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promotes cell adhesion and outgrowth of embryonic hippocampal neurons [RGD, Feb 2006].. De plus, nous expédions SPON2 Anticorps (102) et SPON2 Protéines (12) et beaucoup plus de produits pour cette protéine.
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Human SPON2 Kit ELISA pour Sandwich ELISA - ABIN421013
Lucarelli, Rutigliano, Bettocchi, Palazzo, Vavallo, Galleggiante, Trabucco, Di Clemente, Selvaggi, Battaglia, Ditonno: Spondin-2, a secreted extracellular matrix protein, is a novel diagnostic biomarker for prostate cancer. dans The Journal of urology 2013
Show all 3 Pubmed References
Spondin-2 overexpression contributes to tumor aggressiveness and prognosis in gastric cancer.
SPON2 is a transcriptional target of the metastasis gene MACC1 (Montrer MACC1 Kits ELISA). SPON2 induces cell motility in vitro and CRC (Montrer CALR Kits ELISA) metastasis in mice. In patients, SPON2 serves as prognostic indicator for CRC (Montrer CALR Kits ELISA) metastasis and survival, and might represent a promising target for therapeutic approaches.
Data suggest that spondin-2 could be an independent diagnostic and prognostic biomarker of colon cancer.
Mindin protects against vascular hyperplasia by suppression of abnormal VSMC proliferation, migration and phenotypic switching in an AKT (Montrer AKT1 Kits ELISA)-dependent manner.
Using tissue microarray by immunohistostaining, we found SPON2 to be over-expressed in prostate cancer (PCa). SPON2 staining was more intense in Gleason score sum 7-8 and in PCa patients with metastasis.
mindin serves as a novel mediator that protects against cardiac hypertrophy and the transition to heart failure by blocking AKT (Montrer AKT1 Kits ELISA)/GSK3beta and TGF-beta1 (Montrer TGFB1 Kits ELISA)-Smad (Montrer SMAD1 Kits ELISA) signalling.
High Mindin is associated with diabetic nephropathy in type 2 diabetes.
Spondin 2, which is regulated by T(3), has an important role in cell invasion, cell migration, and hepatoma tumor progression.
The structure of the F-spondin (Montrer SPON1 Kits ELISA) domain of mindin was determined at 1.8-A resolution.
Mindin is a novel modulator of hepatic I/R injury through regulating inflammatory responses, as well as hepatocyte apoptosis and proliferation via inactivation of the Akt (Montrer AKT1 Kits ELISA) signaling pathway.
Spondin 2 regulates hepatic lipid metabolism and alleviates hepatic steatosis, obesity, inflammation, and insulin (Montrer INS Kits ELISA) resistance via its interaction with PPARalpha (Montrer PPARA Kits ELISA).
The data of this study demonstrated that mindin is a critical mediator of ischemic brain injury in an experimental stroke model.
Data show that mindin, as an intrinsic cardioprotective factor, prevents maladaptive remodelling and the transition to heart failure by blocking AKT/GSK3beta signalling.
Mindin expression is upregulated during intestinal inflammation and may induce NF-kappaBp65 promoter activation in a TLR-9 (Montrer TLR9 Kits ELISA) mediated manner.
Mindin is a pattern-recognition molecule for microbial pathogens.
Mindin-integrin interactions play a key role in regulating Rho GTPase (Montrer RACGAP1 Kits ELISA) expression in DCs and DC priming of T lymphocytes.
mindin plays an essential role in the host innate immune response to influenza virus infection and suggest that mindin may be used as an immune-enhancing agent in influenza infection.
mindin participates in integrin-dependent trafficking of eosinophils and can contribute to the severity of allergic airways disease.
promotes cell adhesion and outgrowth of embryonic hippocampal neurons
, differentially expressed in cancerous and non-cancerous lung cells 1