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Involved in the conversion of lanosterol to cholesterol.. De plus, nous expédions Transmembrane 7 Superfamily Member 2 Anticorps (36) et Transmembrane 7 Superfamily Member 2 Kits (6) et beaucoup plus de produits pour cette protéine.
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Tm7sf2 deficiency during liver regeneration alters lipid metabolism and generates a stress condition, which, in turn, transiently unbalances hepatocytes cell cycle progression.
The Tm7sf2 insufficiency is responsible for the inhibition of the NF-kappaB (Montrer NFKB1 Protéines) signaling.
the loss of Tm7sf2 alters the expression of proteins involved in epidermal differentiation by reducing the levels of cholesterol sulfate
These results show a significant increase in renal TNFalpha (Montrer TNF Protéines) expression after tunicamycin exposure and in the oedematogenic response in Tm7sf2(-/-) mice.
several genes involved in cell-cycle progression are downregulated in the liver of Tm7sf2((-/-)) mice, whereas genes involved in xenobiotic metabolism are upregulated.
Data suggest that, besides the SRE motif, both the inverted CCAAT-box and GC-box2 are essential for full TM7SF2 promoter activation by SREBP-2 (Montrer SREBF2 Protéines).
LBR (Montrer LBR Protéines) mutant variants and sterol reductases can severely interfere with the regular organization of the nuclear envelope and the endoplasmic reticulum.
Results show that the TM7SF2 gene product SR-1 and six chimeras containing SR-1 and Neurospora erg-3 (Montrer SC5DL Protéines) sequences fail to complement sterol c-14 reductase mutants in Neurospora and yeast.
Involved in the conversion of lanosterol to cholesterol.
transmembrane 7 superfamily member 2
, C-14 sterol reductase
, delta(14)-sterol reductase
, sterol C14-reductase
, another new gene 1 protein
, putative sterol reductase SR-1
, transmembrane 7 superfamily member 2 L homeolog