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Contributes to the lung's defense against inhaled microorganisms. De plus, nous expédions Surfactant Protein D Kits (85) et Surfactant Protein D Protéines (28) et beaucoup plus de produits pour cette protéine.
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Pig (Porcine) Monoclonal SFTPD Primary Antibody pour ICC, IHC (fro) - ABIN445379
Soerensen, Holmskov, Aalbaek, Boye, Heegaard, Nielsen: Pulmonary infections in swine induce altered porcine surfactant protein D expression and localization to dendritic cells in bronchial-associated lymphoid tissue. dans Immunology 2005
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Human Polyclonal SFTPD Primary Antibody pour IF (p), IHC (p) - ABIN731678
Sung, Liang, Lee, Yen, Hsiao, Wang, Jiang-Shieh, Tsai, Chen: The protective effect of eupafolin against TNF-?-induced lung inflammation via the reduction of intercellular cell adhesion molecule-1 expression. dans Journal of ethnopharmacology 2015
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Rat (Rattus) Monoclonal SFTPD Primary Antibody pour IA, IHC (p) - ABIN2192111
Kasper, Albrecht, Grossmann, Grosser, Schuh, Müller: Monoclonal antibodies to surfactant protein D: evaluation of immunoreactivity in normal rat lung and in a radiation-induced fibrosis model. dans Experimental lung research 1995
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Pig (Porcine) Monoclonal SFTPD Primary Antibody pour IHC (fro), IF - ABIN2476640
: The influence of varicocele on parameters of fertility in a large group of men presenting to infertility clinics. World Health Organization. dans Fertility and sterility 1992
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Human Monoclonal SFTPD Primary Antibody pour IHC, WB - ABIN395257
Uhliarova, Kopincova, Adamkov, Svec, Calkovska: Surfactant proteins A and D are related to severity of the disease, pathogenic bacteria and comorbidity in patients with chronic rhinosinusitis with and without nasal polyps. dans Clinical otolaryngology : official journal of ENT-UK ; official journal of Netherlands Society for Oto-Rhino-Laryngology & Cervico-Facial Surgery 2017
Human Monoclonal SFTPD Primary Antibody pour EIA, IHC (fro) - ABIN111974
Bräuer, Kindler, Jäger, Sel, Nölle, Pleyer, Ochs, Paulsen: Detection of surfactant proteins A and D in human tear fluid and the human lacrimal system. dans Investigative ophthalmology & visual science 2007
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these findings indicate that the membrane-type surfactant protein D serve as an effective therapeutic strategy for inhibiting macrophage-mediated xenograft rejection in xenotransplantation
Assays that can separate SP-D proteolytic breakdown products or modified forms from naturally occurring SP-D trimers may result in optimal disease markers for pulmonary inflammatory diseases
TGF beta (Montrer TGFB1 Anticorps) markedly decreased expression of SP-A, SP-B, SP-C (Montrer SFTPC Anticorps), fatty acid synthase (Montrer FASN Anticorps), and the phospholipid transporter ABCA3 (Montrer ABCA3 Anticorps). However, TGF beta (Montrer TGFB1 Anticorps) increased protein levels of SP-D with little change in mRNA levels.
the SPA (Montrer FASL Anticorps) and SPD (Montrer HOXD13 Anticorps) levels in EBC were correlated with lung function, which contributed to COPD (Montrer ARCN1 Anticorps) diagnosis.
Studied predictive value of surfactant protein D (SP-D) in lung cancer patients with interstitial lung disease induced by anticancer agents (ILD-AA). Results suggest that SP-D level change was a risk factor for mortality in patients with ILD-AA, and that SP-D might be a predictive prognostic biomarker of ILD-AA.
SP-D also delays FasL (Montrer FASL Anticorps)-induced death of primary human T cells. SP-D delaying the progression of the extrinsic pathway of apoptosis could have important implications in regulating immune cell homeostasis at mucosal surfaces
findings suggest that these pulmonary markers could be useful to assess CAP severity and, especially YKL-40 (Montrer CHI3L1 Anticorps) and CCL18 (Montrer CCL18 Anticorps) by helping predict CAP caused by atypical pathogens
Trimeric SP-D wildtype recognized larger LPS (Montrer IRF6 Anticorps) inner core oligosaccharides with slightly enhanced affinity than smaller compounds suggesting the involvement of stabilizing secondary interactions.
rs2819096 in the surfactant protein D (SFTPD) gene was associated with a higher risk of COPD (Montrer ARCN1 Anticorps) GOLD III + IV.
