temporomandibular Joint Internal Derangement.

  6. This report is the first to show the involvement of MATN3 in C-type natriuretic peptide/natriuretic peptide receptor-B signaling pathway during the process of transforming growth factor-beta induced chondrogenic differentiation of mesenchymal stem cells.

  7. MATN3 plays a regulatory role in cartilage homeostasis due to its capacity to induce IL-1Ra, upregulate gene expression of major cartilage matrix components, and downregulate the expression of OA-associated matrix-degrading proteinases in chondrocytes.

  8. MATN3 may have the inherent ability to inhibit premature chondrocyte hypertrophy by suppressing BMP-2/Smad1 activity

  9. The VWA1 domain of matrilin-3 is primarily responsible for the induction of IL-6 release from primary human chondrocytes.

  10. Polymorphism in the MATN3 gene might play a role in osteoarthritis in the Chinese Han population.

  11. Haplotype-4 of MATN3 is associated with vertebral fracture risk independent of bone mineral density in Chinese postmenopausal women.

  12. MATN3 mutations were identified in 13 multiple epiphyseal dysplasia patients and comprised predominantly of missense mutations.

  13. Radiographic findings in patients with COMP and MATN3 mutations showed marked abnormalities in hip and knee joints.

  14. a matrilin-3 mutation associated with osteoarthritis does not affect collagen affinity but promotes the formation of wider cartilage collagen fibrils

  15. increased expression of MATN3 in osteoarthritis might contribute to the degeneration of articular cartilage.

  16. potential of matrilin-3 to modulate gene expression profile of primary chondrocytes; tested matrilin3-dependent induction of pro-inflammatory cytokines, inducible nitric oxide synthetase & cyclooxygenase-2, MMP1, -3 & -13, & matrilin-3 itself

  17. Mutation in MATN3 had significant association for patients with osteoarthritis.

  18. Four novel missense mutations and one recurrent missense mutation were identified in MATN3 in seven families with multiple epiphyseal dysplasia.

  19. MATN3 mutations is associated with multiple epiphyseal dysplasia

  20. Contrary to the previous assumption that the MATN3 mutation in multiple epiphyseal dysplasia is confined to the beta-sheet regions, one novel mutation is located outside the beta-sheet region, within an alpha-helix region

Mouse (Murine) Matrilin 3 (MATN3) interaction partners

  1. Data show that hand osteoarthritis (HOA)-related matrilin-3 mutation (T298M) leads to a high expression level of growth arrest DNA damage-inducible gene 153 (GADD153)

  2. MATN3 plays a regulatory role in cartilage homeostasis due to its capacity to induce IL-1Ra, upregulate gene expression of major cartilage matrix components, and downregulate the expression of OA-associated matrix-degrading proteinases in chondrocytes.

  3. MATN3 may have the inherent ability to inhibit premature chondrocyte hypertrophy by suppressing BMP-2/Smad1 activity

  4. study will facilitate better awareness of the differential diagnoses that might be associated with the PSACH/MED spectrum and subsequent care of PSACH/MED patients

  5. Lack of COMP and matrilin 3 leads to increased deposition of TIMP-3, which causes partial inactivation of matrix metalloproteinases in bone, including MMP-13.

  6. Secretion of matrilin 3 V194D mutant protein is not dependent on hetero-oligomerization with matrilin 1.

  7. matrilin-3 mutation associated with osteoarthritis does not affect collagen affinity but promotes the formation of wider cartilage collagen fibrils

  8. potential of matrilin-3 to modulate gene expression profile of primary chondrocytes; tested matrilin3-dependent induction of pro-inflammatory cytokines, inducible nitric oxide synthetase & cyclooxygenase-2, MMP1, -3 & -13, & matrilin-3 itself

  9. Expression of matrilin-3 during maturation of mouse skeletal tissues

  10. To assess the function of matrilin-3 during skeletal development, we have generated Matn-3 null mice; phenotypes of multiple epiphyseal dysplasia disorders are not caused by the absence of matrilin-3 in cartilage ECM.

  11. Matrilin-3 can be considered as an interface component, capable of interconnecting macromolecular networks

  12. The lack of Matn3 does not lead to postnatal chondrodysplasia but accounts for higher incidence of osteoarthritis.

  13. Matrilin-1 and matrilin-3 modulate collagen fibrillogenesis in cartilage and show that biochemical compensation might exist between matrilins, evidenced by Matn3 deficiency.

  14. a matn3 mutation causes decreased chondrocyte proliferation and dysregulated apoptosis leading to epiphyseal dysplasia

Cow (Bovine) Matrilin 3 (MATN3) interaction partners

  1. Data suggest that matrilins 1 and 3 dose-dependently mediate weak cell attachment without promoting focal adhesion formation.

Matrilin 3 (MATN3) profil antigène

Antigen Summary

This gene encodes a member of von Willebrand factor A domain containing protein family. This family of proteins is thought to be involved in the formation of filamentous networks in the extracellular matrices of various tissues. This protein contains two von Willebrand factor A domains\; it is present in the cartilage extracellular matrix and has a role in the development and homeostasis of cartilage and bone. Mutations in this gene result in multiple epiphyseal dysplasia.

Gene names and symbols associated with Matrilin 3 (MATN3) Kits ELISA

  • matrilin 3 (MATN3) anticorps
  • matrilin 3 (Matn3) anticorps
  • AV009181 anticorps
  • DIPOA anticorps
  • EDM5 anticorps
  • HOA anticorps
  • OADIP anticorps
  • OS2 anticorps

Protein level used designations for Matrilin 3 (MATN3) Kits ELISA

matrilin 3 , matrilin-3-like , matrilin-3

GENE ID SPECIES
459514 Pan troglodytes
482985 Canis lupus familiaris
716677 Macaca mulatta
100071902 Equus caballus
100221699 Taeniopygia guttata
100392004 Callithrix jacchus
100470832 Ailuropoda melanoleuca
100603025 Nomascus leucogenys
4148 Homo sapiens
17182 Mus musculus
313954 Rattus norvegicus
395954 Gallus gallus
540041 Bos taurus
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