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TNFAIP2 was identified as a gene whose expression can be induced by the tumor necrosis factor alpha (TNF) in umbilical vein endothelial cells. De plus, nous expédions TNFAIP2 Kits (15) et TNFAIP2 Protéines (3) et beaucoup plus de produits pour cette protéine.
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Knockdown of TNFaip2 in Raw264.7 cells inhibited formation of tunneling nanotubes.
A polarized secretion assay suggested that the silencing of endothelial myeloid-secretory impairs T effector transendothelial migration by reducing the preferential secretion of endothelial-produced CCL2 (Montrer CCL2 Anticorps).
Data indicate that TNFAIP2 overexpression may facilitate proliferation and metastasis via activation of the Wnt (Montrer WNT2 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) signaling pathway in esophageal squamous cell carcinoma.
Taken together, genome-wide chromatin analysis of Legionella pneumophila-infected macrophages demonstrated induction of TNFAIP2, a NF-kappaB (Montrer NFKB1 Anticorps)-dependent factor relevant for bacterial replication.
data suggest that TNFAIP2 is a novel inhibitor of NF-kappa-B (Montrer NFKB1 Anticorps) that acts as an autoinhibitor of the TNFalpha (Montrer TNF Anticorps) response during septic shock.
findings collectively suggest that TNFAIP2 is a direct KLF5 (Montrer KLF5 Anticorps) target gene, and both KLF5 (Montrer KLF5 Anticorps) and TNFAIP2 promote breast cancer cell proliferation, migration and invasion through Rac1 and Cdc42 (Montrer CDC42 Anticorps)
A functional TNFAIP2 3'-UTR (Montrer UTS2R Anticorps) rs8126 genetic polymorphism contributes to risk of esophageal squamous cell carcinoma.
the regulation of TNFAIP2 and its role in promoting NPC (Montrer NPC1 Anticorps) tumor progression
The TNFAIP2 miRNA binding site rs8126 T>C single nucleotide polymorphism may be a marker for susceptibility to gastric cancer.
A functional variant at the miR (Montrer MLXIP Anticorps)-184 binding site in TNFAIP2 is associated with squamous cell carcinoma of the head and neck.
TNFAIP2 may serve as a useful new marker of dendritic and histiocytic sarcomas.
This gene was identified as a gene whose expression can be induced by the tumor necrosis factor alpha (TNF) in umbilical vein endothelial cells. The expression of this gene was shown to be induced by retinoic acid in a cell line expressing a oncogenic version of the retinoic acid receptor alpha fusion protein, which suggested that this gene may be a retinoic acid target gene in acute promyelocytic leukemia.
TNF alpha-induced protein 2
, primary response gene B94 protein
, tumor necrosis factor alpha-induced protein 2
, tumor necrosis factor induced protein 2
, exocyst complex component 3-like 3