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Dietary fat absorption from the small intestine is facilitated by acyl-CoA:monoacylglycerol transferase (MOGAT\; EC 22.214.171.124) and acyl-CoA:diacylglycerol acyltransferase (DGAT\; see MIM 604900) activities. De plus, nous expédions MOGAT2 Anticorps (40) et MOGAT2 Protéines (5) et beaucoup plus de produits pour cette protéine.
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MGAT2 (Montrer MGAT2 Kits ELISA) in the intestine plays an indispensable role in enhancing metabolic efficiency but also raise the possibility that MGAT2 (Montrer MGAT2 Kits ELISA) in other tissues may contribute to the regulation of energy metabolism.
MGAT2 possessed an intrinsic acyl-CoA:diacylglycerol acyltransferase (DGAT) activity which could provide an alternative pathway for triacylglycerol synthesis in the absence of DGAT
Mogat2 is a key determinant of energy metabolism in response to dietary fat and suggest that inhibition or deletion of this enzyme may prove to be a useful strategy for treating obesity.
the release of gut (Montrer GUSB Kits ELISA) peptides following oral triglyceride ingestion in MGAT2 (Montrer MGAT2 Kits ELISA) and DGAT1 (Montrer DGAT1 Kits ELISA) knock-out mice was investigated.
MGAT (Montrer MGAT2 Kits ELISA)catalyzes the synthesis of diacylglycerol, a precursor of triacylglycerol. MGAT mediates the absorption of dietary fat by catalyzing the resynthesis of triacylglycerol in enterocytes, a step required for delivering absorbed fat to other tissues.
MGAT2 (Montrer MGAT2 Kits ELISA) is a monoacylglycerol acyltransferase expressed in the small intestine
MGAT2 (Montrer MGAT2 Kits ELISA) functions as a dimeric or tetrameric protein and selectively heterodimerizes with DGAT1 (Montrer DGAT1 Kits ELISA) in mammalian cells
Mogat2(IKO) mice increased energy expenditure although to a lesser degree than Mogat2(-/-) mice and were protected against diet-induced weight gain and associated comorbidities
The described cell-based assay adds a new methodology for the development and evaluation of MGAT2 (Montrer MGAT2 Kits ELISA) inhibitors for the treatment of obesity and type 2 diabetes
Diacylglycerol acyltransferase-2 (Montrer AWAT1 Kits ELISA) and monoacylglycerol acyltransferase-2 are ubiquitinated proteins that are degraded by the 26S proteasome (Montrer Psmd4 Kits ELISA)
The use of 1-oleoyl-glycerol-d5 and (U13)C-TG oil followed by LC/ESI (Montrer PI3 Kits ELISA)/MS/MS detection of stable-isotopic labeled DAG, TG, or glycerol provides a wide range of applications to study pathophysiological regulation of the monoacylglycerol pathway and MGAT2 (Montrer MGAT2 Kits ELISA) activity.
MGAT2 (Montrer MGAT2 Kits ELISA) may play an important role in dietary fat absorption
Dietary fat absorption from the small intestine is facilitated by acyl-CoA:monoacylglycerol transferase (MOGAT\; EC 126.96.36.199) and acyl-CoA:diacylglycerol acyltransferase (DGAT\; see MIM 604900) activities. MOGAT catalyzes the joining of monoacylglycerol and fatty acyl-CoAs to form diacylglycerol (Yen and Farese, 2003
2-acylglycerol O-acyltransferase 2
, acyl CoA:monoacylglycerol acyltransferase 2
, monoacylglycerol O-acyltransferase 2
, putative monoacylglycerol acyltransferase 2
, 2-acylglycerol O-acyltransferase 2-like
, acyl-CoA:monoacylglycerol acyltransferase 2
, diacylglycerol acyltransferase 2-like protein 5
, diacylglycerol O-acyltransferase candidate 5
, 2-acylglycerol O-acyltransferase 2-B
, acyl CoA:monoacylglycerol acyltransferase 2-B
, acyl-CoA:monoacylglycerol acyltransferase 2-B
, monoacylglycerol O-acyltransferase 2-B