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anti-Human Angiotensin I Converting Enzyme 1 Anticorps:
anti-Mouse (Murine) Angiotensin I Converting Enzyme 1 Anticorps:
anti-Rat (Rattus) Angiotensin I Converting Enzyme 1 Anticorps:
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Human Monoclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour ELISA, WB - ABIN560577
Dekanty, Romero, Bertolin, Thomas, Leishman, Perez-Perri, Boccaccio, Wappner: Drosophila genome-wide RNAi screen identifies multiple regulators of HIF-dependent transcription in hypoxia. dans PLoS genetics 2010
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Human Polyclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour CyTOF, FACS - ABIN4900150
Rockx, Sheahan, Donaldson, Harkema, Sims, Heise, Pickles, Cameron, Kelvin, Baric: Synthetic reconstruction of zoonotic and early human severe acute respiratory syndrome coronavirus isolates that produce fatal disease in aged mice. dans Journal of virology 2007
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Human Monoclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour FACS - ABIN2688976
Sinka, Biasch, Khazaal, Péault, Tavian: Angiotensin-converting enzyme (CD143) specifies emerging lympho-hematopoietic progenitors in the human embryo. dans Blood 2012
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Mouse (Murine) Polyclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour CyTOF, FACS - ABIN4900261
Park, Bivona, Kobori, Seth, Chappell, Lazartigues, Harrison-Bernard: Major role for ACE-independent intrarenal ANG II formation in type II diabetes. dans American journal of physiology. Renal physiology 2009
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Cat (Feline) Monoclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour FACS - ABIN2478516
Danilov, Muzykantov, Martynov, Atochina, Sakharov IYu, Trakht, Smirnov: Lung is the target organ for a monoclonal antibody to angiotensin-converting enzyme. dans Laboratory investigation; a journal of technical methods and pathology 1991
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Cat (Feline) Monoclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour IHC (fro), FACS - ABIN2478509
Danilov, Jaspard, Churakova, Towbin, Savoie, Wei, Alhenc-Gelas: Structure-function analysis of angiotensin I-converting enzyme using monoclonal antibodies. Selective inhibition of the amino-terminal active site. dans The Journal of biological chemistry 1994
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Human Polyclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour IF (p), IHC (p) - ABIN668631
Liu, Jiang, Zhang, Li, Li, Xie, Hu: Pulmonary artery denervation improves pulmonary arterial hypertension induced right ventricular dysfunction by modulating the local renin-angiotensin-aldosterone system. dans BMC cardiovascular disorders 2016
Human Polyclonal Angiotensin I Converting Enzyme 1 Primary Antibody pour IHC (p), WB - ABIN3042841
Yan, Shen: Aliskiren has chondroprotective efficacy in a rat model of osteoarthritis through suppression of the local renin-angiotensin system. dans Molecular medicine reports 2018
This study suggests that the ACE and CKMM polymorphisms influence the endurance performance phenotype in non-trained adolescent males.
ACE genetic variation may modify the effect of adult adiposity on increasing BP and risk of hypertension in adulthood. Individuals with ACE DD (or GG) and/or ID (or AG) genotypes, compared with those with II (or AA) genotype, appear more vulnerable to the impact of excess adiposity.
The autonomic nervous system response to the acute volume removal during hemodialysis may be different between angiotensin-converting enzyme insertion/deletion polymorphisms.
No Angiotensin 2 Type 1 Receptor genotype associations with patient characteristics and outcome measures were identified in univariable analysis for the entire patient group and those older than 55 years.
The polymorphic allele of the ACE gene is capable of modulating triglyceride levels, insulin levels and homeostatic model assessment for Insulin Resistance index in the evaluated population; it must be highlighted that this has not been reported in other studied populations elsewhere.
We have hypothesized the mechanism that reverses the downregulation of the ACE2-angiotensin 1-7/Mas receptor axis path and the upregulation of angiotensin receptor type 1-mediated signaling. Thus, we posit that ACE2, Ang-(1-7), and the Mas receptor could be novel therapeutic targets for treating benign prostatic hyperplasia
The obtained data showed that the presence of the D allele of ACE might be a risk factor for the development of glioma
The results of the current meta-analysis indicate that the angiotensin-converting enzyme D allele might be a risk factor against the risk of Henoch-Schonlein purpura, especially in Caucasians.
The ACE gene I/D polymorphism was not associated with an increased risk of diabetic nephropathy in the Moroccan population.