SP-D increases the formation of nuclear and membrane blebs. Inhibition of caspase-8 (Montrer CASP8 Anticorps) confirms the effect of SP-D is unique to the caspase-8 (Montrer CASP8 Anticorps) pathway.
Elevation of SP-D is found in bronchoalveolar lavage fluid from inoculated lobes of calves infected with bovine adenovirus type 3.
The bSP (Montrer KLK6 Anticorps)-D gene has 9 exons spanning about 10.5 kb on chromosome 28 at position q1.8. The 5'-upstream sequence has cis (Montrer CISH Anticorps)-regulatory elements. It has 2 (Montrer HAS2 Anticorps) polymorphisms in the carbohydrate recognition domain (242 Glu (Montrer DCTN1 Anticorps)/Val & 268 Ala/Gly).
The authors demonstrated that the number and position of glycosylation sites determine viral susceptibility to the neutralizing activity of porcine SP-D.
Data show that interactions of surfactant protein D (pSP-D) with influenza A virus mediated by the N-linked carbohydrate moiety in pSP-D depend on the terminal sialic acids present on this moiety.
The presence of SP-D in endothelial cells which is NO-, PI(3 (Montrer PI3 Anticorps))K/Akt (Montrer AKT1 Anticorps)- and Erk (Montrer MAPK1 Anticorps)-dependent is demonstrated. It suggests a protective role of SP-D in these cells.
The importance of SP-D in innate immunity is underlined by its expression at the potential ports of entry of pathogens: lung, tongue, intestinal tract, thymus, skin, gall bladder, lacrimal gland, esophagus, stomach, kidney, liver, prostate and spleen.
Colitis caused a notable increase in Sftpd gene expression in the gallbladder, but not in the lung, via the activity of glucocorticoids produced in the liver.
SP-D deficiency reduces atherosclerosis in ApoE (Montrer APOE Anticorps)-knockout mice by decreasing the accumulation and proliferation of macrophages and by reducing IL-6 (Montrer IL6 Anticorps) levels systemically.
The data provide evidence SP-A (Montrer SFTPA1 Anticorps) and SP-D attenuate S. aureus pneumonia severity resulting in decreased intestinal mucosal injury, apoptosis, and inflammation.
In triple KO mice, the HO-induced lung injury was associated with decreased surfactant protein (SP) A (Montrer SFTPA1 Anticorps) and SPC (Montrer SFTPC Anticorps) but not SPB and SPD expression.
Our study provides comprehensive information regarding spatial and temporal expression of collectins (SP-A (Montrer SFTPA1 Anticorps), SP-D and MBL-A (Montrer Mbl1 Anticorps)) and their cellular sources in murine testis. Positive regulation by testosterone and alteration in their level upon LPS (Montrer TLR4 Anticorps) challenge are clues relevant to suggest their involvement in testicular immune privilege.
Our data may promote further studies on the morphological development of the aging lung and the role of SP-D in this process.
The experimentally induced alterations were more severe in the SP-D(-/-) than in the WT mice, particularly with respect to the surfactant-storing lamellar bodies which were sometimes extremely enlarged in SP-D(-/-) mice.
these results demonstrate SP-D plays protective roles by inhibiting apoptosis and modulating NF-kappaB-mediated inflammation in CLP-induced acute pancreatic injury .
SP-A (Montrer SFTPA1 Anticorps)/D double knock-out mice showed increased susceptibility to urinary tract infection
Human and murine data together indicate that SP-A, SP-D and MBL are synthesized in early gestational tissues, and may contribute to regulation of immune response at the feto-maternal interface during pregnancy.
Surfactant protein D and haptoglobin (Montrer HP Anticorps) serum concentrations could be a diagnostic aid for equine inflammatory airway disease.
The levels of surfactant protein D and serum amyloid A protein in normal horses and those with bacterial pneumonia are reported.
Contributes to the lung's defense against inhaled microorganisms. May participate in the extracellular reorganization or turnover of pulmonary surfactant. Binds strongly maltose residues and to a lesser extent other alpha-glucosyl moieties.
, lung surfactant protein D
, pulmonary surfactant apoprotein
, pulmonary surfactant-associated protein D
, surfactant, pulmonary-associated protein D
, surfactant-associated protein, pulmonary 4
, pulmonary surfactant protein D
, surfactant associated protein D
, surfactant protein D
, carbohydrate recognition domain
, LOW QUALITY PROTEIN: pulmonary surfactant-associated protein D