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PLAGL1 encodes a C2H2 zinc finger protein with transactivation and DNA-binding activities. De plus, nous expédions PLAGL1 Anticorps (64) et PLAGL1 Protéines (7) et beaucoup plus de produits pour cette protéine.
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ZAC expression was studied in transgenic mice.
report here a novel PLAGL1 promoter (P5) derived from the insertion of a primate-specific, MIR3 SINE retrotransposon. P5 is highly utilized in lymphocytes, particularly in T cells, and like P2, directs biallelic transcription
Plagl1 is a transcription factor that coordinated the regulation of a subset of imprinted gene network genes and controlled extracellular matrix composition.
PLAGL1 methylation/expression may be altered after assisted reproductive technologies.
These results suggest that OCT (Montrer Plxna2 Kits ELISA) attenuates SGC-7901 cell proliferation by enhancing P300-HAT (Montrer EP300 Kits ELISA) activity through the interaction of ZAC and P300 (Montrer EP300 Kits ELISA), causing a reduction in pS10-H3 and an increase in acK14-H3. These findings provide insight for future research on OCT (Montrer Plxna2 Kits ELISA) and further demonstrate the potential of OCT (Montrer Plxna2 Kits ELISA) to be used as a therapeutic agent for gastric cancer
High PLAGL1 mRNA and protein levels were associated with Clear Cell Renal Cell Carcinoma.
Results suggest that dysregulation of PLAGL1 expression may be involved in the progression of colorectal cancer (CRC (Montrer CALR Kits ELISA)) but the patient survival data do not confirm applicability of the PLAGL1 expression level as a prognostic factor in CRC (Montrer CALR Kits ELISA).
Fetal growth can be influenced by altered expression of the PLAGL1 gene network in human placenta.
DNA deletion and promoter hyper-methylation both contribute to the down-regulation of PLAGL1 in gastric adenocarcinoma
There is positive correlations between the ZAC1 DMR methylation index (MI) and estimated fetal weight (EFW) at 32 weeks of gestation, weight at birth and weight at one year of age (respectively, r = 0.15, 0.09, 0.14; P values = 0.01, 0.15, 0.03).
PLAGL1 is maternally imprinted in swine. Expression profiling using short-oligonucleotide microarrays of parthenotes and control fetal tissues supports imprinted isoform-specific expression.
The expression patterns of PLAGL1 and PEG10 (Montrer PEG10 Kits ELISA) genes in two breeds of swine are reported.
Zac1/GPR39 system promotes the formation of type II muscle fibers by phosphorylation of CaMK-II. Cells lacking Zac1/GPR39 system tend to remain stemness and form type I muscle fibers after induced differentiation.
expression levels of Zac1 need to be kept under strict control to avoid premature cell cycle exit, disrupted neurogenesis and aberrant expression of non-neuronal genes including pluripotency associated factors.
Transcription factor MEF2 (Montrer MEF2C Kits ELISA) and Zac1 mediate MIF (Montrer MIF Kits ELISA)-induced GLUT4 (Montrer SLC2A4 Kits ELISA) expression through CD74 (Montrer CD74 Kits ELISA)-dependent AMPK (Montrer PRKAA1 Kits ELISA) activation in cardiomyocytes
we show that the maternally imprinted gene Zac1 is a critical regulator of neocortical development
Zac1 interacts with two cis-regulatory elements in the Tcf4 (Montrer TCF4 Kits ELISA) gene locus.
results indicate ZAC is a negative regulator of the acute stimulatory effects of glucose on beta-cells. It provides an explanation for both insulin (Montrer INS Kits ELISA) deficiency in the neonate and the later relapse of diabetes in patients with transient neonatal diabetes mellitus cases
Hymai and Plagl1it RNAs both have potentially high affinity for Trithorax chromatin regulators.
identified the guanine nucleotide exchange factor Rasgrf1 (Montrer RASGRF1 Kits ELISA) as a direct Zac1/Plagl1 target gene in beta cells
Zac1 inhibits nuclear factor Kappa B activity by interacting with the C-terminus of the p65 (Montrer NFkBP65 Kits ELISA) subunit, which suppresses the phosphorylation of p65 (Montrer NFkBP65 Kits ELISA) at Ser468 and Ser536 residues
This gene encodes a C2H2 zinc finger protein with transactivation and DNA-binding activities. It has been shown to have anti-proliferative properties, and thus thought to function as a tumor suppressor. In addition, overexpression of this gene during fetal development is believed to underlie the rare disorder, transient neonatal diabetes mellitus (TNDM). This gene is imprinted, with preferential expression of the paternal allele in many tissues, however, biallelic expression has been noted in peripheral blood leucocytes. A recent study reports that tissue-specific imprinting results from variable utilization of monoallelic and biallelic promoters. Many transcript variants differing in the 5' UTR and encoding two different isoforms, have been found for this gene.
pleiomorphic adenoma gene-like 1
, zinc finger protein PLAGL1-like
, PLAG-like 1
, lost on transformation 1
, pleiomorphic adenoma-like protein 1
, tumor suppressor ZAC
, tumor supressor ZAC
, zinc finger protein PLAGL1
, zinc finger protein Zac 1
, zinc finger protein regulator of apoptosis and cell cycle arrest
, Lost on transformation 1