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proton cotransporter\; mediates the uptake of neurotransmitters into vesicles [RGD, Feb 2006].. De plus, nous expédions SV2B Protéines (8) et et beaucoup plus de produits pour cette protéine.
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Human Polyclonal SV2B Primary Antibody pour ICC, IP - ABIN1742258
Boido, Farisello, Cesca, Ferrea, Valtorta, Benfenati, Baldelli: Cortico-hippocampal hyperexcitability in synapsin I/II/III knockout mice: age-dependency and response to the antiepileptic drug levetiracetam. dans Neuroscience 2010
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Human Monoclonal SV2B Primary Antibody pour ICC, IP - ABIN1742257
Dagar, Nagar, Goel, Cherukuri, Dhingra: Loss of photoreceptors results in upregulation of synaptic proteins in bipolar cells and amacrine cells. dans PLoS ONE 2014
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To study the role of SV2B in maintaining the podocyte function and to better understand the function of the neuron-like vesicle expressing SV2B in the podocyte, we analyzed them with SV2B knockout (KO) mice.
Glutamatergic & GABAergic transmission are unaffected by deletion of the SV2B protein. Additional deletion of SV2B does not affect the electrophysiological phenotype of SV2A deletion.
Loss of synaptic vesicle glycoprotein 2b is associated with inhibition of tetanus neurotoxin into central neurons.
These results indicate that SV2 performs at least two actions at the synapse that contribute to neurotransmitter release.
SV2 controls key aspects of synaptic functionality via its ability to regulate presynaptic Ca(2+).
SV2B-synaptotagmin 1 complex is involved in the regulation of synaptotagmin 1 stability and/or trafficking
SV2 action modulates synaptic networks by ensuring that low-frequency neurotransmission is faithfully conveyed.
findings show that botulinum neurotoxin (BoNT/A) enters neurons by binding to the synaptic vesicle protein SV2 (isoforms A, B, and C);SV2 acts as the protein receptor for BoNT/A
Data demonstrate that glycosylated SV2A and SV2B act in conjunction with gangliosides to mediate the entry of BoNT/E into neurons.
SV2B contributes to the modulation of synaptic vesicle exocytosis and plays a significant role in regulating synaptic protein content
In classical mesial temporal sclerosis 1A, the expression of SV2 isoforms is altered with a marked decrease of SV2B paralleling synaptic loss.
Synaptic vesicle protein 2 binds adenine nucleotides
proton cotransporter\; mediates the uptake of neurotransmitters into vesicles
synaptic vesicle protein 2B homolog
, synaptic vesicle glycoprotein 2B
, synaptic vesicle glycoprotein 2B-like
, synaptic vesicle protein 2B