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ZMAT3 encodes a protein containing three zinc finger domains and a nuclear localization signal. De plus, nous expédions Zinc Finger, Matrin-Type 3 Kits (7) et Zinc Finger, Matrin-Type 3 Protéines (7) et beaucoup plus de produits pour cette protéine.
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The Xpa (Montrer XPA Anticorps)/p53 (Montrer TP53 Anticorps)+/- mouse model is an excellent candidate for a future replacement of the chronic mouse bioassay for certain classes of carcinogens.
identification of functional p53 (Montrer TP53 Anticorps)-binding motifs in promoter
WIG1 governs the miRNA-dependent and the miRNA-independent recruitment of AGO2 (Montrer EIF2C2 Anticorps) to lower the stability of and suppress the translation of ACOT7 (Montrer ACOT7 Anticorps) mRNA.
we propose that Wig1, a key p53 (Montrer TP53 Anticorps) downstream molecule in HD condition, play an important role in stabilizing mutant Htt (Montrer HTT Anticorps) mRNA and thereby accelerating HD pathology in the mHtt-p53 (Montrer TP53 Anticorps)-Wig1 positive feedback manner
expression of the p53 (Montrer TP53 Anticorps) target Wig-1 is associated with HPV status and patient survival in cervical carcinoma
Results suggest a role of Wig-1 as a survival factor that directs the p53 (Montrer TP53 Anticorps) stress response toward cell cycle arrest rather than apoptosis through the regulation of FAS (Montrer FAS Anticorps) and 14-3-3sigma mRNA levels.
PSF and MATR3 are cellular host factors that bind viral RNA and promote Rev activity.
The data indicated a novel role for Wig1 in RNA-induced silencing complex target accessibility, which is a key step in RNA-mediated gene silencing. Fine tuning of p21 (Montrer CDKN1A Anticorps) levels by Wig1 was essential for the prevention of cellular senescence.
Wig-1 knockdown causes a dramatic inhibition of N-Myc (Montrer MYCN Anticorps) expression and triggers differentiation in neuroblastoma (Montrer ARHGEF16 Anticorps) cells carrying amplified N-Myc (Montrer MYCN Anticorps).
WIG-1 might be involved in the zinc-induced cell cycle arrest and apoptosis of human esophageal squamous cell carcinoma cells
WIG-1 might be a novel modifier in esophageal carcinogenesis, and the WIG-1 MAb should be useful in further study of the mechanism in WIG-1-related physiological reactions.
The cellular distribution of the nuclear matrix protein matrin 3 (Montrer MATR3 Anticorps) is altered by novel phosphorylation induced by the alphaherpesvirus US3 kinase family.
This gene encodes a protein containing three zinc finger domains and a nuclear localization signal. The mRNA and the protein of this gene are upregulated by wildtype p53 and overexpression of this gene inhibits tumor cell growth, suggesting that this gene may have a role in the p53-dependent growth regulatory pathway. Alternative splicing of this gene results in two transcript variants encoding two isoforms differing in only one amino acid.
p53-activated gene 608 protein
, wild-type p53-induced gene 1 protein
, zinc finger matrin-type protein 3
, zinc finger protein WIG-1
, p53 target zinc finger protein
, wild-type p53-induced gene 1
, WIG-1/PAG608 protein
, zinc finger protein WIG1
, zinc finger, matrin type 3