Protein S (PROS) Kits ELISA

PROS encodes a vitamin K-dependent plasma protein that functions as a cofactor for the anticoagulant protease, activated protein C (APC) to inhibit blood coagulation. De plus, nous expédions PROS1 Anticorps (170) et PROS1 Protéines (20) et beaucoup plus de produits pour cette protéine.

list all ELISA KIts Gène GeneID UniProt
PROS 5627 P07225
PROS 81750  
PROS 19128 Q08761
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Top PROS1 Kits ELISA sur anticorps-enligne.fr

Showing 10 out of 55 products:

Catalogue No. Reactivité Sensibilité Gamme Images Quantité Fournisseur Livraison Prix Détails
Humain 27.7 pg/mL 62.5 pg/mL - 4000 pg/mL 96 Tests Connectez-vous pour afficher 13 to 16 Days
$700.00
Détails
Singe 3.9 ng/mL 15.6-1000 ng/mL Typical standard curve 96 Tests Connectez-vous pour afficher 15 to 18 Days
$788.33
Détails
Rat 1.95 ng/mL 7.8-500 ng/mL Typical standard curve 96 Tests Connectez-vous pour afficher 15 to 18 Days
$788.33
Détails
Lapin 1.17 ng/mL 4.7-300 ng/mL Typical standard curve 96 Tests Connectez-vous pour afficher 15 to 18 Days
$1,026.67
Détails
Souris 1.56 ng/mL 6.25-400 ng/mL   96 Tests Connectez-vous pour afficher 15 to 18 Days
$910.56
Détails
Boeuf (Vache) 1.95 ng/mL n/a   96 Tests Connectez-vous pour afficher 15 to 18 Days
$1,026.67
Détails
Cobaye 1.0 5.0   96 Tests Connectez-vous pour afficher 15 to 18 Days
$707.14
Détails
Poulet 0.188 ng/mL 0.313-20 ng/mL   96 Tests Connectez-vous pour afficher 12 to 14 Days
$715.00
Détails
Porc 0.188 ng/mL 0.313-20 ng/mL   96 Tests Connectez-vous pour afficher 12 to 14 Days
$715.00
Détails
Chien 1.0 5.0   96 Tests Connectez-vous pour afficher 15 to 18 Days
$707.14
Détails

PROS1 Kits ELISA mieux référencés

  1. Human PROS1 Kit ELISA pour Sandwich ELISA - ABIN456818 : Deng, Zhang, Chen, Yan, Han: Toll-like receptor-mediated inhibition of Gas6 and ProS expression facilitates inflammatory cytokine production in mouse macrophages. dans Immunology 2011 (PubMed)

Plus Kits ELISA pour PROS1 partenaires d'interaction

Human Protein S (PROS) interaction partners

  1. Protein S is an important in vivo inhibitor of FIXa. Disruption of the interaction between PS and FIXa causes an increased rate of thrombus formation in mice.

  2. The frequency of SERPINC1, PROC, and PROS1 mutations and their thromboembolic manifestations was characterized in patients with AT, PC, or PS deficiencies, inhabiting southern Poland.

  3. The authors provide strong evidence that the rare Protein S Heerlen (PSH) mutation variant is associated with venous thrombosis in unselected individuals.

  4. Functional PROS1 variants are more common in the general Swedish population than anticipated.

  5. The present case combined with the review of the literature suggests that p.Arg451* in the PROS1 gene mainly leads to clinically evident thrombosis following trauma, surgery or serious comorbidities especially malignancy.

  6. Protein S and Gas6 mediates phagocytosis of HIV-1-infected cells by bridging receptor tyrosine kinase Mer to phosphatidylserine exposed on infected cells.

  7. these results suggest a novel pathogenic role of SPE B that initiates protein S degradation followed by the inhibition of apoptotic cell clearance by macrophages

  8. Developed functional protein S assays that measure both the activated protein C- and TFPI-cofactor activities of protein S in plasma, which are hardly if at all affected by the FV Leiden mutation.

  9. Taken together, our gain-of-function, loss-of-function analyses suggest that PROS may facilitate cell proliferation and promote castration resistance in human castration-resistant PCa-like cells via its apoptosis-regulating property.

  10. protein S has a clinical relevance and a protective role in pulmonary fibrosis

  11. Due to the versatility exemplified, this probe holds great promise for exploring the role of protein S-nitrosylation in the pathophysiological process of a variety of vascular diseases

  12. There was no association between PS-Tokushima and recurrent pregnancy loss and a PS deficiency or low PS activity was shown not to serve as a reliable clinical predictor of subsequent miscarriage.

