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The protein encoded by SMAD9 is a member of the SMAD family, which transduces signals from TGF-beta family members. De plus, nous expédions SMAD9 Protéines (7) et SMAD9 Kits (6) et beaucoup plus de produits pour cette protéine.
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Human Polyclonal SMAD9 Primary Antibody pour EIA, ELISA - ABIN251308
Shi, Massagué: Mechanisms of TGF-beta signaling from cell membrane to the nucleus. dans Cell 2003
Phosphorylated Smad1/5/8/9 specifically bound to the BREs of Smad8/9 gene. The present study reveals that Smad8/9 is a unique R-Smad regulated through the BMP pathway at the mRNA level.
Case Report: hamartomatous olyposis and gastrointestinal ganglioneuromas with SMAD9 germline mutation that reduces PTEN (Montrer PTEN Anticorps) expression.
a detailed computational model for TGF-beta (Montrer TGFB1 Anticorps) signalling that incorporates elements of previous models together with crosstalking between Smad1 (Montrer GARS Anticorps)/5/8 and Smad2 (Montrer SMAD2 Anticorps)/3 channels through a negative feedback loop dependent on Smad7 (Montrer SMAD7 Anticorps).
ATP production by NaF (Montrer IL8 Anticorps) promotes hypertrophy-like changes via activation of phospho-Smad8
TNF (Montrer TNF Anticorps) activated NF-kappaB (Montrer NFKB1 Anticorps) pathway and inhibited the phosphorylation of Smad 1 (Montrer GARS Anticorps)/5/8 and BMP-2 (Montrer BMP2 Anticorps)-induced osteoblastic differentiation in BMMSCs
CREBZF (Montrer CREBZF Anticorps), a novel Smad8-binding protein.
Data suggest that Smads 1, 5 and 8 as potential prognostic markers and therapeutic targets for mTOR (Montrer FRAP1 Anticorps) inhibition therapy of prostate cancer.
Endoglin (Montrer ENG Anticorps) promotes transforming growth factor beta-mediated Smad 1 (Montrer GARS Anticorps)/5/8 signaling and inhibits endothelial cell migration through its association with GIPC (Montrer GIPC1 Anticorps)
Study described the first mutation in SMAD8 in a patient with IPAH, and suggested the involvement of SMAD8 in the pathogenesis of PAH.
5-HT (Montrer DDC Anticorps) transactivates the serine kinase receptor, BMPR 1A (Montrer BMPR1A Anticorps), to activate Smads 1/5/8 via Rho and Rho kinase (Montrer ROCK1 Anticorps) in in bovine and human pulmonary artery smooth muscle cells
Sphingosine 1 phosphate also up-regulated runt-related transcription factor 2 (Runx2 (Montrer RUNX2 Anticorps)) expression through S1PR2 (Montrer S1PR2 Anticorps)/RhoA (Montrer RHOA Anticorps)/ROCK/Smad1 (Montrer SMAD1 Anticorps)/5/8 signaling.
BetA can enhance in vivo osteogenic potentials of BMP2 (Montrer BMP2 Anticorps), possibly via stimulating Smad 1 (Montrer SMAD1 Anticorps)/5/8 and p38 (Montrer CRK Anticorps) pathways, and combination of both agents can be considered as a therapeutic strategy for bone diseases.
findings suggest that Smad9 is a new type of transcriptional regulator in BMP signaling.
The findings demonstrate for the first time that KMUP-1 can promote osteoblast maturation and differentiation in vitro via BMP-2 (Montrer BMP2 Anticorps)/Smad1 (Montrer SMAD1 Anticorps)/5/8 and Wnt (Montrer WNT2 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) pathways.
Data indicate that PDGF (Montrer PDGFA Anticorps)-AA promotes mesenchymal stem cel migration via bone morphogenetic protein 2 (BMP2 (Montrer BMP2 Anticorps))-smad (Montrer SMAD1 Anticorps) proteins smad1 (Montrer SMAD1 Anticorps)/5/8 activation requires lysosome-mediated degradation of PDGF (Montrer PDGFA Anticorps) alpha Receptor (PDGFRalpha).
our data suggest a pivotal role for canonical BMP signaling demonstrating distinguished nonoverlapping function of pSmad8 with pSmad1 and pSmad5 in hfSCs regulation and hair morphogenesis but a redundant role in adult hair progenitors differentiation.
Data show that HtrA1 (Montrer HTRA1 Anticorps) is produced during osteoclastogenesis, and negatively regulates osteoblast differentiation, osteoblast differentiation and BMP2 (Montrer BMP2 Anticorps)-induced Smad1 (Montrer SMAD1 Anticorps)/5/8, ERK1/2 (Montrer MAPK1/3 Anticorps) and p38 (Montrer CRK Anticorps) phosphorylation in pre-osteoblasts.
FGF2 (Montrer FGF2 Anticorps) inhibited BMP9 (Montrer GDF2 Anticorps)-induced osteogenic differentiation by blocking BMP9 (Montrer GDF2 Anticorps)-induced Smads signaling and subsequently reducing Smads dependent up-regulation of ALK1 (Montrer ACVRL1 Anticorps) and ALK2 (Montrer ACRV1 Anticorps) in mesenchymal stem cells.
a detailed computational model for TGF-beta (Montrer TGFB1 Anticorps) signalling that incorporates elements of previous models together with crosstalking between Smad1 (Montrer SMAD1 Anticorps)/5/8 and Smad2 (Montrer SMAD2 Anticorps)/3 channels through a negative feedback loop dependent on Smad7 (Montrer SMAD7 Anticorps).
The protein encoded by this gene is a member of the SMAD family, which transduces signals from TGF-beta family members. The encoded protein is activated by bone morphogenetic proteins and interacts with SMAD4. Two transcript variants encoding different isoforms have been found for this gene.
MAD, mothers against decapentaplegic homolog 9
, Mothers against decapentaplegic-like protein 9
, mothers against decapentaplegic homolog 9
, SMAD family member 9
, mothers against decapentaplegic homolog 9-like
, MAD homolog 9
, Mothers against decapentaplegic, drosophila, homolog of, 9
, SMAD, mothers against DPP homolog 9
, mothers against DPP homolog 9
, SMAD 9
, SMAD, mothers against DPP 8
, TGF-beta signal transducer Smad8