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The protein encoded by NPPA belongs to the natriuretic peptide family.
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Human Polyclonal NPPA Primary Antibody pour IHC, IHC (p) - ABIN4282388
Somanna, Yariswamy, Garagliano, Siebenlist, Mummidi, Valente, Chandrasekar: Aldosterone-induced cardiomyocyte growth, and fibroblast migration and proliferation are mediated by TRAF3IP2. dans Cellular signalling 2015
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Human Polyclonal NPPA Primary Antibody pour IF (p), IHC (p) - ABIN676928
Straub, Ehrenstein, Günther, Rauch, Trendafilova, Boschiero, Grifka, Fleck: Increased extracellular water measured by bioimpedance and by increased serum levels of atrial natriuretic peptide in RA patients-signs of volume overload. dans Clinical rheumatology 2016
Human Monoclonal NPPA Primary Antibody pour IHC (p), RIA - ABIN2477552
Meyer, Keller, Bur, Hollender: [The giant ulcer of the stomach. Notes concerning 30 cases (author's transl)]. dans La semaine des hôpitaux : organe fondé par l'Association d'enseignement médical des hôpitaux de Paris 1980
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Human Polyclonal NPPA Primary Antibody pour FACS, IHC (p) - ABIN652474
Nam, Lubczyk, Bhakta, Zang, Fernandez-Perez, McAnally, Bassel-Duby, Olson, Munshi: Induction of diverse cardiac cell types by reprogramming fibroblasts with cardiac transcription factors. dans Development (Cambridge, England) 2014
Expression of atrial natriuretic peptide, C-type natriuretic peptide-1, and the guanylyl cyclase-linked (GC) natriuretic peptide receptor-A (Montrer NPR1 Anticorps) and -B-type receptors was determined by PCR from the mRNA of head kidney leucocytes
Atrial natriuretic peptide and vasopressin (Montrer AVP Anticorps)-presence in the ciliary body of eye in the pig (sus domesticus).
ANP and BNP (Montrer BNC2 Anticorps) gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.
Up-regulation of ANP and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase (Montrer CA-P60A Anticorps) 2a and phospholamban (Montrer PLN Anticorps) in failing myocardium are observed in a porcine model of tachycardia.
Arial natriuretic factor (ANF (Montrer HESX1 Anticorps)) and N-terminal proANF may be better suited than brain natriuretic peptide (Montrer BNP Anticorps) as markers of cardiac preload during the development and treatment of acute heart failure.
Stimulation of prepubertal granulosa cells with FSH (Montrer BRD2 Anticorps) resulted in a moderate expression of ANP. When FSH (Montrer BRD2 Anticorps) treatment was followed by LH, the pronounced expression of ANP was observed in all cells.
ANP in plasma is increased in horses with heart disease associated with altered left-sided chamber dimensions and/or function.
Cardiac ANP was increased in horses with mitral regurgitation (MR) and cardiac troponin levels were low in healthy and MR affected horses.
An increase in year-by-year BP variability over the past 6 years is associated with elevated plasma levels of ANP and BNP (Montrer BNC2 Anticorps) in the Japanese general population.
In summary, there were no significant differences in ANP and BNP (Montrer BNC2 Anticorps) levels after percutaneous epicardial left atrial appendage suture ligation using LARIAT device 3 months after procedure.
NPPA SNPs were not associated with insulin (Montrer INS Anticorps) resistance in older adults.
TIMP-4 (Montrer TIMP4 Anticorps), NT-proANP, NT-proBNP were strongest associated with PAF (Montrer PAF Anticorps) and AHRE (Montrer AHR Anticorps). The discriminatory performance of CHADS2-VASc for PAF (Montrer PAF Anticorps) was increased by addition of selected biomarkers.
ANP inhibits TGF-beta1 (Montrer TGFB1 Anticorps)-induced EMT (Montrer ITK Anticorps) in 16HBE-14o and A549 cells through cGMP/PKG (Montrer PRKG1 Anticorps) signaling, by which it targets TGF-beta1 (Montrer TGFB1 Anticorps)/Smad3 (Montrer SMAD3 Anticorps) via attenuating phosphorylation of Smad3 (Montrer SMAD3 Anticorps). These findings suggest the potential of ANP in the treatment on pulmonary diseases with airway remodeling.
the ANP T1766C gene polymorphism may be associated with the risk of essential hypertension, and the 1766C allele may be a protective factor against the disease
Corin (Montrer CORIN Anticorps) and atrial natriuretic peptide A were most abundant in the proximal convoluted tubules and the medullary connecting ducts.