ACE insertion/deletion gene polymorphism D allele may be considered as a risk factor for both the development of diabetic nephropathy and the progression of DN to ESRD in Lebanese population with T2DM.
In the group of shors with arterial hypertension, high albuminuria was associated with polymorphisms of the ACE genes--- The primary predictors determining the development of high albuminuria among patients with arterial hypertension in both ethnic groups were genetic ones.
In Liver Cirrhosis, ACE I/D polymorphism might play a role in determining the severity of Portal Hypertension and the risk of Variceal Bleeding, regardless of the severity of Liver disease.
Review/Meta-analysis: D allele in the ACE gene was associated with the risk of atherosclerosis, especially in Europeans. Increased copy number of D allele was significantly associated with increased AS risk in a dose-dependent manner.
This cross-sectional study demonstrates that sIL-2R is a useful marker for diagnosing sarcoidosis in patients with uveitis and has slightly better diagnostic value than ACE.
Mutations at the ACE rs121912703, rs767880620, and rs397514689 loci affect the efficacy of perindopril on ventricular remodeling and hemodynamics in Chinese Han Acute Myocardial Infarction (AMI) patients. The 3-year survival of AMI patients harboring the variant alleles is less than that of the patients harboring the wild-type gene.
Report alterations in the expression of cannabinoid receptors, apelin and S100A6 in the hearts of women over 50.
This study showed that in an elderly population in Jakarta, the DD genotype was associated with low muscle mass. Individuals with the DD genotype showed lower muscle mass that was significantly different compared to the muscle mass in individuals with the II/ID genotype (II 16.14 +/- 0.38, ID 15.71 +/- 0.59; DD 13.95 +/- 0.61 kg), after adjusting for % fat as a covariate.
ACE I/D polymorphism is associated with increased risk of Recurrent Pregnancy Loss in Caucasian and West-Asian populations, but not in East-Asians (Meta-Analysis)
the insertion/deletion polymorphism of the angiotensin-converting enzyme gene in mothers might be associated with preterm birth (Meta-Analysis)
No association has been found between ACE gene polymorphism and arterial hypotension in premature infants with early onset bacterial infections.
ACE's two catalytically independent domains, the N- and C-domains, can process a variety of substrates other than angiotensin I. In models lacking either a functional ACE N-domain (NKO) or C-domain (CKO), in response to a saline challenge, diabetic NKO mice excreted 32% more urinary sodium compared with diabetic wild-type or CKO mice.
We show sex dimorphism in the severity of diabetes- and ANGII-related renal lesions, and demonstrate that ACE2- and ACE-related compensatory mechanisms are sex-specific. Supporting our previous findings, the modulation and ANGII-mediated crosstalk between ACE2 and ACE in diabetic nephropathy progression was more evident in males.
Endothelial ACE is a central regulator of the glomerular filtration rate while tubular ACE is a key player in the development of tubular injury and albuminuria in diabetic mice.
ACE expression/phosphorylation in the bone-marrow niche interface negatively regulates G-CSF-induced signaling and hematopoietic progenitor cell mobilization.
first evidence indicating the causation between ACE DD or B2R+9bp genotype and the increased risk for diabetic nephropathy, broadening our horizon about the role of genetic modulators in this disease.
a small increase in angiotensin-converting enzyme activity in diabetic animals leads to greater impairment of autonomic function, as demonstrated by increased sympathetic modulation and reduced cardiac vagal modulation along with increased renal expression of Ang II.
Resveratrol upregulated ACE2 and inhibited abdominal aortic aneurysm growth in a mouse model.
In mice with renal injury induced by L-NAME pretreatment, renal tubular epithelial ACE is essential for renal angiotensin II accumulation and salt-sensitive hypertension
Studied ACE role in peptide processing and for MHC class II antigen presentation; found ACE level effects efficiency of antigen presentation, and overexpression or inhibition of ACE alters the CD4(+) T-cell and antibody response.
ACE enhances the oxidative response and bactericidal activity of neutrophils.
this study shows that angiotensin-converting enzyme inhibitor captopril rescues mice from endotoxin-induced lethal hepatitis
Smooth muscle cell-derived ACE contributes to atherosclerosis, independent of circulating ACE activity and blood pressure.
Intestinal ACE shedding is increased by DSS-induced intestinal inflammation and parallels local corticosterone production. ACE product angiotensin II stimulates corticosterone formation in healthy intestine.