  13. we identify PROS1 as a driver of Oral Squamous Cell Carcinoma tumor growth and a modulator of AXL expression

  14. The prevalence of PS de fi ciency in the present study was higher than in Western countries and con fi rms the high prevalence of PS de fi ciency in Asian populations

  15. Patients with type 2 diabetes had significantly lower circulating free protein S than healthy control subjects

  16. In the present study, gene analysis of six unrelated Japanese families diagnosed with congenital protein S deficiency identified five missense mutations in the PROS1 gene - c.757C>T (Ala139Val; A139V), c.1346 G>T (Cys449Phe; C449F), c.1352G>A (Arg451Gln; R451Q), c.1424G>T (Cys475Phe; C475F) and c.1574C>T (Ala525Val; A525V) - and one frameshift mutation, c.2135delA (Asp599ThrfsTer13; D599TfsTer13).

  17. The odds ratio of developing idiopathic fatal pulmonary embolism as a variant carrier for PROS1 is 56.4 (95% CI, 5.3-351.1; P = 0.001).

  18. described a novel PROS1 frameshift mutation, c.74dupA, in a hereditary protein S deficiency family. Interestingly, both of the proband and his mother carried the mutation and had a protein S deficiency, however, only the proband suffered a pulmonary embolism while his mother had no history of any thrombosis, suggesting that a triggering event might have been involved in the thrombus formation.

  19. PROS1 may play an important role in the development of glioblastoma multiforme through cellular proliferation, migration and invasion as well as apoptosis.

  20. Low protein S expression is associated with diabetes mellitus.

Mouse (Murine) Protein S (PROS) interaction partners

  1. Protein S is an important in vivo inhibitor of FIXa. Disruption of the interaction between PS and FIXa causes an increased rate of thrombus formation in mice.

  2. An inhibitory role for PROS1 on BM-derived LCs. Collectively.

  3. This study identifies a duple role for PROS1 in stem-cell quiescence and as a pro-neurogenic factor, and highlights a unique segregation of increased stem cell proliferation from enhanced neuronal differentiation.

  4. Mice overexpressing protein S showed significant improvements in blood glucose level, glucose tolerance, insulin sensitivity, and insulin secretion compared with wild-type counterparts. diabetic protein S transgenic mice developed significantly less severe diabetic glomerulosclerosis than controls.

  5. results support a causal relationship between the PS-K196E mutation and venous thrombosis

  6. By revealing that neural stem-like cells act within the SVZ neurogenic niche as phagocytes and that the ProS/MerTK path represents an endogenous regulatory mechanism for SVZ cell phagocytic activity

  7. Optimal TAM signaling requires coincident TAM ligand engagement of both its receptor and the phospholipid phosphatidylserine regulating TAM receptor tyrosine kinases Tyro3, Axl, and Mer and their ligands Gas6 and Protein S.

  8. Data indicate that activated T cells express Pros1.

  9. Results demonstrate that Protein S is a Mer ligand, and is active in Mer-driven phagocytosis in the retina.

  10. A self-regulatory mechanism of Toll-like receptor signalling through the suppression of Gas6 and ProS expression is described.

  11. Protein S controls hypoxic/ischemic blood-brain barrier disruption through the TAM receptor Tyro3 and sphingosine 1-phosphate receptor1.

  12. Data show that the spectrum of bioactivities of recombinant murine protein S with mouse plasma and smooth muscle cells is similar to that of human protein S.

  13. results demonstrate that ProS is a pleiotropic anticoagulant with activated Protein C-independent activities and highlight new roles for ProS in vascular development and homeostasis

  14. Pregnancy causes a decrease in APC resistance in mice, which can be explained by the elevation of protein S levels and increased TFPI activity in plasma.

Cow (Bovine) Protein S (PROS) interaction partners

  1. Activated protein C(APC) combined with protein S(PS) had significant antithrombotic effect. APC combined with PS prolonged clotting time. Dependence on APC-cofactor activity of PS for expression of anticoagulant activity by APC.

PROS1 (PROS) profil antigène

Antigen Summary

This gene encodes a vitamin K-dependent plasma protein that functions as a cofactor for the anticoagulant protease, activated protein C (APC) to inhibit blood coagulation. It is found in plasma in both a free, functionally active form and also in an inactive form complexed with C4b-binding protein. Mutations in this gene result in autosomal dominant hereditary thrombophilia. An inactive pseudogene of this locus is located at an adjacent region on chromosome 3.

Gene names and symbols associated with Protein S (PROS) Kits ELISA

  • protein S (PROS1) anticorps
  • protein S (Pros1) anticorps
  • protein S (pros1) anticorps
  • protein S (alpha) (Pros1) anticorps
  • AW214361 anticorps
  • PROS anticorps
  • PS21 anticorps
  • PS22 anticorps
  • PS23 anticorps
  • PS24 anticorps
  • PS25 anticorps
  • PSA anticorps
  • THPH5 anticorps
  • THPH6 anticorps
  • zgc:154001 anticorps

Protein level used designations for Protein S (PROS) Kits ELISA

protein Sa , vitamin K-dependent plasma protein S , vitamin K-dependent protein S

GENE ID SPECIES
5627 Homo sapiens
81750 Rattus norvegicus
768180 Danio rerio
19128 Mus musculus
282006 Bos taurus
100009314 Oryctolagus cuniculus
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