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GABA is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. De plus, nous expédions GABRA3 Protéines (6) et GABRA3 Kits (4) et beaucoup plus de produits pour cette protéine.
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Mouse (Murine) Polyclonal GABRA3 Primary Antibody pour ICC, IF - ABIN253227
Schwirtlich, Emri, Antal, Máté, Katarova, Szabó: GABA(A) and GABA(B) receptors of distinct properties affect oppositely the proliferation of mouse embryonic stem cells through synergistic elevation of intracellular Ca(2+). dans FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2010
Show all 2 Pubmed References
Rat (Rattus) Polyclonal GABRA3 Primary Antibody pour ICC, IHC - ABIN1742459
Tzanoulinou, García-Mompó, Castillo-Gómez, Veenit, Nacher, Sandi: Long-term behavioral programming induced by peripuberty stress in rats is accompanied by GABAergic-related alterations in the Amygdala. dans PLoS ONE 2014
Rat (Rattus) Polyclonal GABRA3 Primary Antibody pour IHC - ABIN1742460
Eyre, Nusser: Only a Minority of the Inhibitory Inputs to Cerebellar Golgi Cells Originates from Local GABAergic Cells. dans eNeuro 2016
These small-molecule GlyR PAMs have high potential both as early tool compounds to enable pharmacological studies of GlyR inhibitory neurotransmission and as a starting point for the development of potent, selective GlyRalpha3 (Montrer GLRa3 Anticorps) PAMs as novel analgesics.
GABA alpha2 and/or alpha3 receptor subtypes are involved in GABAergic modulation of prolactin (Montrer PRL Anticorps) secretion.
GABRA3 induced MMP-2 (Montrer MMP2 Anticorps) and MMP-9 (Montrer MMP9 Anticorps) expression through activation of the JNK (Montrer MAPK8 Anticorps)/AP-1 (Montrer FOSB Anticorps) signaling pathway, promoting lymphatic metastasis in lung adenocarcinoma.
Gabra3 activates the AKT (Montrer AKT1 Anticorps) pathway to promote breast cancer cell migration, invasion and metastasis. The A-to-I RNA-edited form of Gabra3 is found only in non-invasive breast cancers. Edited Gabra3 suppresses breast cancer cell invasion and metastasis.
The N-terminal region of GABRA3 and the GlyR beta subunit (Montrer POLG Anticorps) occupies the same binding site of gephyrin (Montrer GPHN Anticorps).
high expression of GABBR2 (Montrer GABBR2 Anticorps) with a low expression of GABR(A3) may predict a better outcome in non-small cell lung cancer
A novel role for GA-repeat core promoter to regulate gene expression in the genes such as GABRA3 involved in development and evolution.
We identified truncating mutations in distinct X-linked gamma-aminobutyric acid A (GABAA (Montrer GABRg1 Anticorps)) receptor subunit-encoding genes, GABRQ (Montrer GABRQ Anticorps) and GABRA3.this is the first report of ASD (Montrer ARSD Anticorps) patients with truncating mutations in GABA receptors genes.
The results indicate that there may be specific GABA receptor gene expression variation in migraine, particularly involving the GABRA3 and GABBR2 (Montrer GABBR2 Anticorps) genes.
The ratios of beta(3)/beta(2) and alpha(5)/alpha(1) subunit protein expression in Angelman syndrome (Montrer ube3a Anticorps) cortex were significantly decreased when compared with controls.
Absence of the Gabra3 receptor subunit slows recovery from ethanol-induced incoordination.
Quantitative trait locus analysis identifies Gabra3 as a regulator of behavioral despair in mice
Despite the exclusive expression of alpha3-containing GABAA (Montrer GABRg1 Anticorps) receptors in the reticular thalamic nucleus, these receptors do not seem to be critical for the mediation of the effects of diazepam on the sleep EEG
editing of the Gabra-3 mRNA is important for normal brain development.The extent of editing is low at birth but increases with age, reaching close to 100% in the adult brain.
The phenotypes following global deletion of the GABAA (Montrer GABRg1 Anticorps) receptor alpha3 subunit provided relevant insights into the potential behavioural relevance of this specific receptor subtypes in the modulation of both affective and cognitive functions.
Deletion of the GABAAR (Montrer GABRG2 Anticorps) alpha3 subunit reduces evoked thalamic oscillatory activity.
Resutls show that the variation in editing gave receptors like HTR2C (Montrer HTR2C Anticorps) and GABA(A) (gamma-aminobutyric acid type A) a different set of protein isoforms during development from those in the adult animal.
GABA is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. Chloride conductance of these channels can be modulated by agents such as benzodiazepines that bind to the GABA-A receptor. At least 16 distinct subunits of GABA-A receptors have been identified.
GABA(A) receptor subunit alpha-3
, GABA(A) receptor, alpha 3
, gamma-aminobutyric acid receptor subunit alpha-3
, gamma-animobutyric acid (GABA) A receptor, alpha 3
, gamma-aminobutyric acid (GABA) A receptor, alpha 3
, gamma-aminobutyric acid A receptor, alpha 3
, gamma-aminobutyric acid A receptor, alpha 3-like
, gamma-aminobutyric acid receptor subunit alpha-3-like
, GABA-A receptor alpha-3 subunit
, Gamma-animobutyric acid (GABA) A receptor alpha 3
, Gamma-animobutyric acid (GABA) A receptor, alpha 3
, gamma-aminobutyric acid (GABA-A) receptor, subunit alpha 3
, gamma-aminobutyric acid receptor, subunit alpha 3