Serum Amyloid A3 (SAA3) Kits ELISA

Major acute phase reactant. De plus, nous expédions et beaucoup plus de produits pour cette protéine.

list all ELISA KIts Gène GeneID UniProt
SAA3 20210 P04918
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Top SAA3 Kits ELISA sur

Showing 6 out of 7 products:

Catalogue No. Reactivité Sensibilité Gamme Images Quantité Livraison Prix Détails
Souris 0.039 ng/mL 0.156-10 ng/mL Typical standard curve 96 Tests 13 to 16 Days
Humain 1.2ng/ml 4.7-300 ng/mL   96 Tests 13 to 16 Days
Porc 0.469 ng/mL 0.781-50 ng/mL   96 Tests 12 to 14 Days
Poulet 0.469 ng/mL 0.781-50 ng/mL   96 Tests 12 to 14 Days
Lapin 0.375 ng/mL 0.625-40 ng/mL   96 Tests 12 to 14 Days
Singe 0.469 ng/mL 0.781-50 ng/mL   96 Tests 12 to 14 Days

SAA3 Kits ELISA mieux référencés

  1. Human SAA3 Kit ELISA pour Sandwich ELISA - ABIN365343 : Puthanveetil, Chen, Feng, Gautam, Chakrabarti: Long non-coding RNA MALAT1 regulates hyperglycaemia induced inflammatory process in the endothelial cells. dans Journal of cellular and molecular medicine 2015 (PubMed)

  2. Mouse (Murine) SAA3 Kit ELISA pour Sandwich ELISA - ABIN1095248 : Zhuang, Xu, Wang, Sun, Huang, Wang: IL-6 induced lncRNA MALAT1 enhances TNF-α expression in LPS-induced septic cardiomyocytes via activation of SAA3. dans European review for medical and pharmacological sciences 2017 (PubMed)

Plus Kits ELISA pour SAA3 partenaires d'interaction

Cow (Bovine) Serum Amyloid A3 (SAA3) interaction partners

  1. Twenty four hours post E. coli stimulation, IL1b, IL6, IL8, TNF and LAP gene expression was decreased compared to 6h, whereas TAP and SAA3 expression was further increased to 209- and 3452-fold (P<0.05).

  2. SAA3 serves an important tissue-specific function for the welfare of the mammary gland during both bacterial infection and tissue remodeling.

Mouse (Murine) Serum Amyloid A3 (SAA3) interaction partners

  1. Findings indicate that endogenous SAA3 regulates lung development and homeostasis, and is required for protection against H1N1 influenza infection.

  2. SAA3 is a major hepatic acute-phase SAA in mice that may produce systemic effects during inflammation

  3. SAA3 augments atherosclerosis in ApoE-knockout mice.

  4. N-terminal region of serum amyloid A3 is responsible for up-regulation of MUC2 mRNA expression in mouse epithelial cells.

  5. High SAA3 expression in the stromal component is associated with pancreatic tumors.

  6. This study suggests that the level of expression of the Saa3 gene could be utilized for the number of infiltrated macrophages in obese adipose tissue.

  7. BMDC lacking SAA3 demonstrate an impaired endotoxin tolerance response and inhibited responses to retinoic acid. Our findings indicate that endogenous SAA3 modulates metabolic and immune homeostasis

  8. The induction of Saa3 by PTH may explain the suppression of bone formation when PTH is applied continuously and may be a new therapeutic target for osteoporosis.

  9. results also suggest that Saa3 influences liver-specific SAA1/2 expression, and that SAA3 could play a larger role in the acute phase response than previously thought

  10. Expression of Saa3 in osteoblasts positively correlates with increased cellular maturation toward the osteocyte phenotype.

  11. Serum amyloid A is a retinol binding protein that transports retinol during bacterial infection.

  12. These results suggest that SAA3 plays a role in host innate immunity in the colon by up-regulating MUC2 mucin production, which builds a physiological barrier of colonic epithelia against bacterial invasion.

  13. these data suggest a novel mechanism by which Mo MDSCs mediate inflammation through SAA3-TLR2 signaling and thus exacerbate cancer progression by a STAT3-dependent mechanism.

  14. Hypoxia leads to a substantial increase in SAA3 mRNA and protein level, apparently in a time-dependent manner (threefold in 48 h), in fully differentiated 3T3-L1, followed by reestablishment of gene expression to basal levels after 24 h of reoxygenation.

  15. Using various synthetic peptide fragments, it was shown that SAA3 directly binds MD-2 and activates the MyD88-dependent TLR4/MD-2 pathway, induced IL-6 and TNF-alpha, and recruited CD11b(+)Gr-1(+) cells to the lung.

  16. HSV-1 induces and activate TLR2 and TLR4 receptors directly through interaction of astrocytes with the pathogen and also indirectly by endogenous ligands produced locally, such as serum amyloid A, potentiating the neuroinflammatory response.

  17. Saa3 is expressed in the lungs of mice exposed to several mixed T helper (Th) type 2/Th17-polarizing allergic sensitization regimen and is implicated in the pathogenesis of experimental allergic asthma.

  18. cAMP in combination with TNF specifically induced C/EBPbeta protein, leading to enhanced SAA3 expression but requiring NF-kappaB in mouse granulose cells. The data indicate SAA may play a role in events occurring during the ovulation process.

  19. A 210-bp fragment of the mouse SAA3 promoter when placed in front of the LacZ gene was sufficient to confer basal and inflammation-induced reporter gene expression.

  20. tumor necrosis factor-alpha likely increased serum amyloid A 3 promoter activity and protein by activating nuclear factor-kappaB signaling via tumor necrosis factor receptor 1 in mouse granulosa cells

SAA3 profil antigène

Antigen Summary

Major acute phase reactant. Apolipoprotein of the HDL complex.

Gene names and symbols associated with Serum Amyloid A3 (SAA3) Kits ELISA

  • serum amyloid A 3 (SAA3) anticorps
  • serum amyloid A 3 (Saa3) anticorps
  • serum amyloid A-3 (SAA3) anticorps
  • serum amyloid A3 (LOC100860781) anticorps
  • AV098916 anticorps
  • l7R3 anticorps
  • Saa-3 anticorps

Protein level used designations for Serum Amyloid A3 (SAA3) Kits ELISA

serum amyloid A-3 protein , serum amyloid A

281474 Bos taurus
20210 Mus musculus
100009138 Oryctolagus cuniculus
100860781 Capra hircus
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