Renin angiostensin system contributes to 20-HETE-mediated microvascular remodeling in hypertension and that 20-HETE-driven microvascular remodeling independent of blood pressure elevation does not fully rely on ACE activity in the vascular endothelium.
angiotensin-converting enzyme has an essential role in hypertension induced by nitric oxide synthesis inhibition
Suggest that the ACE2-ACE imbalance plays an important role in the pathogenesis of severe acute pancreatitis and that pancreatic ACE2 is an important factor in determining the severity of SAP.
Show that tissue-specific targets are critical for the effects of miR-143/145 on smooth muscle differentiation and that angiotensin converting enzyme is one such target.
Tissue-specific expression of transgenic secreted ACE in vasculature can restore normal kidney functions, but not blood pressure, of Ace-/- mice.
ACE-null mice produce large numbers of myeloid-derived suppressor cells during chronic inflammation.
Myelomonocytic expression of catalytically active ACE, prevents cognitive decline in a murine model of Alzheimer disease.
Maintaining the balance of the ACE2/ACE axis is important for inhibiting pulmonary apoptosis during acute pulmonary embolism.
The molecular model shows for the first time the relative orientation of the sACE catalytically active domains and their spatial distance.
Tissue Ang I-II conversion depends exclusively on the ACE C-domain, whereas both domains contribute to conversion by soluble ACE and to bradykinin degradation at tissue sites.
The contributions of the C-domain and N-domain differ between DDs and IIs genotype
Corneal cells express ACE, AT(1) and AT(2)receptors. ACE inhibitor enalapril decreased corneal angiogenesis in VEGF-induced corneal neovascularization. ACE inhibitors may be novel therapy to treat corneal angiogenesis.
Angiotensin-converting enzyme (ACE) residues encompassing 343 to 655 of the germinal form are required for its cleavage-secretion.
a portion of the extracellular region of tACE (containing its catalytic site) is released from bovine sperm during capacitation, and tACE activity may be required for sperm capacitation.
The results indicate that the two active sites within bovine somatic ACE exhibit strong negative cooperativity.
Phorbol 12-myristate 13-acetate (PMA) induced Egr-1 and ATF-2 binding to the ACE promoter, whereas Ets-1 binding was suppressed by PMA.
Precompetition angiotensin converting enzyme (ACE) activity in endurance horses competing in an 80 km event was not associated with either finishing position or heart rates, indicating that the enzyme is not a good predictor of performance.
This research investigated angiotensin-converting enzyme activity in the plasma of racehorses and demonstrated that its activity is increased in horses with higher degrees of hemorrhage and is a promising biomarker for pulmonary hemorrhage.
These results show an increase in ACE activity up to fatigue and a return to baseline values at 30 min post exercise.
The aim of this study was to investigate the correlation between angiotensin-converting enzyme (ACE) activity in the equine endometrium and the degree of endometrial periglandular fibrosis.
This gene encodes an enzyme involved in catalyzing the conversion of angiotensin I into a physiologically active peptide angiotensin II. Angiotensin II is a potent vasopressor and aldosterone-stimulating peptide that controls blood pressure and fluid-electrolyte balance. This enzyme plays a key role in the renin-angiotensin system. Many studies have associated the presence or absence of a 287 bp Alu repeat element in this gene with the levels of circulating enzyme or cardiovascular pathophysiologies. Multiple alternatively spliced transcript variants encoding different isoforms have been identified, and two most abundant spliced variants encode the somatic form and the testicular form, respectively, that are equally active.
, angiotensin I converting enzyme (peptidyl-dipeptidase A) 1
, angiotensin I converting enzyme peptidyl-dipeptidase A 1 transcript
, angiotensin converting enzyme, somatic isoform
, angiotensin-converting enzyme
, dipeptidyl carboxypeptidase 1
, dipeptidyl carboxypeptidase I
, kininase II
, peptidase P
, testicular ECA
, dipeptidyl peptidase
, Dipeptidyl carboxypeptidase 1 (Angiotensin I-converting enzyme)
, angiotensin 1 converting enzyme 1
, angiotensin I converting enzyme 1
, angiotensin I-converting enzyme (Dipeptidyl carboxypeptidase 1)
, Angiotensin-converting enzyme, testis-specific isoform
, dipeptidyl carboxy peptidase 1
, angiotensin converting enzyme
, angiotensin-I converting enzyme
, peptidyl dipeptidase i
, angiotensin I-converting enzyme
, Dipeptidyl carboxypeptidase I
, Kininase II
, LOW QUALITY PROTEIN: angiotensin-converting enzyme