Higher levels of PCT (Montrer UROD Anticorps), a marker of infection, and MRproANP, a marker for hemodynamic stress, were independently associated with ischemic stroke risk. PCT (Montrer UROD Anticorps) was specifically associated with small vessel and MRproANP with cardioembolic stroke risk.
The present results demonstrate that C2238/ANP-minor allele carrier status is an independent risk factor for acute coronary syndromes recurrence in an Italian cohort of ischemic heart disease patients over the long term, and they support the role of C2238/ANP-minor allele as a negative prognostic factor in coronary artery disease patients.
Both NT-proANP and NT-proBNP levels were higher in coronary artery disease patients experiencing MACEs following percutaneous coronary intervention in a non-primary setting. Notably, only NT-proANP levels significantly affected prognosis after PCI (Montrer SERPINA5 Anticorps).
ANP acts as an anti-permeability factor by inhibiting the signaling functions of vascular permeability factor (Montrer VEGFA Anticorps) and by preserving the endothelial cell tight junctions functional morphology
These studies support the therapeutic potential of dual inhibition of NEP (Montrer MME Anticorps) and ACE (Montrer ACE Anticorps) in the prevention of increased arterial ECP (Montrer ECP Anticorps) and atherogenesis which may be linked to the ANP/cGMP system.
Diabetes may modify the vascular actions of ANP in isolated rabbit carotid arteries and the mechanisms involved in these actions; ANP induces relaxation of precontracted carotid arteries, which is lower in diabetic than in control rabbits.
The study suggests that Beta1-adrenergic receptor activation decreases atrial natriuretic peptide release via cAMP- and Ca(2 (Montrer CA2 Anticorps)+)-dependent mechanisms.
The present study provides a novel insight into the potential underlying mechanisms of telmisartan-induced inhibition of cardiomyocyte hypertrophy, which involves inhibition of NFAT (Montrer NFATC1 Anticorps) activation, nuclear translocation and the ANP/BNP (Montrer BNC2 Anticorps) cascade.
we investigated pregnancies of ANP wild type, heterozygous, and knockout mice. Maternal blood pressure did not differ between genotypes, and fetal weight was unaffected. Placental weight was greater in ANP(-/-) versus ANP(+/+) mice. Therefore, in our hands, the ANP model does not express phenotypic features of preeclampsia with fetal growth restriction.
Data (including data from studies in knockout mice) suggest expression of ANP/BNP (Montrer BNC2 Anticorps)/GC-A (Montrer GUCA1A Anticorps) (atrial natriuretic peptide, brain natriuretic peptide (Montrer BNP Anticorps), and guanylyl cyclase-A (Montrer NPR1 Anticorps)) is important in embryonic neovascularization and organogenesis.
Goalpha plays a role in ANF (Montrer HESX1 Anticorps) sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF (Montrer HESX1 Anticorps) and BNP (Montrer BNC2 Anticorps).
ANP prevents cancer metastasis by inhibiting the adhesion of tumor cells to inflamed endothelial cells
Expression of NPPA splice variants is differentially regulated in the developing mouse heart.
NPPA acts on the NPR1 receptor, is expressed in mouse dorsal root and trigeminal ganglia, and the spinal cord.
Natriuretic peptides buffer renin (Montrer REN Anticorps)-dependent hypertension.
Data indicate that the identified stress-responsive enhancer of Nppa and Nppb (Montrer BNP Anticorps), the most common markers of heart failure, is a 650-bp fragment within 50 kb of the Nppa and Nppb (Montrer BNP Anticorps) loci.
Absence of maternal atrial natriuretic peptide leads to gestational hypertension and adaptive changes fetal cardiac growth and blood pressure regulation.
The protein encoded by this gene belongs to the natriuretic peptide family. Natriuretic peptides are implicated in the control of extracellular fluid volume and electrolyte homeostasis. This protein is synthesized as a large precursor (containing a signal peptide), which is processed to release a peptide from the N-terminus with similarity to vasoactive peptide, cardiodilatin, and another peptide from the C-terminus with natriuretic-diuretic activity. Mutations in this gene have been associated with atrial fibrillation familial type 6.
atrial natriuretic peptide
, atrial natriuretic factor
, natriuretic peptides A
, natriuretic peptide precursor A
, prohormone for atrial natriuretic polypeptide (ANP)
, Natriuretic peptide precursor A (pronatriodilatin, also Anf, Pnd)
, natriuretic peptide precursor type A
, Atrial natriuretic factor
, Atrial natriuretic peptide
, natriuretic peptide type A
, Natriuretic peptides A